Endoplasmic reticulum stress: molecular mechanism and therapeutic targets

查看全文 作　　者：Xingyi [1,2,3]Chen;Chaoran [3]Shi;Meihui [1,2,3]He;Siqi [1,2,3]Xiong;Xiaobo [1,2,3]Xia

机构地区：[1]Eye Center of Xiangya Hospital,Central South University,410008 Changsha,Hunan,China;[2]Hunan Key Laboratory of Ophthalmology,Central South University,410008 Changsha,China;[3]National Clinical Research Center for Geriatric Disorders,Xiangya Hospital,Central South University,Changsha,China

出　　处：《Signal Transduction and Targeted Therapy》索引2023年第8卷第10期,共40页

基　　金：This study was financially supported by The National Key Research and Development Program of China(grant number 2020YFC2008205);The National Natural Science Foundation of China(grant numbers 82171058,81974134,and 81974137);The Key R&D Plan of Hunan Province of China(No.2020SK2076);Natural Science Foundation of Hunan Province(No.2019JJ40507 and 2023JJ30956). 摘　　要：The endoplasmic reticulum(ER)functions as a quality-control organelle for protein homeostasis,or'proteostasis'.The protein quality control systems involve ER-associated degradation,protein chaperons,and autophagy. 关键词：reticulum HOMEOSTASIS STASIS

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Endoplasmic reticulum stress: molecular mechanism and therapeutic targets

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