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32篇 您的检索式：作者名="Wambolt"

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	题名	作者	年代	出处	被引量
1	An imbalance between glycolysis and glucose oxidation is a possible explanation for the detrimental effects of high levels of fatty acids during aerobic reperfusion of ischemic hearts显示文摘	Lopaschuk GD Wambolt RB Barr RL	1993	J Pharmacol Exp Ther1993，264，1：	1
2	Inhibition of nitric oxide synthassis protects the isolated working rabbit heart from ischemica-reperfusion injury 显示文摘	Schulz R Wambolt R	1995	Cardiovasc Res1995，30，3：	1
3	Dichloroacetete improves postischemic function of hypertrophied rat heart显示文摘	Wambolts RB Lopschuk GD Browney RW	2000	J Am Coll Cardiol2000，36，4：	1
4	Regulation of myocardial fatty acid oxidation by sustrate supply 显示文摘	Longnus S1 Wambolt RB Ban RI	2001	Am J Physiol2001，28，1：	1
5	Regulation of myocardial fatty acid oxidation by sustrate supply显示文摘	Longnus S L Wambolt R B Barr R L	2001	Am J Physiol Heart Circ Physiol2001，281，4：	1
6	Inhibition of nitric oxide synthesis protects the isolated working rabbit heart from ischemia reperfusion injury显示文摘	Schulz R Wambolt R	1995	Cardiovas Res1995，30，：	1
7	Regulation of myocardial fatty acid oxidation by sustrate supply显示文摘	Longnus SL Wambolt RB Ban RI	2001	Am J Physiol Heart Circ Physiol2001，281，4：	1
8	Inhibition of nitric oxide synthassis protects the isolated working rabbit heart from ischemica - reperfusion injury显示文摘	SCHULZ R WAMBOLT R	1995	Cardiovasc Res1995，30，3：	1
9	Dichloroacetate improves postischemic function of hypertrophied rat hearts显示文摘	Richard B Wambolts BSC Lopaschuk GD	2000	J Am Coll Cardiol2000，36，4：	1
10	An imbalance between glycolysis and glucose oxidation is a possible explanation for the detrimental effects of high levels of fatty acids during aerobic perfusion of ischemic hearts显示文摘	Lopaschuk GD Wambolt RB Barr RL	1993	J Pharmacol Exp Ther1993，264，：	1
11	An imbalance between glycolysis and glucose oxidation is a possible explanation for the detrimental effects of high levels of fatty acids during aerobic reperfusion of ischemic hearts显示文摘	Lopaschuk GD Wambolt RB Barr RL	1993	J Pharmacol ExpTherapeut1993，264，：	1
12	An imbalance between glyeolysis and glucose oxidation is a possible explanation for the detrimental effects of high levels of fatty acids during aerobic reporfusion of ischemic hearts 显示文摘	Lopaschuk G D Wambolt R B Barr R L	2003	J Pharmacol Exp Ther2003，264，1：	1
13	Inhibition of nitric oxide synthesis protects the isolated working rabbit heart from ischemia-repedusion injury显示文摘	Schulz R Wambolt R	1995	Cardiovasc Res1995，30，3：	1
14	An inbalance between glycolysis and glucose oxidation is a possible explanation for the detrimental effects of high levels of fatty acids during aerobic reperfusion of ischemic hearts显示文摘	Lopaschuk GD Wambolt RB Barr RL	1993	J Pharmacol Exp Ther1993，264，：	1
15	Pyruvate dehydrogenase and the regulation of glucose oxidation in hypertrophied rat hearts显示文摘	Lydell CP Chan A Wambolt RB	2002	Cardiovasc Res2002，53，4：	1
16	Metabolic actions of metformin in the heart can occur by AMPK-independent mechanisms显示文摘	PARSONS HL WAMBOLT RB	2008	Am J Physiol Heart Circ Physiol2008，294，6：	1
17	Regulationof myocardial fatty acid oxidation by substrate supply 显示文摘	Longnus S L Wambolt R B Barr R L	2001	Am J Physiol Heart Circ Physiol2001，281，4：	1
18	Inhibition of nitric oxide synthesis protects the isolated working rabbit heart from ischemia reperfusion injury显示文摘	Schulz R Wambolt R	1995	Cardiovas Res1995，30，2：	1
19	An imbalance betweenglycolysis and glucose oxidation is a possible explanation for thedetrimental effects of high levels of fatty acids during aerobic perfu-sion of ischemic hearts显示文摘	Lopaschuk GD Wambolt RB Barr RL	1993	J Pharmacol Exp Ther1993，264，28：	1
20	Dichloroacetate improves postischemic function of hypertrophied rat hearts显示文摘	WAMBOLT R B LOPASCHUK G D BROWNSEY R W	2000	J Am Coll Cardiol2000，36，4：	1

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