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| 1 | Type 1 diabetes and polyglandular autoimmune syndrome: A review显示文摘Type 1 diabetes(T1D) is an autoimmune disorder caused by inflammatory destruction of the pancreatic tissue. The etiopathogenesis and characteristics of the pathologic process of pancreatic destruction are well described. In addition, the putative susceptibility genes for T1 D as a monoglandular disease and the relation to polyglandular autoimmune syndrome(PAS) have also been wellexplored. The incidence of T1 D has steadily increased in most parts of the world, especially in industrialized nations. T1 D is frequently associated with autoimmune endocrine and non-endocrine diseases and patients with T1 D are at a higher risk for developing several glandular autoimmune diseases. Familial clustering is observed, which suggests that there is a genetic predisposition. Various hypotheses pertaining to viral- and bacterialinduced pancreatic autoimmunity have been proposed, however a definitive delineation of the autoimmune pathomechanism is still lacking. In patients with PAS, pancreatic and endocrine autoantigens either colocalize on one antigen-presenting cell or are expressed on two/various target cells sharing a common amino acid, which facilitates binding to and activation of T cells. The most prevalent PAS phenotype is the adult type 3 variant or PAS type Ⅲ, which encompasses T1 D and autoimmune thyroid disease. This review discusses the findings of recent studies showing noticeable differences in the genetic background and clinical phenotype of T1 D either as an isolated autoimmune endocrinopathy or within the scope of polyglandular autoimmune syndrome. | Martin P Hansen Nina Matheis George J Kahaly | 2015 | World Journal of Diabetes2015，6，1： | 7 |
| 2 | Forest planning using co-ev-olutionary cellular automata显示文摘 | Mathey E Krcmar J Innes D Tait | 2007 | Forest Ecology and Management2007，239，13： | 1 |
| 3 | Assessment of the vortex method for large eddy simulation inlet conditions显示文摘 | Mathey F Cokljat D Bertoglio J P | 2006 | Progressin Computational Fluid Dynamics2006，6，13： | 1 |
| 4 | Enzymatic cross-linking of proteins applicable to foods 显示文摘 | MATHEIS G WHITAKER J R | 1987 | Food Biochem1987，11，： | 1 |
| 5 | Molecular epidemiology of hepatitis C among drug users in Flanders, Belgium :association of genotype with clinical parameters and with sex - and drug - related risk behaviours 显示文摘 | MATHEI C WOLLANTS E VERBEECK J | 2005 | Eur J Clin Micrnbiol2005，24，8： | 1 |
| 6 | Avian encephalomyelitis in pheasant显示文摘 | Mathey W J | 1995 | Cornell Veterinarian1995，45，： | 1 |
| 7 | Surface condition effect on bond strength of steel beams in concrete显示文摘 | Bryson J O Mathey R G | 1962 | Journal of the American Concrete Institute1962，59，3： | 1 |
| 8 | Early accumulation of the terminal complement-complex in the ischaemic myocardium after reperfusion显示文摘 | Koji D Mathey D Schofer J | 1994 | European Heart Journal1994，15，： | 1 |
| 9 | Prevention of bone loss by phloridzin, an apple polyphenol, in ovariecto- mized rats under inflammation conditions 显示文摘 | Puel C Quintin A Mathey J | 2005 | Calcified Tissue International2005，77，5： | 1 |
| 10 | 查看详情显示文摘 | E Wintermantel J Mayer J Blum K-L Eckert P.Lüscher M Mathey | | 0，，： | 1 |
| 11 | Fructooligosaccharides maximize bone-sparing effects of soy isoflavone-enfiched diet in the ovariectomized rat 显示文摘 | Mathey J Puel C Kati-Coulibaly S | 2004 | Calcified Tissue International2004，75，2： | 1 |
| 12 | Reduction of ethylene-induced physiological disorders of carrots and iceberg lettuce by 1 -methycyclopropene 显示文摘 | Fan X Matheis J P | 2000 | Hortscience2000，35，7： | 1 |
| 13 | Early accumulation of the terminal complement complex in ischaemic myocardium after reperfusion显示文摘 | Mathey D Schafer J Schafer HJ | 1994 | Eur Heart J1994，15，： | 1 |
| 14 | Yellowing of broccoli in storage is reduced by 1-methycyclopropene 显示文摘 | Fan X T James P Matheis J P | 2000 | Hortscience2000，35，5： | 1 |
| 15 | Scintigraphic evidence of the 'no reflow'phenomenon in human beings after coronary thrombolysis 显示文摘 | Montz R Mathey DG | 1985 | J Am Coll Cardiol1985，5，3： | 1 |
| 16 | Influ-ence of lifelong soy isoflavones consumption on bone mass inthe rat显示文摘 | MARDON J MATHEY J KATI-COULIBALY S | 2008 | Exp Biol Med(Maywood)2008，233，2： | 1 |
| 17 | Scintigraphic evidence of the“ no reflow” phnomenon in human beings after coronary thrombolysis 显示文摘 | Schofer J Montz R Mathey DG | 1985 | J Am Coll Cardiol1985，5，3： | 1 |
| 18 | Ef- fect of a transpositional muscle flap on VEGF mRNA expression in a canine fracture model 显示文摘 | KHODAPARAST O COBERLY DM MATHEY J | 2003 | Plast Reconstr Surg2003，112，1： | 1 |
| 19 | Early accumulation of the terminal complement complex in the ischaemic myocardium after reperfusion显示文摘 | Mathey D Schofer J Schafen HJ | 1994 | Eur Heart J1994，15，3： | 1 |
| 20 | Modulation of soy isoflavones bioavailability and subsequent effects on bone health in ovariectomized rat, the case for equol显示文摘 | Mathey J Mardon J Fokialakis N | 2007 | Osteoporos Int2007，5，： | 1 |
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