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17篇 您的检索式:作者名="Haelst PL"
    题名 作者 年代 出处 被引量
1No long-lasting or intermittent mast cell activation in acute coronary syndromes显示文摘Van Haelst PL Timmer JR Grijns HJGM Kauffman HF Gans ROB van Doormaal JJ 2001Internat J Cardiol2001,78,:1
2The effect of aggressive versus conventional lipid-lowering therapy on markers of inflammatory and oxidative stress 显示文摘Mulder D J van Haelst PL Wobbes MH 2007Cardiovasc Drugs Ther2007,21,2:1
3Correlates of endothelial function and their relationship with inflammation in patients with familial hypercholestelolaemia显示文摘Haelst PL van Doormaal JJ van Asselbergs FW 2003Clin Sci2003,104,:1
4Circulating monocytes in patients with acute coronary syndromes lack sufficient interleukin - 10 production after lipopolysaccharide stimulation 显示文摘Van Haelst PL Tervaert JW Bijzet J 2004Clin Exp Immunol2004,138,2:1
5Circulating monocytes in patients with acute coronary syndromes lack sufficient interleukin-10 production after lipopolysaccharide stimulation显示文摘Van Haelst PL Tervaert JW Bijet J 0,,02:1
6Antineutro- phil cytoplasmatic antibodies in patients with premature ather- osclerosis: prevalence and association with risk factors显示文摘Haelst PL Asselbergs FW Doormaal JJ 2002J Intern Med2002,251,1:1
7Circulating monocytes in patients with acuter coronary syndromes lack sufficient interleukin-10 production after lipopolysaccharide stimulation 显示文摘Van Haelst PL Tervaert JW Bijzet J 2004Clin Exp Immunol2004,138,2:1
8The effect of aggressive versus conventional lipid-lowering therapy on markers of inflammatory and oxidative stress显示文摘 van Haelst PL Wobbes MH 2007Cardiovasc Drugs Ther2007,21,:1
9The effect of aggressive versus conventional lipid-lowering therapy on markers of inflammatory and oxidative stress 显示文摘Mulder D J van Haelst PL Wobbes MH 2007Cardiovasc Drugs Ther2007,21,2:1
10Secondary prevention with fluvastatin decreases levels of adhesion molecules,neopterin and C-reactive protein显示文摘 van-Doormaal JJ May JF 2001Eur J Intern Med2001,12,6:1
11Circulation monocytes in patients with acute coronary syndromes lack sufficient interleukin-10 production after lipopolysaccharide stimulation显示文摘Van Haelst PL Tervaert JW Bijet J 2004Clin Exp Immunol2004,138,2:1
12Circulating monocytes in patients with acute coronary syndromes lack sufficient interleukin-10 production after lipopolysaccharide stimulation显示文摘Van Haelst PL Tervaert JW Bijet J 2004Clin Exp Immunol2004,138,2:1
13Endothelial dysfunction in patients withcoronary artery disease: a comparison of three frequently reported tests 显示文摘Monnink SH van Haelst PL van Boven A J 2002J Invest Med2002,50,1:1
14Circulating monocytes in patients with acute coronary syndromes lack sufficient interleukin -10 production after lipopolysaccharide stimulation 显示文摘Van Haelst PL Tervaert J W Bijzet J 2004Clin Exp Immunol2004,138,2:1
15Usefulness of elevated neopterin and C-reactive protein levels in predicting cardiovascular events in patients with non-Q-wave myocardial infarction显示文摘Van Haelst PL Liem A van Boven A J 2003Am J C- ardiol2003,92,10:1
16Circulating monocytes in patients with acute coronary syndromes lack sufficient interleukin-10 production after lipopolysaccharide stimulation显示文摘van Haelst PL Tervaert JW Bijzet J etal 2004Clin Explmmunol2004,,:1
17Long term angiotensin converting enzymeinhibition in patients after coronary artery bypass grafting reduces levels of soluble intercellular cell adhesion molecule-1显示文摘van Haelst PL Tervaert JW van Geel PP 2003Eur J Vasc Endovase Surg2003,26,:1
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