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83篇 您的检索式:作者名="Landreth GE"
    题名 作者 年代 出处 被引量
1Antineoplastic effects of peroxisome proliferator-activated receptor gamma agonists显示文摘Grommes C Landreth GE Heneka MT 2004Lancet Oncol2004,5,:2
2The role of peroxisome proliferator-activated receptor-γ (PPARγ) in Alzheimer's disease:therapeutic implications显示文摘Jiang Q Heneka M Landreth GE 2008CNS Drugs2008,22,1:1
3Inflammation, mieroglia, and Alzheimer's disease显示文摘Brent C Landreth GE 2010Neurobiol Dis2010,37,3:1
4The microglial NADPH oxidase complex as a source of oxidative stress in Alzheimer's disease显示文摘Wilkinson BL Landreth GE 2006J Neuroinflammation2006,3,:1
5Identification of microglial signal transduction pathways mediating a neurotoxic response toamyloidogenic fragments of beta-amyloid and pfion proteins显示文摘Combs CK Johnson DE Cannady SB Lehman TM Landreth GE 1999J Neurosci1999,19,3:1
6Peroxisome proliferator-activated receptor-gamma ligands reduce inflammation and infarction size in transient focal ischemia 显示文摘Sundararajan S Gamboa JL Victor NA Wandefi EW Lust WD Landreth GE 2005Neuroscience2005,130,3:1
7Inflammatory mechanisms in Alzheimer′s disease: inhibition of beta-amyloid-stimulated proinflammatory responses and neurotoxicity by PPARgamma agonists显示文摘Combs CK Johnson DE Karlo JC Cannady SB Landreth GE 2000J Neurosci2000,20,2:1
8PPARs in the brain显示文摘Heneka MT Landreth GE 2007Biochim Biophys Acta2007,1771,8:1
9Toll-like receptors in Alzheimer's disease 显示文摘LANDRETH GE REED-GEAGHAN EG 2009Curr Top Microbiol lmmunol2009,336,:1
10Microglial interaction with beta-amyloid:implications for the pathogenesis of Alzheimer's disease显示文摘Bamberger ME Landreth GE 2001Microsc Res Tech2001,54,2:1
11beta-Amyloid stimulation of microglia and monocytes results in TNF alpha-dependent expression of inducible nitric oxide synthase and neuronal apoptosis 显示文摘Combs CK Karlo JC Kao SC Landreth GE 2001J Neurosci2001,21,4:1
12Inflammation,apoptosis,and Alzheimer′s disease显示文摘Bamberger ME Landreth GE 2002Neuroscientist2002,8,3:1
13Nonsteroidal anti-inflammatory drugs and peroxisome proliferator-activated receptorgamma agonists modulate immunostimulated processing of amyloid precursor protein through regulation of beta-secretase 显示文摘Sastre M Dewachter I Landreth GE 2003J NeUrosci2003,23,30:1
14Amyloid fibrils activate tyrosine kinase-dependent signaling and superoxide production in microglia显示文摘McDonald DR Brunden KR Landreth GE 1997J Neurosci1997,17,7:1
15Regulation of beta-amyloid stimulated proinflammatory responses by peroxisome proliferator-activated receptor alpha显示文摘Combs CK Bates P Karlo JC Landreth GE 2001Neurochem Int2001,39,56:1
16Identification of microglial signal transduction pathways mediating a neurotoxic response to amyloidogenic fragments of beta-amyloid and prion proteins显示文摘Combs CK Johnson DE Cannady SB Lehman TM Landreth GE 1999J Neurosci1999,19,3:1
17Inflammation, microglia,and Alzheimer's disease 显示文摘Brent C Landreth GE 2010Neurobiol Dis2010,37,3:1
18The role of peroxisome proliferator-activated receptor-gamma (PPARgamma) in Alzheimer's disease : therapeutic implications 显示文摘Jiang Q Heneka M Landreth GE 2008CNS Drugs2008,22,1:1
19MAP’ing CNS development andcognition:an ERKsome process显示文摘Samuels IS Saitta SC Landreth GE 2009Neuron2009,61,:1
20Microglial phagocytosis induced by fibrillar beta -amyloid and IgGs are differentially regulated byproinflammatory cytokines 显示文摘Koenigsknecht -Talboo J Landreth GE 2005J Neurosci2005,25,36:1
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