Multifaceted roles of miR-ls in repressing the fetal gene program in the heart

查看全文 作　　者：Yusheng [1,5]Wei;Siwu [1]Peng;Meng [1]Wu;Ravi [2]Sachidanandam;Zhidong [2]Tu;Shihong [3]Zhang;Christine [4]Falce;Eric A [4]Sobie;Djamel [3]Lebeche;Yong [1]Zhao 机构地区：[1]Mindich Child Health and Development Institute, Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, Box 1040, New York, NY 10029, USA;[2]Institute for Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, New York, NY 10029, USA;[3]Cardiovascular Institute, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, New York, NY 10029, USA;[4]pharmacology and Systems Therapeutics, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, New York, NY 10029, USA;[5]College of Life Sciences, Peking University, Beijing100871, China 出　　处：《Cell Research》索引2014年第24卷第3期,共15页 关 键 词：心脏功能 相关基因 程序规范 胎儿 miRNA 细胞增殖 肝糖原 糖酵解