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7篇 您的检索式：作者名="KITAH"

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		题名	作者	年代	出处	被引量
	1	The eosinophil a eytokine-producing cell 显示文摘	KitaH	2007	J Allergy Clin Immunol2007，97，4：	1
	2	A hybrid EP and SQP for dynamic economic dispatch with nonsmooth fuel cost function显示文摘	PARK PA KITAH TANAKAE	2002	IEEE Transaction Power Systems2002，17，2：	1
	3	Clinical outcomes of double-balloon endoscopy for the diagnosis and treatmentof small intestinaldiseases 显示文摘	YamamotoH KitaH Sunada K	2004	Clin Gastro- enterol Hepatol2004，2，11：	1
	4	Protective role of hydroxysteroid sulfotransferase in lithocholic acid—in—duced liver toxicity显示文摘Supplement of 1% lithocholic acid (LCA) in the diet for 5-9d resulted in elevated levels of the marker for liver damage AST and ALP activities in both FXR-null and wild-type female mice.The levels were clearly higher in wild-type mice than in FXR-null mice,in spite of the diminished expression of a bile salt export pump(Bsep) in the latter.Consistent with liver toxicity marker activities,serum and liver levels of bile acide,particularly LCA and tauro LCA,were clearly higher in wild-type mice than in FXR-null mice after 1% LCA supplement.Marked increases in hepatic sulfating activity for LCA(5.5-fold) and hydroxysteroid sulfotransferase St2a(5.8-fold) were detected in liver of FXR-null mice.Liver St2a content was inversely correlated with levels of ALP.In contrast,microsomal LCA 6-hydroxylation was not increased and in fact lower in FXR-null mice compared in wild-type mice.Clear decreases in mRNA encoding Ntcp,Oatpl and Lst-1 transporters function in bild acid import were detected in LCA fed mice.These transporter levels are higher in FXR-null mice than in wild-type mice after 1% LCA supplement.No obvious changes were detected in the Mrp2,Mrp3 and Mrp4 mRNAs.These results indicate that hydroxysteroid sulfotransferase is reuired for protection against LCA-induced liver damage.	YamaY KitaH	2002	中国药理学与毒理学杂志2002，16，6：	1
	5	Cellular immune re-sponse in primary biliary cirrhosis显示文摘	KitaH Imawari M Gershwin M E	2004	Hepatol Res2004，28，1：	1
	6	Quantita- tion and phenotypic analysis of natural killer T cells in primary biliary cirrhosis using a human CD1 d tetramer显示文摘	KITAH NAIDENKO OV KRQNENBERG M etal	2002	Gastroen- terology2002，123，：	1
	7	An evolutionary programming to the unit commitment problem显示文摘	JusteKA KitaH TanakaEetal	1999	IEEE Transaction on power systems1999，14，4：	1

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