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| 1 | Report of an international workshop to standardize baseline evaluation and response criteria for primary CNS lymphoma.显示文摘 | Abrey LE Batchelor TT Ferreri A J Gospodarowicz M Pulczynski EJ Zucca E Smith JR Korfel A Soussain C DeAngelis LM Neuwelt EA O′Neill BP Thiel E Shenkier T Graus F van den Bent M Seymour JF Poortmans P Armitage JO Cavalli F | 2005 | 中国神经肿瘤杂志2005，3，3： | 53 |
| 2 | Alcohol metabolites and lipopolysaccharide: Roles in the development and/or progression of alcoholic liver disease显示文摘The onset of alcoholic liver disease (ALD) is initiated by different cell types in the liver and a number of different factors including: products derived from ethanol-induced inflammation, ethanol metabolites, and the indirect reactions from those metabolites. Ethanol oxidation results in the production of metabolites that have been shown to bind and form protein adducts, and to increase inflammatory, fibrotic and cirrhotic responses. Lipopolysaccharide (LPS) has many deleterious effects and plays a significant role in a number of disease processes by increasing inflammatory cytokine release. In ALD, LPS is thought to be derived from a breakdown in the intestinal wall enabling LPS from resident gut bacterial cell walls to leak into the blood stream. The ability of adducts and LPS to independently stimulate the various cells of the liver provides for a two-hit mechanism by which various biological responses are induced and result in liver injury. Therefore, the purpose of this article is to evaluate the effects of a two-hit combination of ethanol metabolites and LPS on the cells of the liver to increase inflamma-tion and fi brosis, and play a role in the development and/or progression of ALD. | Courtney S Schaffert Michael J Duryee Carlos D Hunter Bartlett C Hamilton 3rd Amy L DeVeney Mary M Huerter Lynell W Klassen Geoffrey M Thiele | 2009 | World Journal of Gastroenterology2009，15，10： | 20 |
| 3 | Immunological response in alcoholic liver disease显示文摘The development of alcoholic liver disease (ALD) can be attributed to many factors that cause damage to the liver and alter its functions. Data collected over the last 30 years strongly suggests that an immune component may be involved in the onset of this disease. This is best evidenced by the detection of circulating autoantibodies, infiltration of immune cells in the liver, and the detection of hepatic aldehyde modified proteins in patients with ALD. Experimentally, there are numerous immune responses that occur when proteins are modified with the metabolites of ethanol. These products are formed in response to the high oxidative state of the liver during ethanol metabolism, causing the release of many inflammatory processes and potential of necrosis or apoptosis of liver cells. Should cellular proteins become modified with these reactive alcohol metabolites and be recognized by the immune system, then immune responses may be initiated. Therefore, it was the purpose of this article to shed some insight into how the immune system is involved in the development and/or progression of ALD. | Michael J Duryee Lynell W Klassen Geoffrey M Thiele | 2007 | World Journal of Gastroenterology2007，13，37： | 9 |
| 4 | 2008WHO骨髓增殖性肿瘤及骨髓增生异常综合征分型显示文摘2008年WHO骨髓增殖性肿瘤(MPN)/骨髓增生异常综合征(MDS)分型共分成4大类,每类又分多种不同疾病. | Vardiman J W Thiele J Arber DA 俞文娟(节译) 金洁(审校) | 2010 | 国际输血及血液学杂志2010，，3： | 3 |
