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| 1 | Portal hypertension due to portal venous thrombosis:Etiology, clinical outcomes显示文摘The thrombophilia in adult life has major implications in the hepatic vessels. The resulting portal vein thrombosis has various outcomes and complications. Esophageal varices, portal gastropathy, ascites, severe hypersplenism and liver failure needing liver transplantation are known well. The newly formed collateral venous circulation showing itself as pseudocholangicarcinoma sign and its possible clinical reflection as cholestasis are also known from a long time. The management strategies for these complications of portal vein thrombosis are not different from their counterpart which is cirrhotic portal hypertension, but the prognosis is unquestionably better in former cases. In this review we present and discuss the portal vein thrombosis, etiology and the resulting clinical pictures. There are controversial issues in nomenclature, management (including anticoagulation problems), follow up strategies and liver transplantation. In the light of the current knowledge, we discuss some controversial issues in literature and present our experience and our proposals about this group of patients. | Ozgur Harmanci Yusuf Bayraktar | 2007 | World Journal of Gastroenterology2007，13，18： | 21 |
| 2 | How can portal vein cavernous transformation cause chronic incomplete biliary obstruction?显示文摘Biliary disease in the setting of non-cirrhotic portal vein thrombosis(and similarly in portal vein cavernous transformation) can become a serious problem during the evolution of disease.This is mostly due to portal biliary ductopathy.There are several mechanisms that play a role in the development of portal biliary ductopathy,such as induction of fibrosis in the biliary tract(due to direct action of dilated peribiliary collaterals and/or recurrent cholangitis),loss of biliary motility,chronic cholestasis(due to fibrosis or choledocholithiasis) and increased formation of cholelithiasis(due to various factors).The management of cholelithiasis in cases with portal vein cavernous transformation merits special attention.Because of a heterogeneous clinical presentation and concomitant pathophysiological changes that take place in biliary anatomy,diagnosis and therapy can become very complicated.Due to increased incidence and complications of cholelithiasis,standard treatment modalities like sphincterotomy or balloon sweeping of bile ducts can cause serious problems.Cholangitis,biliary strictures and hemobilia are the most common complications that occur during management of these patients.In this review,we specifically discuss important issues about bile stones related to bile duct obstruction in non-cirrhotic portal vein thrombosis and present evidence in the current literature. | Ozgur Harmanci Yusuf Bayraktar | 2012 | World Journal of Gastroenterology2012，18，26： | 19 |
| 3 | Etiology and consequences of thrombosis in abdominal vessels显示文摘能在成年生命先天或获得的血栓形成倾向在腹的容器有主要含意。产生的门静脉血栓, Budd-Chiari 症候群和 mes 伤寒静脉血栓有许多后果从急腹症到唯一的发现在是各类血细胞减少的长期的肝肿大和甚至完全无征状的病人。复杂并发症喜欢食道的静脉曲张,门胃病,腹水,严重脾机能亢进,肝失败要求肝移植是众所周知的。作为胆汁郁积显示出 pseudocholangiocarcinoma 签名的本身和它的可能的临床的思考的并行的静脉的循环的有趣的特征也从很长时间被知道。为 intraabdominal 容器血栓的这些复杂并发症的管理策略不与他们是肝脏硬化症的门静脉高血压的对应物不同,但是预后在以前的情况中无疑地更好。在这评论,我们介绍了并且讨论腹的静脉血栓形成,病原学并且结果临床的图画。包括反凝结问题在名称,和管理有争论问题并且在策略上面列在后面。考虑到当前的知识,我们在文学讨论了一些争论问题并且关于这组病人介绍了我们的经验和我们的建议。 | Yusuf Bayraktar Ozgur Harmanci | 2006 | World Journal of Gastroenterology2006，12，8： | 17 |
| 4 | Clinical characteristics of idiopathic portal hypertension显示文摘Idiopathic portal hypertension is one of the interesting causes of portal hypertension. Even in very developed medical centers, this disorder is still one of the most important misdiagnoses of clinical practice. To inexperienced physicians, presenting esophageal varices and upper gastrointestinal bleeding usually prompt an unfortunate diagnosis of cirrhosis. A heterogenous clinical presentation and progression of this disorder should be recognized by physicians, and management should be directed towards some specific problems confined to this disorder. Although a genetic basis and other factors are implicated in its pathogenesis, exact underlying mechanism(s) is (are) unknown. In this review, we discuss the heterogeneity of idiopathic portal hypertension, its etiopathogenesis, clinical presentation and management issues. With the expectation of an excellent prognosis, a practicing gastroenterologist should be aware that 'not all varices mean cirrhosis'. | Ozgur Harmanci Yusuf Bayraktar | 2007 | World Journal of Gastroenterology2007，13，13： | 6 |
