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| 1 | Histopathological characteristics and causes of kidney graft failure in the current era of immunosuppression显示文摘BACKGROUND The histopathological findings on the failing kidney allograft in the modern era is not well studied. In this study, we present our experience working with kidney transplant recipients with graft failure within one year of the biopsy.AIM To report the histopathological characteristics of failed kidney allografts in the current era of immunosuppression based on the time after transplant, cause of the end-stage renal disease and induction immunosuppressive medications.METHODS In a single-center observational study, we characterized the histopathological findings of allograft biopsies in kidney transplant recipients with graft failure within one year after the biopsy.RESULTS We identified 329 patients with graft failure that met the selection criteria between January 1, 2006 and December 31, 2016. The three most common biopsy findings were interstitial fibrosis and tubular atrophy(IFTA, 53%), acute rejection (AR, 43%) and transplant glomerulopathy(TG, 33%). Similarly, the three most common causes of graft failure based on the primary diagnosis were AR(40%),TG(17%), and IFTA(13%). Most grafts failed within two years of post-transplant(36%). Subsequently, approximately 10%-15% of grafts failed every two years: >2-4 years(16%), > 4-6 years(13%), > 6-8 years(11%), > 8-10 years(9%) and > 10 years(16%). AR was the most common cause of graft failure in the first six years(48%), whereas TG was the most prevalent cause of graft failure after 6 years(32%) of transplant.CONCLUSION In the current era of immunosuppression, AR is still the most common cause of early graft failure, while TG is the most prevalent cause of late graft failure. | Sandesh Parajuli Fahad Aziz Neetika Garg Sarah E Panzer Emily Joachim Brenda Muth Maha Mohamed Justin Blazel Weixiong Zhong Brad C Astor Didier A Mandelbrot Arjang Djamali | 2019 | World Journal of Transplantation2019，9，6： | 2 |
| 2 | Compartment-specific expression and function of the chemokine IP-10/CXCL10 in a model of renal endothelial microvascular injury显示文摘 | Panzer U Steinmetz O M Reinking R R Meyer T N Fehr S Schneider A | 2006 | J Am Soc Nephrol2006，17，： | 1 |
| 3 | The influence of team moni- toring on team processes and performance显示文摘 | Marks M A Panzer F J | 2004 | Human Performance2004，17，1： | 1 |
| 4 | Temperature-dependent phonon conduction and nanotube engagement in metalized single wall carbon nanotube films 显示文摘 | PANZER M A DUONG H M OKAWA J | 2010 | Nano Lett2010，10，7： | 1 |
| 5 | Infrared microscopy thermal characterization of opposing carbon nanotube arrays显示文摘 | Hu X Jack Matthew A Panzer Kenneth E Goodson | 2007 | J Heat Trans2007，129，： | 1 |
| 6 | Association of fasting plasma glu- cose with heart rate recovery in healthy adults: a population - based study 显示文摘 | Panzer C Lauer MS Brieke A | 2002 | Diabetes2002，51，： | 1 |
| 7 | The chemokine receptor antagonist AOP-RANTES reduces monocyte infiltration in experimental glomerulonephritis显示文摘 | Panzer U Schneider A Wilken J | 1999 | Kidney Int1999，56，6： | 1 |
| 8 | Melatonin has no effect on the growth, morphology or cell cycle of human breast cancer (MCF-7),cervical cancer (HeLa),osteosarcoma (MG-63) or lymphoblastoid (TK6) cells 显示文摘 | PANZER A LOTFERING ML BIANCHI P | 1998 | Cancer Lett1998，122，12： | 1 |
| 9 | Glutamatergic autoencephalitides: an emerging field 显示文摘 | Panzer JA Gleichman A J Lynch DR | 2014 | J Neural Transm2014，121，8： | 1 |
| 10 | Preventive andtherapeutic types of environmental enrichment counteractbeta amyloid pathology by different molecular mechanisms显示文摘 | Herring A Lewejohann L Panzer AL | 2011 | Neurobiol Dis2011，42，3： | 1 |
| 11 | Association of fasting plasma glu- cose with heart rate recovery in healthy adults : a population - based stffdy显示文摘 | Panzer C Lauer MS Brieke A | 2002 | Diabetes2002，51，： | 1 |
| 12 | The neurobiological basis of fear: a concise review显示文摘 | PANZER A VILJOEN M ROOS J L | 2007 | African Psychiatry Review2007，10，： | 1 |
| 13 | Monocyte chemoattractant protein- 1 mediates collagen deposition in experimental glomerulonephritis by transforming growth factor-beta显示文摘 | SCHNEIDER A PANZER U ZAHNER G | 1999 | Kidney Int1999，56，： | 1 |
| 14 | Prevention ofLype 2 diabetes and its macrovascular complication: when, when, And how should we treat显示文摘 | Panzer C B rieke A Rudeman N | 2003 | Curr Op h Endocrino 1 Diabeles2003，19，： | 1 |
| 15 | Effects of different PPAR gamma agonists on MC P1 expression and monocytere eruitment in experiment glomerulonephritis 显示文摘 | Panzer U Schneider A Guan Y | 2002 | Kindney Int2002，62，2： | 1 |
| 16 | The chemokine receptor antagonist APO-RANTES reduces monocyteinfiltration in experimental glomerulonephritis显示文摘 | Panzer U Schneider A | 1999 | Kidney Int1999，56，6： | 1 |
| 17 | Effects of different PPAR-γ agonists on MCP-1 expression and monocyte recruitment in experimental glomerulonephritis显示文摘 | Panzer U Schneider A Guan Y | 2002 | Kidney Int2002，62，2： | 1 |
| 18 | Monocyte chemoattractant protein- 1 mediates collagen deposition in experimental glomerulonephritis by transforming growth factor-beta 显示文摘 | Schneider A Panzer U Zahner G | 1999 | Kidney Int1999，56，1： | 1 |
| 19 | Mitochondrial DNA mutations,oxidative stress,and apoptosis in mammalian aging显示文摘 | Kujoth G C Hiona A Pugh T D Someya S Panzer K Wohlgemuth S E Hofer T Seo A Y | | 0，，5733： | 1 |
| 20 | The chemokine receptor antagonist AOP-RANTES reduces monocyte infiltration in experimental glomerulonephritis显示文摘 | Panzer U Schneider A Wilken J | 1999 | Kidney Int1999，56，6： | 1 |
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