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| 1 | Lupus pathogenesis: role of IgE autoantibodies显示文摘 | Jagadeesh Bayry | 2016 | Cell Research2016，26，3： | 3 |
| 2 | Th17 cells, pathogenic or not? TGF-β3 imposes the embargo显示文摘 | Meenu Sharma | 2013 | Cellular & Molecular Immunology2013，10，2： | 2 |
| 3 | mpaired regulatory T cell function in autoimmune Jiseases: are microRNAs the culprits?显示文摘 | Varun K Sharma Srini V Kaveri Jagadeesh Bayry | 2016 | Cellular & Molecular Immunology2016，13，2： | 2 |
| 4 | Emergence of a nephropathogenic avian infectious bronchitis virus with a novel genotype in India显示文摘 | JAGADEESH BAYRY MALLIKARJUN S PRASHANT K | 2005 | J Clin Microbiol2005，43，2： | 1 |
| 5 | Intravenous immunoglobulin-mediated expansion of regulatory T cells in autoimmune patients is associated with increased prostaglandin E2 levels in the circulation显示文摘 | Mohan S Maddur Jamma Trinath Magalie Rabin Francis Bolgert Moneger Guy Jean-Michel Vallat Laurent Magy Kithiganahalli N Balaji Srini V Kaveri Jagadeesh Bayry | 2015 | Cellular & Molecular Immunology2015，12，5： | 1 |
| 6 | Surveillance of Antigen-Presenting Cells by CD4+CD25+ Regulatory T Cells in Autoimmunity显示文摘 | Sébastien André David F. Tough Sébastien Lacroix-Desmazes Srini V. Kaveri Jagadeesh Bayry | 2009 | The American Journal of Pathology2009，，： | 1 |
| 7 | Intravenous immunoglobulin for infectious diseases: tailor - made or universal显示文摘 | Jagadeesh Bayry Sebastien Lacmix - Desmazes Michel D Kazatchkine | 2003 | The Journal of Infectious Diseases2003，188，： | 1 |
| 8 | Emergence of a Nephropathogenic Avian Infectious Bronchitis Virus with a Novel Genotype in India显示文摘 | Jagadeesh Bayry Mallikarjun S Goudar Prashant K Nighot | 2004 | Journal of clinical microbiology2004，43，： | 1 |
| 9 | Cutting edge: Human CD4+ CD25+ T cells restrain the maturation and antigen-presenting function of dendritic cells 显示文摘 | Jagadeesh Bayry Sebastien Lacroix-Desmazes | 2004 | J Immunol2004，172，8： | 1 |
| 10 | Regulatory T cells as adjuvant target for enhancing the viral disease vaccine efficacy显示文摘 | Jagadeesh Bayry | 2014 | Indian Journal of Virology2014，，1： | 1 |
| 11 | Restoration of established systemic inflammation and autoimmunity by Foxp3^(+) regulatory T cells显示文摘Immune tolerance ensures the disease-free status of an individual by preventing the appearance of pathological conditions,such as autoimmune and inflammatory diseases.Among the various players implicated in the maintenance of immune tolerance,CD4^(+)CD25^(+)regulatory T(Treg)cells that express the X-linked transcription factor Forkhead box P3(Foxp3)play a major role.FoxP3 governs the functions of Treg cells.In fact,a deficiency in Treg cells due to mutations in FoxP3 leads to fatal autoimmune and inflammatory conditions in humans called immunodysregulation polyendocrinopathy enteropathy x-linked(IPEX)syndrome.Similar observations have also been made in mice,in which FoxP3 deficiency leads to autoimmune pathology and death early in life.In addition,various mutations in Treg-associated immunoregulatory molecules lead to inflammatory conditions[1]. | Varun Kumar Sharma Jagadeesh Bayry | 2022 | Cellular & Molecular Immunology2022，19，2： | 0 |
| 12 | Induction of human dendritic cell maturation by naïve and memory B-cell subsets requires different activation stimuli显示文摘Dendritic cells(DCs)are the master regulators of the immune response and engage in constant cross-talk with other immune cells to initiate an appropriate immune response.The outcome of cross-talk with various immune cells towards DC functions has been shown to be dependent on the particular subset of cells and their activation stimuli.Here we show that for human B cells to induce maturation of DCs with an enhanced T-cell stimulatory capacity,B-cell receptor(BCR)signaling is sufficient for memory B-cells,whereas additional CD40 signaling is required for naïve B cells to induce complete activation of DCs. | Mohan S.Maddur Srini V.Kaveri Jagadeesh Bayry | 2018 | Cellular & Molecular Immunology2018，15，12： | 0 |
| 13 | Gasdermin D as a cellular switch to orientate immune responses via IL-33 or IL-1β显示文摘Interleukin(IL)-33 is a nuclear cytokine in the IL-1 family that is constitutively expressed in the epithelial cells of environmentally exposed tissues(skin,gastrointestinal tract and lungs)and endothelial cells.IL-33 is involved in type 2 innate immunity via the activation of eosinophils,basophils,mast cells,group 2 innate lymphoid cells and macrophages through its receptor ST2(also called IL1RL1)[1].Due to the absence of a secretory signal sequence,IL-33 was thought to be passively released during cell necrosis,physical stress or tissue damage and was accordingly considered an alarmin[2]. | Camille Chauvin Sruthi Vijaya Retnakumar Jagadeesh Bayry | 2023 | Cellular & Molecular Immunology2023，20，1： | 0 |