| 5 | Does endothelium agree with the concept of idiopathic hepatic vessel thrombosis?显示文摘瞄准:为了调查血栓形成的主要的步并且在这些识别差别,走在自发的耐心的组和控制组之间。方法:Fibrinogenesis 被测量激活的第七因子,总数和织物因素小径禁止者(TFPI ) 的免费层次学习。纤维蛋白溶解作用步被决定全球 fibrinolytic 能力调查。内皮功能被测量可溶的粘附分子的层次估计,也就是可溶的细胞间的粘附分子 1 (sICAM-1 ) ,可溶的脉管的房间粘附分子 1 (sVCAM-1 ) 并且可溶的 E-selectin 分子。从“自发”的耐心的组的排除标准是腹的外科,怀孕,口服避孕药的使用, anti-phospholipid 症候群, Behcet 的疾病,癌症, myeloproliferative 疾病。先天的因素喜欢 factor-V Leiden 的变化,凝血素,蛋白质 C 和 S 的缺乏,反凝血酵素, hyperhomocysteinemia 和 hyperfibrinogenemia 被排除。病人的全部的数字从 96 ~ 9 被减少(7 与门静脉血栓, 2 Budd Chiari 症候群) 由排除标准。结果:粘附分子 sICAM-1, sVCAM-1,免费 TFPI 层次和全球 fibrinolytic 能力的层次是显著地不同的(P<0.05 ) 在耐心的组显示一个内皮机能障碍和一项更低的 fibrinolytic 活动。结论:这些结果证明这个耐心的组应该借助于内皮机能障碍被测试并且不同地设法。 | Ozgur Harmanci Yahya Buyukasik Serafettin Kirazli Ferhun Balkanci Yusuf Bayraktar | 2006 | World Journal of Gastroenterology2006，12，8： | 4 |
| 6 | Gaucher disease:New developments in treatment and etiology显示文摘Gaucher disease (GD) is an autosomal recessive disease which if undiagnosed or diagnosed late results in devastating complications. Because of the heterozygous nature of GD, there is a wide spectrum of clinical presentation. Clinicians should be aware of this rare but potentially treatable disease in patients who present with unexplained organomegaly, anemia, massive splenomegaly, ascites and even cirrhosis of unknown origin. The treatment options for adult type GD include enzyme replacement treatment (ERT) and substrate reduction treatment (SRT) depending on the status of the patient. Future treatment options are gene therapy and 'smart molecules' which provide specifi c cure and additional treatment options. In this review, we present the key issues about GD and new developments that gastroenterologists should be aware of. | Ozgur Harmanci Yusuf Bayraktar | 2008 | World Journal of Gastroenterology2008，14，25： | 2 |
| 7 | Adrenocortical steroid re- sponse to ACTH in different phenotypes of non-obese polycystic ovary syndrome显示文摘 | Cinar N Harmanci A Aksoy DY | 2012 | J Ovarian Res2012，5，1： | 1 |
| 8 | Specific im- munotherapy improves asthma related quality of life in child- hood显示文摘 | Gozde Kanmaz H Harmanci K Razi C | 2011 | Allergol Immunopathol (Madr)2011，39，： | 1 |
| 9 | A Generalization of CS-modules显示文摘 | HARMANC A SMITH P F | 1995 | Comm Algebra1995，23，4： | 1 |
| 10 | The immunostimulant 0M-85 BV prevents wheezing attacks in preschool children显示文摘 | Razi CH Harmanci K Abaci A | 2010 | J Allergy Clin Immunol2010，126，4： | 1 |
| 11 | Color image segmentation using histogram multithresholding and fusion显示文摘 | Kurugollu F Sankur B Harmanci A E | 2001 | Journal of Image and Vision Computing2001，19，13： | 1 |
| 12 | A generalization of CS-modules显示文摘 | Celik C Harmanc A Smith P F | 1995 | Comm In Alg1995，23，1： | 1 |
| 13 | Hospital-acquired pneumonia:Challengs and options for dignosis and treatment显示文摘 | Harmanci O Akora M | 2002 | J Hosp Infect2002，51，3： | 1 |
| 14 | Aucte renal failure and mortality after open-heart surgery in infants 显示文摘 | Baskin E Saygili A Harmanci K | 2005 | Renal Failure2005，27，5： | 1 |
| 15 | Role of procalcitonin and CRP in diagnosis and follow-up of neonatal sepsis 显示文摘 | Koksal N Harmanci R Cetinkaya M | 2007 | Turk J Pediatr2007，49，1： | 1 |
| 16 | Nasal and bronchial response to exercise in children with seasonal allergic rhinitis out of the pollen season显示文摘 | Koray Harmanc? Bar?s Urhan Hülya An?l Abdulkad?r Kocak | 2015 | Int Forum Allergy Rhinol2015，，： | 1 |
| 17 | Factors affecting bronchial hyperreactivity in asthmatic children显示文摘 | Harmanci K Bakirtas A Turktas I | 2008 | J Asthma2008，45，9： | 1 |
| 18 | Integrative Annotation of Variants from 1092 Humans: Application to Cancer Genomics显示文摘 | Ekta Khurana Yao Fu Vincenza Colonna Xinmeng Jasmine Mu Hyun Min Kang Tuuli Lappalainen Andrea Sboner Lucas Lochovsky Jieming Chen Arif Harmanci Jishnu Das Alexej Abyzov Suganthi Balasubramanian Kathryn Beal Dimple Chakravarty Daniel Challis Yuan Chen Dec | 2013 | Science2013，，6154： | 1 |
| 19 | The Genetic Basis of the Polycystic Ovary Syndrome : A Literature Review Including Discussion of' PPAR - γ显示文摘 | Ugur Unluturk Ayla Harmanci Cetin Kocaefe | 2007 | BJOG2007，10，： | 1 |
| 20 | A new population based adaptive dominance change mechanism for diploid genetic algorithms in dynamic environments显示文摘 | UYAR A S HARMANCI A E | 2005 | Soft Computing2005，9，11： | 1 |
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