nterferon alpha (IFNα)-induced TRIM22 interrupts HCV 'eplication by ubiquitinating NS5A

查看全文 作　　者：Chen [1]Yang;Xinhao [2]Zhao;Dakang [5]Sun;Leilei [2]Yang;Chang [2]Chong;Yu [3]Pan;Xiumei [3]Chi;Yanhang [3]Gao;Moli [4]Wang;Xiaodong [3]Shi;Haibo [3]Sun;Juan [3]Lv;Yuanda [3]Gao;Jin [2]Zhong;Junqi [3]Niu;Bing [1,2]Sun

机构地区：[1]State Key Laboratory of Cell Biology, Institute of Biochemistry and Cell Biology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China;[2]Key Laboratory of Molecular Virology and Immunology, Institut Pasteur of Shanghai, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China;[3]Hepatology Section, First Hospital, University of Jilin, Changchun, China;[4]Infectious Diseases Department, Fourth Hospital, University of Jilin, Changchun, China;[5]Experiment Center of Clinical Medicine, Affiliated Hospital of Binzhou Medical University, Binzhou, China

出　　处：《Cellular & Molecular Immunology》索引2016年第13卷第1期,共9页

关键词：HCV 干扰素Α 诱导 IFNΑ 外周血单个核细胞泛素化中断丙型肝炎病毒

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nterferon alpha (IFNα)-induced TRIM22 interrupts HCV 'eplication by ubiquitinating NS5A

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