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35篇 您的检索式:作者名="POGRIBNA M"
    题名 作者 年代 出处 被引量
1Homocysteine metabolism in children with Down syndrome: in vitro modulation 显示文摘Pogribna M Melnyk S Pogribny I 2001Am J Hum Genet2001,69,:1
2Abnormal folate metabolism and mutation in the methylenetetrahydrofolate reductase gene may be maternal risk factors for Down syndrome显示文摘James SJ Pogribna M Pogribny IP 1999Am J Clin Nutr1999,70,4:1
3Measurement of Plasma and Intracellular S - Adenosylmethionine and S - Adenosylhomocysteine Utilizing Coulometric Electrochemical Detection: Alterations with Plasma Homocysteine and Pyridoxal 5'- Phosphate Concentrations 显示文摘Melnyk S Pogribna M Igor P 2000Clin Chem2000,46,:1
4Increase in plasma homocysteine associated with parallel increases in plasma S - adenosylhomocys- teine and lymphocyte DNA hypomethylation 显示文摘Yi P Melnyk S Pogribna M 2000J Biolo Chem2000,275,29:1
5Elevation in s-adenosylhomocysteine and DNA hypomethylation: Potential epigenetie mecha-nism for homocysteine-related pathology显示文摘James S J Melnyk S Pogribna M 2002J Nutr2002,132,:1
6Characterization of cellular response to silicone implants in rats: Implications for foreign-body carcinogenesis显示文摘James SJ Pogribna M Miller BJ 1997Biomaterials1997,18,9:1
7Homocysteine metabolism in children with Down syndrome: in vitro modulation 显示文摘Pogribna M Melnyk S Pogribny I 2001AmJ HumGenet2001,69,:1
8Abnormal folate metabolism and mutation in the methylenetetrahydrofolate reductase gene may be maternal risk factors for Down syndrome显示文摘James SJ Pogribna M Pogribny IP 1999Am J Clin Nutr1999,70,:1
9Mechanisms of DNA damage,DNA hypomethylation,and tumor progression in the fo- late/methyl-deficient rat model of hepatocarcinogenesis显示文摘Janies S J Pogribny I P Pogribna M 2003The Journal of nutrition2003,133,11:1
10Abnormal folate metabolism and mutation in the methylenetetrahydrofolate reductase gene may be maternal risk factorsfor Down syndrome 显示文摘James SJ Pogribna M Pogribny IP 1999Am J Clin Nutr1999,70,4:1
11Modifying metaboli-cal y sensitive histone marks by inhibiting glutamine metabolism affects gene expression and alters cancer cellphenotype显示文摘Simpson NE Tryndyak VP Pogribna M 0,,12:1
12Uracil misincorporation,DNA strand breaks,and gene amplification are associated with tumorigenic cell transformation in folate deficient/repleted Chinese hamster ovary cells显示文摘Melnyk S Pogribna M 1999Cancer Lett1999,146,1:1
13Uracil misincorporation, DNA strand breaks, and gene amplification are associated with tumorigenic cell transfor- mation in folate deficient/repleted Chinese hamster ovary cells 显示文摘Melnyk S Pogribna M Miller B J Basnakian A G Pogribny I P James S J 1999Cancer Lett1999,146,:1
14Homocysteine metabolism in children with Down syndrome : in vitro modulation 显示文摘Pogribna M Melnyk S Pogribny I 2001Am J Hum Genet2001,69,1:1
15Apoptosis and proliferation under conditions of deoxynucleotide pool imbalance in liver of folate/methyl deficient rats显示文摘James S J Miller B J Basnakian A G Pogribny I P Pogribna M Muskhelishvili L 1997Carcinogenesis1997,18,:1
16Uracil misincorporation, DNA strand breaks ,and gene amplification are associated with tumorigenic cell transformation in folate deficlent/repleted Chinese hamster ovary cells 显示文摘Melnyk S Pogribna M Miller BJ 1999Cancer Lett1999,146,1:1
17Elevation in S- adenosylhomocysteine and DNA hypomethylation: poten- tial epigenetic mechanism for homocysteine-related pa- thology显示文摘James SJ Melnyk S Pogribna M 2002J Nutr2002,132,8:1
18Measurement of plasma and intracellular S-adenosylmethionine and S-adenosyl homocysteine utilizing coulometric electrochemical detection: alterations with plasma homocysteine and pyridoxal 5 '-phosphate concentrations显示文摘Melnyk S Pogribna M Pogribny IP 2000Clin Chem2000,46,2:1
19Abnormal folate metabolism and mutation in the methylenetetrahydrofolate reductase gene may be maternal risk factors for Down syndrome显示文摘James SJ Pogribna M Pogribny IP 1999Am J Clin Nut1999,70,4:1
20Homocysteine metabolism in children with Down syndrome: in vitro modulation 显示文摘Pogribna M Melnyk S Pogribny I 2001Am J Hum Genet2001,69,:1
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