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37篇 您的检索式：作者名="Breitschopf"

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	题名	作者	年代	出处	被引量
1	The peroxisome proliferator-activated receptor-α (PPAR-α) agonist, AVE8134, attenuates the progression of heart failure and increases survival in rats显示文摘	Wolfgang LINZ Paulus WOHLFART Manuel BAADE Kristin BREITSCHOPF EugenFALK Hans-Ludwig SCHAFER Martin GERL Werner KRAMER Hartmut RUTTEN	2009	Acta Pharmacologica Sinica2009，30，7：	3
2	Posttranslational modification of Bcl-2 facilitates its proteasome-dependent degradation: molecular characterization of the involved signaling pathway显示文摘	Breitschopf K Haendeler J Malchow P	2000	Mol Cell Biol2000，20，：	1
3	Cell death in Alzheimer's disease evaluated by DNA fragmentation in situ显示文摘	Lassmann H Bancher C Breitschopf H	1995	Acta Neuropathol1995，89，1：	1
4	Pro - atherogenic factors induce telomerase inactivation in endothelial cells through an Akt - dependent mechanism 显示文摘	Breitschopf K Zeiher AM Dimmeler S	2001	FEBS Lett2001，493，1：	1
5	Pro-atherogenic factors induce telomerase inactivation in endothelial cells through an Akt-dependent mechanism 显示文摘	Breitschopf K Zeiher AM Dimmeler S	2001	FEBS Lett2001，493，1：	1
6	Mechanisms of cell death in Alzheimer disease显示文摘	Bancher C Lassmann H Breitschopf H	1997	J Neural Transm Suppl1997，50，：	1
7	Pro-atherogenic factors induce telomerase inactivation in endothelial cells through an Akt-dependent mechanism显示文摘	Zeiher AM Dimmeler S	2001	FEBS Lett2001，493，：	1
8	Nitric oxide activates telomerase and delays endothelial cell senescence 显示文摘	Vasa M Breitschopf K Zeiher AM	2000	Circ Res2000，87，7：	1
9	Posttranslational modification of Bcl-2 facilitates its proteasome-dependent degradation: molecular characterization of the involved signaling pathway 显示文摘	Breitschopf K Haendeler J Malchow P	2000	Mol Cell Biol2000，20，5：	1
10	Differentiation between cellular apoptosis and necrosis by the combined use of in situ tailing and nick translation techniques显示文摘	Gold R Schmied M Giegerich G Breitschopf H	1994	Lab Invest1994，71，：	1
11	Nitric oxide activates telomerase and delays endothelial cell senescence 显示文摘	Vasa M Breitschopf K Zeiher A M	2000	Circ Res2000，87，7：	1
12	Dephosphoryla- tion targets Bcl-2 for ubiquitin-dependent degradation: a link between the apoptosome and the proteasome pathway显示文摘	Dimmeler S Breitschopf K Haendeler J	1999	J ExpMed1999，189，11：	1
13	A novel site for ubiq- uitination: the N-terminal residue, and not internal lysines of MyoD, is essential for conjugation and degradation of the protein 显示文摘	BREITSCHOPF K	1998	EMBO1998，17，：	1
14	Bcl-2 for ubiquitin dependent degradation: a link between the apoptosome and the proteasome pathway显示文摘	Dimmeler S Breitschopf Dephosphorylation targets K Haendeler J	1999	ExpMed1999，189，11：	1
15	Apoptosis of T lymphocyte in experimental autoimmune encephalomyeli-tis: Evidence for programmed cell death as a mechanism to control inflammation in the brain显示文摘	Breitschopf H Gold R	1993	Am J Pathol1993，143，2：	1
16	Innovation Dynamics in OECD Countries: Challenges for German Enterprises显示文摘	Barbara Breitschopf Hariolf Grupp	2004	International Economics and Economic Policy (-)2004，，2：	1
17	Nitric oxide activates telomerase and delays endothelial cell senescence显示文摘	Vasa M Breitschopf K Zeiher A M	2000	Circ Res2000，87，：	1
18	Human dendritic cell subsets display distinct interactions with the pathogenic mould Aspergillus fumigatus 显示文摘	LOTHER J BREITSCHOPF T KRAPPMANN S	2014	Int J Med Microbiol2014，304，8：	1
19	Cellular localiza tion of insulin like growth factor Ⅱ mRNA in the human fetus and the placenta: detection with a digoxigenin labeled cRNA probe and immunocytochemistry 显示文摘	Birnbacher R Amann G Breitschopf H	1998	Pediatr Res1998，43，5：	1
20	Nitric oxide inhibits caspase-3 by S-nitrosation in vivo显示文摘	Rossig L Fichtlscherer B Breitschopf K	1999	J Biol Chem1999，274，11：	1

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