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| 1 | Differential Scanning Calorimetry as a Tool to Investigate the Transfer of Anticancer Drugs to Biomembrane Model显示文摘 | Maria Grazia Sarpietro Maria Lorena Accolla Christian Celia Alessandro Grattoni Francesco Castelli Massimo Fresta Mauro Ferrari Donatella Paolino | 2013 | Current Drug Targets2013，，9： | 1 |
| 2 | Simplified Nonlinear Analysis for Settlement Prediction of Pile Groups显示文摘 | FRANCESCO CASTELLI MICHELE MAUGERI | 2002 | Journal of Geotechnical and Geoenvironmental Engineering2002，128，1： | 1 |
| 3 | Simplified nonlinear analysis for settlement prediction of pile groups 显示文摘 | FRANCESCO CASTELLI MICHELE MAUGERI | 2002 | Journal of Geotechnical and Geoenvironmental Engineering2002，128，1： | 1 |
| 4 | Effects of mismatched parameters in MRAS sensorless doubly fed induction machine drives显示文摘 | Maria Stefania Carmeli Francesco Castelli Dezza Matteo Iacchetti | 2010 | IEEE Transactions on Power Electronics2010，24，11： | 1 |
| 5 | Attitudes and Practices in Travel -related Infectious Diseases:The European Airport Survey 显示文摘 | FCoen Van herck Francesco Castelli PieiTe Van Damme Knowledge | 2004 | Journal of Travel Medicine2004，99，1： | 1 |
| 6 | Simplified nonlinear analysis for se ttlement predictionof pile groups显示文摘 | Francesco Castelli Michele Maugeri | 2002 | Journal of Geotechnical and Geoenvironmen tal Engineering2002，，1： | 1 |
| 7 | Knowledge, Attitudes and Practices in Travel-related Infectious Diseases: The European Airport Survey 显示文摘 | Koen Van herck Francesco Castelli Pierre Van Damne | 2004 | Journal of Travel Medicine2004，99，1： | 1 |
| 8 | Simplified nonlinear analysis for settlement prediction of pile groups显示文摘 | Francesco Castelli Michele Maugeri | 2002 | Journal of Geotechnical and Geoenvironmental Engineering2002，128，1： | 1 |
| 9 | Knowledge, Attitudes and Practices in Travel-related Infectious Diseases:The European Airport Survey显示文摘 | Koen Van Herck Francesco Castelli | 2004 | Journal of Travel Medicine2004，99，1： | 1 |
| 10 | Simplified Nonlinear Analysis for Settlement Prediction of Pile Groups显示文摘 | Francesco Castelli Michele Maugeri | 2002 | Journal of Geotechnical and Geoenvironmental Engineering2002，，1： | 1 |
| 11 | Conjugation of squalene to acyclovir improves the affinity for biomembrane models显示文摘 | Maria Grazia Sarpietro Dorotea Micieli Flavio Rocco Maurizio Ceruti Francesco Castelli | 2009 | International Journal of Pharmaceutics2009，，1： | 1 |
| 12 | Enhancement of gemcitabine affinity for biomembranes by conjugation with squalene: Differential scanning calorimetry and Langmuir–Blodgett studies using biomembrane models显示文摘 | Francesco Castelli Maria Grazia Sarpietro Dorotea Micieli Barbara Stella Flavio Rocco Luigi Cattel | 2007 | Journal of Colloid And Interface Science2007，，1： | 1 |
| 13 | Therapeutic advances in neural regeneration for Huntington’s disease显示文摘Huntington’s disease is a neurodegenerative disease caused by the expansion mutation of a cytosine-adenine-guanine triplet in the exon 1 of the HTT gene which is responsible for the production of the huntingtin (Htt) protein. In physiological conditions, Htt is involved in many cellular processes such as cell signaling, transcriptional regulation, energy metabolism regulation, DNA maintenance, axonal trafficking, and antiapoptotic activity. When the genetic alteration is present, the production of a mutant version of Htt (mHtt) occurs, which is characterized by a plethora of pathogenic activities that, finally, lead to cell death. Among all the cells in which mHtt exerts its dangerous activity, the GABAergic Medium Spiny Neurons seem to be the most affected by the mHtt-induced excitotoxicity both in the cortex and in the striatum. However, as the neurodegeneration proceeds ahead the neuronal loss grows also in other brain areas such as the cerebellum, hypothalamus, thalamus, subthalamic nucleus, globus pallidus, and substantia nigra, determining the variety of symptoms that characterize Huntington’s disease. From a clinical point of view, Huntington’s disease is characterized by a wide spectrum of symptoms spanning from motor impairment to cognitive disorders and dementia. Huntington’s disease shows a prevalence of around 3.92 cases every 100,000 worldwide and an incidence of 0.48 new cases every 100,000/year. To date, there is no available cure for Huntington’s disease. Several treatments have been developed so far, aiming to reduce the severity of one or more symptoms to slow down the inexorable decline caused by the disease. In this context, the search for reliable strategies to target the different aspects of Huntington’s disease become of the utmost interest. In recent years, a variety of studies demonstrated the detrimental role of neuronal loss in Huntington’s disease condition highlighting how the replacement of lost cells would be a reasonable strategy to overcome the neurodegeneration. In this view, numerous have been the attempts in several preclinical models of Huntington’s disease to evaluate the feasibility of invasive and non-invasive approaches. Thus, the aim of this review is to offer an overview of the most appealing approaches spanning from stem cell-based cell therapy to extracellular vesicles such as exosomes in light of promoting neurogenesis, discussing the results obtained so far, their limits and the future perspectives regarding the neural regeneration in the context of Huntington’s disease. | Francesco D’Egidio Vanessa Castelli Giorgia Lombardozzi Fabrizio Ammannito Annamaria Cimini Michele d’Angelo | 2024 | Neural Regeneration Research2024，19，9： | 0 |
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