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| 1 | Displacements and stresses resulting from contact of a steel belted radial tire with a flat surface显示文摘 | De Eskinazi Jozef Yang T Y Soedel Werner | 1978 | Tire Science and Technology1978，，： | 1 |
| 2 | Pesticide Residue Analyses in Plant Material by Chromatographic Methods: Clean-up Procedures and Selective Detectors显示文摘 | JOZEF T STEFAN H | 1996 | Journal of Chromatography A1996，754，： | 1 |
| 3 | GFP-FABD2 fusion construct allows in vivo visualization of the dynamic actin cytoskeleton in all cells of Arabidopsis seedlings显示文摘 | Voigt B Antonius C J T Jozef Samaja | 2005 | Eur J Cell Biol2005，84，： | 1 |
| 4 | Morphological and functional alterations in endothelium, smooth muscle, and nerve fibers in rabbit aorta after storage at 4 ℃ 显示文摘 | Frantisek K Jozef T Jana S | 1993 | Cryobiology1993，30，： | 1 |
| 5 | Base-free efficient palladium catalyst of Heck reaction in molten tetrabutylammonium bromide 显示文摘 | Iweta P R Anna M T Jozef J Z | 2006 | J Mol Catal A Chem2006，257，12： | 1 |
| 6 | Hazards of heavy mental contamination at L'ubietova Cu-deposit (Slovakia)显示文摘 | PETER A INGRID T JOZEF K | 2012 | Procedia Environmental Sciences2012，，14： | 1 |
| 7 | Angiotensin Ⅱ type 2 receptor blockade amplifies the early signals of cardiac growth response to angiotensin Ⅱ in hypertrophied hearts显示文摘 | Jozef B Ellen OW Minori T | 1999 | Circulation1999，99，1： | 1 |
| 8 | Base free efficient palladium catalyst of Heck reaction in mohen tctrabulylammo nium bromide显示文摘 | IWETA P R ANNA M T JOZEF J Z | 2006 | J Mol Catal A Chem2006，257，12： | 1 |
| 9 | Base-free efficient palladium catalyst of Heck reaction in molten tetrabutylammonium bromide显示文摘 | Iweta P R Anna M T Jozef J Z | 2006 | J Mol Catal A2006，257，12： | 1 |
| 10 | Pesticide Residue Analyses in Plant Material by Chromatographic Methods : Clean - up Procedures and Selective De- tectors显示文摘 | JOZEF T STEFAN H | 1996 | J Chromatogr A1996，754，： | 1 |
| 11 | MULAN: a java library for multi-label learning 显示文摘 | Grigorios T Eleftherios S Jozef V | 2011 | J Mach Learn Res2011，12，7： | 1 |
| 12 | Understanding the source of muhifractality in financial markets 显示文摘 | Barunik Jozef Aste Tomaso Di Matteo T&Liu Ruipeng | 2012 | Physica A Statistical Mechanics and its Applications2012，88，17： | 1 |
| 13 | Magnetic properties and heating effect in bacterial magnetic nanoparticles显示文摘 | Milan T Anezka D Jozef K | 2009 | J Magn Magn Mater2009，321，： | 1 |
| 14 | Base-free efficient palla- dium catalyst of Heek reaction in molten tetrabutylammo- nium bromide 显示文摘 | Iweta P R Anna M T Jozef J Z | 2006 | J Mol Catal A Chem2006，257，12： | 1 |
| 15 | Rilmenidine prevents blood pressure increase in rats with compromised nitric oxide production显示文摘AIM: To search tools of high blood pressure in the model of nitric oxide (NO)-defective hypertension, and the study focused on the effect of rilmenidine, agonist of imidazoline receptors, which was suggested to modulate central sympathetic outflow. METHODS: Three experimental groups, each consisting of 7 rats, were used: (I) rats with inhibition of NO synthase (NOS) by NG-nitro-L-arginine methyl ester (L-NAME) 40 mg·kg-1·d-1 for 4 weeks in drinking water, (II) rats with inhibited NOS as in group I , plus agonist of imidazoline receptors rilmenidine 3 mg·kg-1·d-1 for 4weeks by gavage, and(III) control rats. Systolic blood pressure wasmeasured weekly noninvasively. At the end of experiment aortic ring isometric tension was followed, NOS expression (aorta, left ventricle), and NOS activity (left ventricle and brain) were determined. RESULTS: In the group I systolic blood pressure in- creased significantly, aortic ring relaxation to acetylcholine was significantly attenuated. Rilmenidine administered simultaneously with L-NAME (group II) prevented the increase of blood pressure which did not differ significantly from control values; aortic ring relaxation to acetylcholine did not differ from control. No change in NOS expres- sion (aorta and left ventricle) was found in groups I and II. Significant decline in NOS activity (left ventricle and brain) wasfound in groups I and II. CONCLUSION: Rilmenidinehas aremarkable role inNO-defective hypertension, possibly by inhibiting central sympathetic outflow and by affecting receptors in vascular smooth muscle also. The prime cause of hypertension in this experimental model - the compromised production of NO due to inhibition of NOS - was not affected by rilmenidine. | Mria GEROV Jozef TRK Ol’ga PECHV Jana MATUKOV | 2004 | Acta Pharmacologica Sinica2004，25，12： | 0 |
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