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35篇 您的检索式：作者名="BALASA B"

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	题名	作者	年代	出处	被引量
1	IFN-gamma is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice显示文摘	Balasa B Deng C Lee J	1997	J Exp Med1997，186，：	1
2	Interferon gamma is necessary for the genesis of actylchline receptor induced clinical experimental autoimmune myasthenia gravis in mice显示文摘	Balasa B Deng C Lee J	1997	J Exp Med1997，186，3：	1
3	CCR2 and CCR5 chemokine receptors differentially influence the development of autoimmune diabetes in the NOD mouse 显示文摘	SOLOMON M BALASA B SARVETNICK N	2010	Autoimmunity2010，43，2：	1
4	CD40 ligand -CD40 interactions are necessary for the initiation of insnlitis and diabetes in nonobese diabetic mice显示文摘	Balasa B Krabl T Patstone G	1997	J Immunnl1997，159，9：	1
5	Interferon gamma (IFN-gamma) is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice显示文摘	Balasa B Deng C Lee J	1997	J Exp Med1997，186，3：	1
6	Interferon gamma (IFN-gamma) is necessary for the genesis of aeetyleholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice 显示文摘	Balasa B Deng C Lee J	1997	J Exp Med1997，186，3：	1
7	Interferon gamma in neeessary for genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice显示文摘	Balasa B Deng C Lee J	1997	J Exp Med1997，186，3：	1
8	Interferon gamma (IFN-gamma) is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice显示文摘	Balasa B Deng C Lee J	1997	J Exp Med1997，186，3：	1
9	Vaccination with glutamic acid decarboxylase plasmid DNA protects mice from spontaneous autoimmune diabetes and B7/CD28 costimulation circumvents that protection 显示文摘	Balasa B Boehm BO Fortnagel A	2001	Clin Immunol2001，99，2：	1
10	Interferon γ is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice显示文摘	Deng C Lee J	1997	J Exp Med1997，186，：	1
11	A mechanism for IL-10-mediated diabetes in the nonobese diabetic (NOD) mouse: ICAM-1 deficiency blocks accelerated diabetes 显示文摘	Balasa B La Cava A Van Gunst K	2000	J Immunol2000，165，12：	1
12	Islet-specific expression of IL-10 promotes diabetes in nonobese diabetic mice independent of fas,pefforin, TNF receptor-l, and TNF receptor-2 molecules显示文摘	Balasa B Gunst KV Jung N	2000	J Immunol2000，165，：	1
13	Is pathogenic humoral autoimmunity a Th1 response ? Lessons from ( for ) myasthenia gravis显示文摘	Balasa B Sarventnick N	2000	Immunol Today2000，21，1：	1
14	CS1,a potential new therapeutic antibody target for the treatment of multiple myeloma显示文摘	Hsi ED Steinle R Balasa B		0，，09：	1
15	Elotuzumab enhances natural killer cell activation and myeloma cell killing through interleukin-2 and TNF-α pathways 显示文摘	Balasa B Yun R Belmar NA	2015	Cancer Immunol Immunother2015，64，1：	1
16	Interferon-gamma is necessary for the genes is of acety choine receptor induced clinical experimental auto immune myasthenia gravis in mice显示文摘	Balasa B Deng C Lee J	1997	J Exp Med1997，186，3：	1
17	Elotuzumab enhances natural killer cell activation and myeloma cell killing through interleukin-2 and TNF-a pathways 显示文摘	Balasa B Yun R Belmar NA	2015	Cancer Immunol lmmunother2015，64，1：	1
18	The Purchasing - Power Parity Doctrine: A Reappraisal 显示文摘	Balasa B	1964	Journal of Political Economy1964，，72：	1
19	CD40 ligand-CD40 interactions are necessary for the initiation of insulitis and diabetes in nonobese diabetic mice显示文摘	Balasa B Krahl T Patstone G	1997	J Immunol1997，159，9：	1
20	CS1, a potential new therapeutic antibody target for the treatment of multiple myeloma 显示文摘	Hsi E D Steinle R Balasa B	2008	Clin Cancer Res2008，14，9：	1

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