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42篇 您的检索式:作者名="Belevych AE"
    题名 作者 年代 出处 被引量
1Genistein inhibits cardiac L-type Ca(2 + ) channel activity by a tyrosine kinase-independent mechanism 显示文摘Belevych AE Warrier S Harvey RD 2002Mol Pharmacol2002,62,3:1
2MicroRNA-1 and -133 increase arrhythmogenesis in heart failure by dissociating phosphatase activity from RyR2 complex显示文摘Belevych AE Sansom SE Terentyeva R 2011PLoS One2011,6,28:1
3miR-1 overexpression enhances Ca^2+ release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKⅡ-dependent hyperphosphorylation of RyR2 显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
4MiR-1 overexpression enhances Ca2+release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56αand causing CaM KⅡ-dependent hyperphosphorylation of RyR2显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
5Muscarinic regulation of cardiac ion channels显示文摘Harvey RD Belevych AE 2003Br J Pharmacol2003,139,:1
6MiR-1 overexpression enhances Ca(2 + ) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII dependent hyperphosphorylation of RyR2显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
7miR-1 overexpression enhances Ca^2+ release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2 显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
8miR - 1 overexprcssion enhances Ca(2 + ) release and promotes cardiac arrhythmogenesis by targeting PP2Aregulatory subunit B56alpha and causing CaMKII - dependent hyperphosphorylation of RyR2 显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
9MiR-1overexpression en-hances Ca 2+release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56αand causing CaMKII-dependent hyperphosphorylation of RyR2显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
10miR-1 overexpression enhances Ca(2+) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
11Redox modification of ryanodine receptors contributes to sarcoplasmic reticulum Ca2+ leak in chronic heart failure显示文摘Terentyev D Gyorke I Belevych AE 2008Circ Res2008,103,12:1
12Genistein inhibits cardiac L- type Ca^2 + channel activity by a tyrosine kinase-independent mecha- nism显示文摘Belevych AE Warrier S Harvey RD 2002Mol Pharmacol2002,62,3:1
13Up-regulatiun of sarco- plasmic reticulum Ca (2 + ) uptake leads to cardiac hypertrophy, contractile dysfunction and early mortality in mice deficient in CASQ2 显示文摘Kalyatmsundaram A Lacombe VA Belevych AE 2013Cardiovasc Res2013,98,2:1
14miR-1 overexpression enhances Ca(2 +) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMK 1Ⅱ-dependent hyperphosphorylation of RyR2显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,:1
15miR- 1 overexpression enhances Ca2+ release and promotes cardiac ar- rhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2 显示文摘Terentyev D Belevych AE Terentyeva R 2009CircRes2009,104,4:1
16MicroRNA 1and 133 increase arrhythmogenesis in heart failure by dissociatingphosphatase activity from RyR2 complex显示文摘Belevych AE Sansom SE Terentyeva R 2011PLoS One2011,6,28:1
17MiR-1 overexpression enhances Ca2+ release and promotescardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
18MiR-1 overexpression enhances Ca (2 + ) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2显示文摘TERENTYEV D BELEVYCH AE TERENTYEVA R 2009Circ Res2009,104,4:1
19Genistein inhibits cardiac L-type Ca^2+ channel activity by a tyrosine kinase-independent mechanism显示文摘Belevych AE Warrier S Harvey RD 2002Mol Pharmacol2002,62,3:1
20miR-1 overexpressi- on enhances Ca (2+) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII- dependent hyperphosphorylation ofRyR2显示文摘Terentyev D Belevych AE Terentyeva R 2009Circ Res2009,104,4:1
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