A MyD88-dependent IFNyR-CCR2 signaling circuit is required for mobilization of monocytes and host defense against systemic bacterial challenge

查看全文 作　　者：Eric M [1]Pietras;Lloyd S [1,2]Miller;Carl T [1]Johnson;Ryan M O'[1]Connell;Paul W [1,5]Dempsey;Genhong [1,2,3,4]Cheng

机构地区：[1]Department of Microbiology, Immunology and Molecular Genetics, David Geffen School of Medicine;[2]Division of Dermatology,Department of Medicine, David Geffen School of Medicine;[3]'Molecular Biology Institute, University of California, Los Angeles,Los Angeles, CA 90095, USA;[4]Jonsson Comprehensive Cancer Center, University of California, Los Angeles, Los Angeles, CA90095, USA;[5]CynvenioBiosystemsLLC,WestlakeVillage,CA91361,USA

出　　处：《Cell Research》索引2011年第21卷第7期,共12页

关键词：单核细胞宿主防御 R2信令细菌电路小鼠骨髓基因缺陷 CC

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A MyD88-dependent IFNyR-CCR2 signaling circuit is required for mobilization of monocytes and host defense against systemic bacterial challenge

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