| 14 | Basophils orchestrate kidney fibrosis显示文摘Basophils,though representing a minor population of leukocytes,have diverse roles in various pathophysiologies.A recent report provides an additional evidence on the pathogenic role of basophils in promoting kidney fibrosis by secreting IL-6 and promoting the Th17 cell differentiation.Basophils are rare granulocytes that participate in T helper(Th)type 2 immunity,principally through the secretion of interleukins IL-4 and IL-13.Besides their role in the defense against helminth infections,basophils play a major role in the pathogenesis of various allergic inflammatory diseases,autoimmune diseases,and cancer.1 Basophils express a diverse array of receptors,and hence could sense signals from various sources including cytokines,tolllike receptor agonists,and allergens,and subsequently undergo rapid activation and release inflammatory mediators.In addition to IL-4 and IL-13,basophils produce histamine,prostaglandins,leukotrienes,IL-6 and chemokines in response to the stimuli.All these inflammatory mediators are potential contributors of basophil-mediated pathogenesis. | Camille Chauvin Jagadeesh Bayry | 2022 | Cell Research2022，32，8： | 0 |
| 15 | For antigen-specific effector or Foxp3^(+) regulatory T cell fate, cyclin-dependent kinases hold the trump card显示文摘Forkhead box p3^(+)(Foxp3^(+))regulatory T cells(Tregs)are indispensable for immune homeostasis and for maintaining immune tolerance.Several studies have confirmed that injection of a T cell population depleted of Tregs causes autoimmunity,rejection of grafts and inflammatory disorders,whereas reconstitution with Tregs inhibits these pathogenic processes.Over the last two decades,intense efforts have been made to identify Treg subsets,Treg differentiation processes,and the molecular signatures and regulators that determine Treg lineage specificity and stability.1–6 Several lines of evidence clearly demonstrate that Foxp3^(+)Tregs do not constitute a homogeneous population,and various subsets of Tregs,such as thymic Tregs(tTregs),in vitro-generated Tregs(iTregs),and peripherally induced Tregs(pTregs),have been identified.5 In addition to Foxp3,epigenetic factors and metabolic processes play key roles in maintaining Treg identity and function,and mediate the switch between effector T cells and Tregs. | Srinivasa Reddy Bonam Jagadeesh Bayry | 2020 | Cellular & Molecular Immunology2020，17，4： | 0 |
| 16 | Anti-IgE IgG autoantibodies isolated from therapeutic normal IgG intravenous immunoglobulin induce basophil activation显示文摘Basophils are rare granulocytes.Despite representing only~0.5%of all leukocytes,basophils have several important physiological functions.1,2 Although basophils lack the classic features of professional antigen-presenting cells,3–7 through the secretion of cytokines,they orient the immune response by polarizing Th2 differentiation and supporting B-cell differentiation and class switching.Basophils are also critical for mediating protection against helminth infection.1,2,8,9 Basophils receive activation signals from diverse sources.It is well recognized that cytokines such as IL-3,granulocyte–macrophage colony-stimulating factor(GM-CSF),thymic stromal lymphopoietin(TSLP)and IL-33;various toll-like receptor ligands;allergen-bound IgE provide activation signals to basophils and induce the release of inflammatory mediators.10–15 In addition,several reports have also demonstrated the existence of anti-IgE autoantibodies that possess the capacity to induce basophil activation in patients with chronic spontaneous urticaria(CSU),atopic or non-atopic asthma or autoimmune disease.16–21 However,isolation and functional exploration of such anti-IgE IgG autoantibodies from either healthy donors or patients have not been attempted yet.By using a pooled normal IgG preparation from healthy donors,specifically intravenous immunoglobulin G(IVIG)22 that represents the complete IgG repertoire of a normal individual,we attempted to address this outstanding question in the field. | Caroline Galeotti Anupama Karnam Jordan D.Dimitrov Alain Chevailler Srini V.Kaveri Jagadeesh Bayry | 2020 | Cellular & Molecular Immunology2020，17，4： | 0 |
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