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7篇 您的检索式:作者名="Mihaela N"
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1The effect of flutamide on the physical working capacity and activity of some of the key enzymes for the energy supply in adult rats显示文摘学习的目的是在老鼠为精力供应在一些关键肌肉酶的物理工作能力和活动上估计雄激素受体对手的效果。年轻成年男 Wistar 老鼠被划分成二个组。一个组一个星期和作为控制为服务 8 个星期的另外的组每天收到了 flutamide 的 15 mg kg-1 6 天。在开始并且在实验的结束,所有老鼠受到 submaximal 跑耐力(SRE ) ,疲劳(MTE ) 的最大的时间,和最大的疾跑速度(女士) 测试。在试用的结束,最大的氧消费(VO2max ) 测试被执行并且睾丸激素,红血球,血红素以及 succinate 脱氢酶(同步数字系列) 的酶活动的层次,喂奶脱氢酶(LDH ) ,和 gastrocnemius 肌肉的 NAD.H2-cytochrome-c reductase (NAD.H2 ) 被测量。对待 flutamide 的老鼠的浆液睾丸激素比控制的高,它验证选择的剂量的有效性。anti-androgen-treated 组的 MTE 和 SRE 与起始的价值相比是更低的。Flutamide 处理减少了同步数字系列和 NAD.H2 的活动与控制相比。我们没在女士, VO2max,跑的经济, LDH 活动,和 hematological 变量上发现反雄激素处理的效果。我们的调查结果显示象一些关键酶的活动一样的 submaximal 和最大的跑耐力的维护把氧化能力与肌肉联系了与雄激素效果被连接由雄激素受体调停了。Katerina N Georgieva Penka A Angelova Fani D Gerginska Dora D Terzieva Mihaela S Shishmanova-Doseva Slavi D Delchev Valentine V Vasilev 2017Asian Journal of Andrology2017,19,4:2
2The Influence of the Lubricant Viscosity on the Rolling Friction Torque 显示文摘Dumitru N O Mihaela R D B Vasile C S 2014Tribology International2014,72,:1
3The Kaldor-Kalecki stochastic model of business eyele显示文摘Gabriela M Mihaela N Dumitru O 2011Nonlinear Analysis: Modelling and Control2011,16,:1
4Estimating the uncertainty related to GC-MS analysis of organo-chlorinated pesticides from water显示文摘MIHAELA P N ALEXANDRU P N GABRIELA R U 0,,06:1
5Phospholupid hydroperoxide glutathione peroxidase : expression pattern during testieular development in mouse and evolutionary conservation in spermatozoa显示文摘Karim N Mihaela D Gerhard A 2004Molecular Reproduction and Development2004,67,:1
6Sedimentology, palaeoenvironments and biostratigraphy of the Pliocene–Pleistocene carbonate platform of Grande‐Terre (Guadeloupe, Lesser Antilles forearc)显示文摘JEAN‐JACQUES CORNéE JEAN‐LèN LéTICéE PHILIPPE MüNCH FRéDéRIC QUILLéVéRé JEAN‐FRéDéRIC LEBRUN PIERRE MOISSETTE JUAN‐CARLOS BRAGA MIHAELA MELINTE‐DOBRINESCU LYVANE DE MIN JULIEN OUDET AURAN RANDRIANASOLO 2012Sedimentology2012,,5:1
7Dynamic epi-transcriptomic landscape mapping with disease progression in estrogen receptor-positive breast cancer显示文摘Dear Editor,The molecular determinants that drive breast cancer progression to metastasis are complex and partly controlled by nucleic acid epi-modifications[1].Although DNA-methyl modifications in breast cancer metastasis have been well described,there is limited understanding of the role of RNA methylation in advanced disease[2].This study aimed to provide an understanding on the role of the epitranscriptome in estrogen receptor-positive(ER+)breast cancer progression to metastasis.Stephen Keelan Mihaela Ola Sara Charmsaz Sinéad Cocchiglia Daniela Ottaviani Seán Hickey Siobhan Purcell Fiona Bane Aisling Hegarty Ben Doherty Katherine Sheehan Lance Hudson Nicola Cosgrove Benjamin Roux Muriel Laine Geoffrey Greene Damir Varešlija Arnold Konrad Hill Leonie Young 2023Cancer Communications2023,43,5:0
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