| 5 | 关于修订WHO真性红细胞增多症、原发性血小板增多症和原发性骨髓纤维化的诊断标准的建议和说明:来自特别国际专家组的推荐显示文摘最近对BCR-ABL阴性骨髓增殖性疾病(MPDs)分子发病机制的新发现为这类疾病的遗传学分类及分子诊断带来了希望。相关报道最早发表在2005年初,这些文献描述了真性红细胞增多症(PV)、原发性血小板增多症(ET)和原发性骨髓纤维化(PMF)患者存在体细胞Janus激酶2(JAK2)的突变(JAK2617V〉F;一种外显子14体细胞1849G〉T突变);在PV患者中突变率约95%,ET和PMF均约50%。 | Tefferi A Thiele J Orazi A 蓝海峰(译) 杨仁池(校) | 2007 | 国际输血及血液学杂志2007，30，6： | 2 |
| 6 | Which Time-to-Peak Threshold Best Identifies Penumbral Flow?: A Comparison of Perfusion-Weighted Magnetic Resonance Imaging and Positron Emission Tomography in Acute Ischemic Stroke显示文摘 | J Sobesky O Zaro Weber F -G. Lehnhardt V Hesselmann A Thiel C Dohmen A Jacobs M Neveling W -D. Heiss | 2004 | Stroke2004，，12： | 2 |
| 7 | Charging processes in low vacuum scanning electron microscopy显示文摘 | THIEL B L TOTH M GRAVEN J P | 2004 | Microscopy and Microanalysis2004，，10： | 1 |
| 8 | Inhibition of pyruvat-edehydrogenase complex by moniliformin显示文摘 | Gathercole P S Thiel P G Hofmeyr J H S | 1986 | Biochemical Journal1986，233，： | 1 |
| 9 | Evidence for the presence of two novel pestivirus species 显示文摘 | Avalos-Ramirez R Orlich M Thiel H J | 2001 | Virology2001，286，： | 1 |
| 10 | Molecular signals for anaerobic methane oxidation in Black Sea seep carbonates and a microbial mat 显示文摘 | Thiel V Peckmann J Richnow H H Luth U Reitner J Michaelis W Peckmann J | 2001 | Mar Chem2001，73，2： | 1 |
| 11 | Influence of observational noise on the recurrence quantification analysis显示文摘 | THIEL M ROMANO M C KURTHS J | 2002 | Physica D-Nonlinear Phenomena2002，171，： | 1 |
| 12 | Molecular signals for anaerobic methane oxidation in Black Sea seep carbonates and a microbial mat显示文摘 | Thiel V Luth U Reitner J | 2001 | Mar Chem2001，73，： | 1 |
| 13 | Simulating flow in heterogeneous systems using streamtubes and streamlines显示文摘 | Thiele M R Batycky R P Blunt M J | 1996 | SPERE1996，10，1： | 1 |
| 14 | Differential capacity of left and fight hemispheric areas for compensation of poststroke aphasia显示文摘 | Heiss WD Kessler J Thiel A | 1999 | Ann Neurol1999，45，4： | 1 |
| 15 | Imaging of posterior cham- ber phakic intraocular lens by optical coherence tomography 显示文摘 | Beelunann M Ullrich S Thiel M J | 2002 | J Cataract Refract Surg2002，28，2： | 1 |
| 16 | The 2008 revision of the World Health Organization (WHO) classication of myeloid ne- oplasms and acute leukemia: rationale and important changes 显示文摘 | Vardiman JW Thiele J Arber DA | 2009 | Blood2009，114，： | 1 |
| 17 | Proposals and rationale for revision of the world health organization diagnostic criteria for polycythemia vera essential thrombocythemia and primary myelofibrosis recommendations from an ad hoe international expert panel 显示文摘 | Tefferi A Thiele J Orazi A | 2007 | Blood2007，110，4： | 1 |
| 18 | An overview on CALR and CSF3R mutations and a proposal for revision of WHO diagnostic criteria for myeloproliferative neoplasms 显示文摘 | Tefferi A Thiele J Vannucchi AM | 2014 | Leukemia2014，28，7： | 1 |
| 19 | A positive geno-type-phenotype correlation in a large cohort of patientswith Pseudohypoparathyroidism Type Ia and Pseudo-pseudohypoparathyroidism and 33 newly identified muta-tions in the GNAS gene显示文摘 | Thiele S Werner R Grotzinger J | 2015 | Mol Genet Genomic Med2015，3，2： | 1 |
| 20 | Valproate-induced encepha lopathy: assessment with MR imaging and 1H MR spectros copy显示文摘 | Ziyeh S Thiel T Spreer J eta| | 2002 | Epilepsia2002，43，9： | 1 |
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