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1Competition between fusion-evaporation and multifragmentation in central collisions in ^(58)Ni+^(48)Ca at 25A MeV显示文摘The experimental data concerning the58Ni+48Ca reaction at Elab(Ni)=25A MeV,collected by using the CHIMERA 4π device,have been analyzed in order to investigate the competition among different reaction mechanisms for central collisions in the Fermi energy domain.As a main criterion for centrality selection we have chosen the flow angle(flow) method,making an event-by-event analysis that considers the shape of events,as it is determined by the eigenvectors of the experimental kinetic-energy tensor.For the selected central events(flow >60°) some global variables,good to characterize the pattern of central collisions have been constructed.The main features of the reaction products were explored by using different constraints on some of the relevant observables,like mass and velocity distributions and their correlations.Much emphasis was devoted,for central collisions,to the competition between fusion-evaporation processes with subsequent identification of a heavy residue and a possible multifragmentation mechanism of a well defined(if any) transient nuclear system.Dynamical evolution of the system and pre-equilibrium emission were taken into account by simulating the reactions in the framework of transport theories.Different approaches have been envisaged(dynamical stochastic BNV calculations + sequential SIMON code,QMD,CoMD,etc.).Preliminary comparison of the experimental data with BNV calculations shows reasonable agreement with the assumption of sequential multifragmentation emission in the mass region of IMFs close to the heavy residues.Possible deviations from sequential processes were found for those IMFs in the region of masses intermediate between the mass of heavy residues and the mass of light IMFs.Further simulations are in progress.The experimental analysis will be enriched also by information obtained inspecting the IMF-IMF correlation function,in order to elucidated the nature of space-time decay property of the emitting source associated with events having the largest IMF multiplicity.FRANCALANZA L ABBONDANNO U AMORINI F BARLINI S BINI M BOUGAULT R BRUNO M CARDELLA G CASINI G AGOSTINO M D' De FILIPPO V De SANCTIS J GERACI E GIUSSANI A GRAMEGNA F GUIOT B KRAVCHUK V La GUIDARA E LANZALONE G Le NEINDRE N MAIOLINO C MARINI P MORELLI L OLMI A PAGANO A PAPA M PIANTELLI S PIRRONE S POLITI G POGGI G PORTO F RUSSOTTO P RIZZO F VANNINI G VANNUCCI L 2013Nuclear Science and Techniques2013,24,5:2
2Multiple Rh messenger RNA isoforms are produced by alternative splicing显示文摘LE VAN K C CHERIF-ZAHAR B RAYNAL V 1992Blood1992,80,:1
3Capacity of myeloid and plasmacytoid dendritic cells especially at mature stage to express and secrete HLA - G molecules 显示文摘Le Friec G Gins F Sebti Y Guilloux V Panganlt C Fauchet R Amiot L 2004J Leukoc Biol2004,76,6:1
4Old age and the hepatic sinusoid 显示文摘Le Couteur D G Warren A Cogger V C 2008Anat Rec ( Hoboken)2008,291,6:1
5Measuring the VC-dimension of a learning machine显示文摘VAPNIK V N LEVIN E LE C Y 1994Neural Computation1994,,6:1
6Measuring the VC-dimension of a learning machines显示文摘Vapnik V Levin E Le C Y 1994Neural Compution1994,,6:1
7Activity of glyco-peptides against Staphylococcus aureus infection in a rabbit endo-carditis model:MICs do not predict in vivo efficacy显示文摘Asseray N Jacqueline C Le Mabecque V 2005AntimicrobAgents Chemother2005,49,2:1
8Accumulation of TDP-43 and alpha-actin in an amyotrophic lateral sclerosis patient with the K17I ANG mutation显示文摘Seilhean D Cazeneuve C Thuriès V Russaouen O Millecamps S Salachas F Meininger V Le 0,,04:1
9Human resources for the control of road traffic injury显示文摘Mock C Kobusingye O Anh Le V 0,,04:1
10From gene profiling to diagnostic markers:IL-18 and FGF-2 complement CA125 as serum-based markers in epithelial ovarian cancer显示文摘Le PAGE C OUELLET V MADORE J 2006Int J Cancer2006,18,7:1
11Polyanllne Fili ts for ProtectionAgainst Corrosion显示文摘M C Bernard A Hugot - Le Goff S Joiret P V Phong 2001Synthetic Metals2001,119,13:1
12A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera显示文摘JAME C UGO V LE COUEDIC J P 2005Nature2005,434,:1
13A unique clonal JAK2 mutation leading to constitutive signalling cau ses polycythaemia vera显示文摘James C Ugo V Le Couédic JP 2005Nature2005,434,7037:1
14A unique clonal JAK2 mutation leading to constitutive signaling causes polycythemia vera显示文摘James C Ugo V Le Couedic JP 0,,:1
15Liver Stiffness Measurement in Children Using FibroScan: Feasibility Study and Comparison With Fibrotest, Aspartate Transaminase to Platelets Ratio Index, and Liver Biopsy显示文摘Victor de Lédinghen Brigitte Le Bail Laurent Rebouissoux Céline Fournier Juliette Foucher Véronique Miette Laurent Castéra Laurent Sandrin Wassil Merrouche Frédéric Lavrand Thierry Lamireau 2007Journal of Pediatric Gastroenterology and Nutrition2007,,4:1
16A unique clonal JAK2 mutation leading to constitutive signaling causes polycythaemia vera显示文摘James C Ugo V Le Couedic JP 2005Nature2005,434,7037:1
17Lung macrophages and dendritic cells express HLA - G molecules in pulmonary diseases 显示文摘Pangault C Le Friec G Caulet - Maugendre S Lena H Amiot L Guilloux V Onno M Fauchet R 2002Hum Immunol2002,63,2:1
18SET complex in serous epithelial ovarian cancer显示文摘Ouellet V Le Page C Guyot MC 2006Cancer2006,119,9:1
19SET complex in serous epithelial ovarian cancer显示文摘Ouellet V Le Page C Guyot MC 2006In Int J Cancer2006,119,9:1
20A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera显示文摘 UGO V Le COUEDIC JP 2005Nature2005,434,:1
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