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15篇 您的检索式:作者名="Funatake C"
    题名 作者 年代 出处 被引量
1Blocking OX- 40/OX- 40 ligand interaction in vitro and in vivo leads to decreased T cell function and amelioration of experimental allergic encephalomyelitis显示文摘Weinberg A D Wegmann K W Funatake C 1999J Immunol1999,162,3:1
2Cutting edge: activation of the aryl hydrocarbon receptor by 2,3,7,8- tetrachlorodibenzo-p-dioxin generates a population of CD4^+ CD25^+ cells with characteristics of regulatory T cells 显示文摘Funatake C J Marshall NB Steppan LB 2005J Immunol2005,175,7:1
3Blocking OX-10/OX-10 ligand interaction in vitro and in vivo leads to decreased T cell function and amelioration of experimental allergic encephalomyelitis显示文摘Weinberg AD Wegmann KW Funatake C 1999J Immunol1999,162,3:1
4Early consequences of 2, 3, 7, 8- tetrachlorodibenzo-p-dioxin exposure on the activation and survival of antigen-specific T cells显示文摘Funatake C J Dearstyne EA Steppan LB 2004Toxicol Sci2004,82,1:1
5Cutting edge: activa- tion of the aryl hydrocarbon receptor by 2, 3, 7, 8-tetrachlorodibenzo- p-dioxin generates a population of CIM ^+ CD25 ^+ cells with character- istics of regulatory T cells显示文摘Funatake C J Marshall NB Steppan LB 2005J Immunol2005,175,7:1
6Blocking OX-40 and OX-40 ligand interaction in vitro and in vivo leads to decreased T cell function and amelioration of experimen- tal allergic encephalomyelitis显示文摘Weinberg AD Wegmann KW Funatake C 1999The Journal of Immunology1999,162,3:1
7Early consequences of 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure on the activation and survival of antigen-specific T cells显示文摘Funatake C J Dearstyne E A Steppan L B 2004Toxicol Sci2004,82,1:1
8Cutting edge : activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo- p-dioxin generates a population of CD4 + CD25 + cells with characteristics of regulatory T cells 显示文摘Funatake C J Marshall N B Steppan L B 2005J Immunol2005,175,7:1
92,3,7,8-tetrachlorodibenzo-p-dioxin alters the differentiation of alloreactive CD8 + T cells toward a regulatory T cell phenotype by a mechanism that is dependent on aryl hydrocarbon receptor in CD4 + T cells 显示文摘Funatake C J Marshall N B Kerkvliet N I 2008J Immunotoxicol2008,5,1:1
10Blocking OX- 40/OX-40 ligand interaction in vitro and in vivo leads to decreased T-cell function and amelioration of experimental allergic en- cephalomyelitis显示文摘Weinberg AD Wegmann KW Funatake C 1999JImmunol1999,162,3:1
11Blocking OX-40/OX-40 ligand interaction in vitro and in vivo leads to decreased T cell function and amelioration of experimental allergic encephalomyelitis显示文摘Weinberg AD Wegmann KW Funatake C 1999J Immunol1999,162,3:1
12Blocking OX-40/OX-40 ligand interaction in vitro and in vivo leads to decreased T cell function and amelioration of experimental allergic encephalomyelitis显示文摘Weinberg AD Wegmann KW Funatake C 1999J Immunol1999,162,3:1
13Blocking OX-10/OX-40 ligand interaction in vitro and in vivo leads to decreased T cell function and amelioration of experimental allergic encephalomyelitis显示文摘Weinberg AD Wengmann KW Funatake C 1999J Immunol1999,162,3:1
14Enhancement of antitumorimmunity by engaging the OX40 receptor (CD134) in vivo显示文摘 Lemon M Funatake C 1998AACRMeeting/New Orleans Abstract1998,3610,:1
15Expression of constitutively-active aryl hydrocarbon receptor in T-cells enhances the down-regulation of CD62L, but does not alter expression of CD25 or suppress the allogeneic CTL response显示文摘Funatake C J Ao K Suzuki T 2009J Immunotoxicol2009,6,3:1
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