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72篇 您的检索式:作者名="VIITASALO"
    题名 作者 年代 出处 被引量
1内部生态系统反馈加重了固氮蓝藻细菌藻华并使波罗的海管理复杂化显示文摘波罗的海的富营养化可能已潜在地增加了蓝藻细菌藻华发生的频率和规模。富营养化导致有机沉积物的增加,底层缺氧状况的加剧以及随后的系统内磷负荷的加重。此外,低氧水体积呈现与无机氮库负相关的关系,提示随低氧程度的增加,总氮的消除也会增加。系统内磷负荷加重和溶解的无机氮消除的增加将导致较低的氮磷比,而低的氮磷比是促进固氮蓝藻细菌藻华发生的主要原因之一。因为波罗的海开阔水域的蓝藻细菌藻华似乎被其内部作用所强有力地调控,因此,外部营养物质消减的影响与尺度相关。在较长的时间尺度上,外部磷负荷的减少可能会降低蓝藻细菌藻华的发生;然而,在较短时间尺度上,内部磷负荷可能会抵消外部磷负荷的降低。这些可诱导内部负荷(增加)、氮清除以及固氮蓝藻细菌占优势的耦合的过程,可被定性地描述为一种具有潜在自驱式的'恶性循环'。为了有效抑制蓝藻细菌藻华以及所有的富营养化征象,削减氮和磷外部负荷似乎是至关重要的。Emil Vahtera Daniel J.Conley Bo G.Gustafsson Harri Kuosa Heikki Pitknen Oleg P.Savchuk Timo Tamminen Markku Viitasalo Maren Voss Norbert Wasmund Fredrik Wulff 陈欣然 2007AMBIO-人类环境杂志2007,36,2:2
2The relationship between current and former shift work and the metabolic syndrome 显示文摘Puttonen S Viitasalo K Hrmi M 2012Scand J Work Environ Health2012,38,4:1
3Dispersio ns of the QT interval in postmyocardial infarction patients presenting with vent ricular tachycardia or with ventricular fibrillation 显示文摘 Viitasalo M Toivonen L 1998Am J Cardiol1998,81,:1
4Ambulatory electrocardiographic evidence of transmuraldispersion of repolarization in patients with long-QT syndrometype 1 and 2显示文摘VIITASALO M OIKARINEN L SWAN H 2002Circulation2002,106,19:1
5Arrhythmic disorder mapped to chromosome 1 q42-q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts显示文摘Swan H Piippo K Viitasalo M 1999J Am Coll Cardiol1999,34,:1
6Food and nutrient intake among workers with different shift systems 显示文摘Hemie K Puttonen S Viitasalo K 2015Occup Environ Med2015,72,7:1
7Muscle metabolism, blood lactate and oxygen uptake in steady state exercise at aerobic and anaerobic thresholds显示文摘H. Rusko P. Luhtanen P. Rahkila J. Viitasalo S. Rehunen M. H?rk?nen 1986European Journal of Applied Physiology and Occupational Physiology1986,,2:1
8Calanoid resting eggs in the Baltic Sea: implications for the population dynamics of Acartia bifilosa (Copepoda)显示文摘M. Viitasalo 1992Marine Biology1992,,3:1
9Analysis of sway in Parkinson's disease using a new inelinometry-based method 显示文摘Viitasalo MK Kampman V Sotaniemi KA 2002Mov Disord2002,17,4:1
10Measurement of QT interval显示文摘Mantysaari M Karjalainen J Viitasalo M 1996J Am Coil Cardiol1996,28,3:1
11Electromyographic activity related to aerobic and anaerobic threshold in ergometer bicycling显示文摘Viitasalo J T Luhtanen P Rahkila P 1985Acta Physiol Scand1985,124,2:1
12Sinus node function and ventricular repolarization during exercise stress test in long QT syndrome patients with KvLQT1 and HERG potassium channel defects显示文摘SWAN H VIITASALO M PIIPPO K 1999J Am Coil Cardiol1999,34,3:1
13Differentiation between LQT1 and LQT2 patients and unaffected subjects using 24-hour electrocardiographic recordings 显示文摘Viitasalo M Oikarinen L Vaananen H 2002Am J Cardiol2002,89,:1
14Electrophysiologic effects of a calcium sensitizer inotrope levosimendan administered intravenously in patients with normal cardiac function 显示文摘Toivonen L Viitasalo M Sundl0erg S 2000J Cardiovasc Pharmacol2000,35,:1
15Electromyographic activity related to aerobic and anaerobic threshold in ergorneter bicycling显示文摘Viitasalo J T Luhtanen P Rahkila P 1985Acta Physiol Scand1985,124,:1
16Interrelationships between electromyographic mechanical muscle structure and reflex time measurement in man显示文摘VIITASALO J H KOMI P V 1981Acta Physiol Scand1981,111,:1
17Selective binding of collagen subtypes by integrin alpha 1I, alpha 2I, and alpha 10I domains显示文摘TULLA M PENTIKAINEN O T VIITASALO T 2001J Biol Chem2001,276,48:1
18Singnal characteristics of EMG during fatigue显示文摘Viitasalo JT Komi PV 1977Eur J Appl Physiol1977,37,:1
19Arrhythmic disorder mapped to chromosome1q42-q43 causes malignant polymorphic ventricular tachycardiain structurally normal hearts显示文摘Swan H Piippo K Viitasalo M Heikkila P Paavonen T Kainulainen K 1999J Am Coll Cardiol1999,34,7:1
20Arrhythmic disorder mapped to chromosome 1q42-q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts显示文摘Swan H Piippo K Viitasalo M 1999Am Coll Cardiol1999,34,7:1
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