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19篇 您的检索式:作者名="MENABO R"
    题名 作者 年代 出处 被引量
1L-Propionyl-carnitine protection of mitochondria in ischemic rat hearts显示文摘Lisa FD Menabo R Siliprandi N 1989Molecular and Cellular Bio- chemistry1989,88,1:1
2Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD^+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart显示文摘DILISA F MENABO R CANTON M 2001J Biol Chem2001,276,4:1
3Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolization 显示文摘Canton M Skyschally A Menabo R 2006Eur Heart J2006,27,:1
4Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolization 显示文摘Canton M Skyschally A Menabo R 2006Eur Heart J2006,27,:1
5Mitochondria and Cardioprotection显示文摘Di Lisa F Canton M Menabo R 2007Heart Fail Rev2007,12,34:1
6Mitochondria and reperfusion injury:the role of permeability transition显示文摘di Lisa F Canton M Menabo R 2003Basic Res Cardiol2003,98,4:1
7The role of mitoehondria in the salvage and the injury of the ischemic myocardium显示文摘Di Lisa F Menabo R Canton M 1998Biochim Biophys Acta1998,1366,12:1
8Oxidative modification of tropamyosin and myocardial dysfunction following coronary microembolizaation显示文摘Canton M Skyschally A Menabo R 2006Eur Heart J2006,27,7:1
9The role of mitochondria in the salvage and the injury of the ischemic myocardium显示文摘Di Lisa F Menabo R Canton M 1998Biochim Biophys Acta1998,1336,:1
10Binding of cytosolic proteins to myofibrils in ischemic rat hearts显示文摘Barbato R Menabo R Dainese P 1996Circ Res1996,78,5:1
11Opening of the mitochondfial permeability transition pore causes depletion of mitochondrial and cytosolic NAD and is a causative event in the death of myocytes in pestischemic reperfusion of the heart 显示文摘di Lisa F Menabo R Canton M 2001J Biol Chem2001,276,:1
12Mi- tochondria and reperfusion injury: the role of permea- bility transition显示文摘CANTON M MENABO R DODONI G 2003Basic Res Cardiol2003,98,:1
13Evidence of myoflbrillar protein oxidation induced by postischcmic reperfusion in isolated rat hearts显示文摘Canton M Neverova I Menabo R 2004Am J Physiol Heart Circ Physiol2004,286,3:1
14Opening of the mitochondrial permeability transition pore causes depletion of mitochondfial and cy- tosolic NAD( + ) and is a causative event in the death' of myocytes in postischemic reperfusion of the heart 显示文摘Di Lisa F Menabo R Canton M 2001J Biol Chem2001,276,:1
15Mitochondria and cardioprotection显示文摘Di-Lisa F Canton M Menabo R 2007Heart Fail Rev2007,12,34:1
16The role of mitochondria in the salvage and the injury of the ischemic myocardium显示文摘Di Lisa F Menabo R Canton M 1998Biochim Biophys Acta1998,1366,12:1
17Opening of the mitoehondrial permeability transition pore causes deple- tion of mitoehondrial and eytosolie NAD + and is a eausa- rive event in the death of myocytes in postischemic reper- fusion of the heart显示文摘Di Lisa F Menabo R Canton M 2001J Biol Chem2001,276,4:1
18Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD + and is a causative event in the death of myocytes in postischemic reperfusion of the heart显示文摘Di Lisa F Menabo R Canton M 2001J Biol Chem2001,276,:1
19Oxidative modification of tropomyosin and myocardial dysfunction following coronary microemboli- zation 显示文摘Canton M Skyschally A Menabo R 2006European Heart Journal2006,27,7:1
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