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| 1 | Both hemopoietic and resident cells are required for MyD88-dependent pulmonary inflammatory, response to inhaled endotoxin显示文摘 | Noulin N Quesniaux VF Schnyder-Candrian S | 2005 | Immunol2005，175，10： | 1 |
| 2 | Cigarette smoke -induced pulmonary inflammation is TLR4/MyD88 and IL - 1R1/MyD88 signaling dependent显示文摘 | Doz E Noulin N Boichot E | 2008 | J Immunol2008，180，2： | 1 |
| 3 | Cigarette smoke-induced pulmonary inflammation is TLR4/MyD88 and IL-1R1/ MyD88 signaling dependent 显示文摘 | Doz E Noulin N Boichot E | 2008 | J Immunol2008，180，2： | 1 |
| 4 | Cigarette smoke-induced pulmonary inf lammation is TLR4/MyD88 and IL1R1/MyD88 signaling dependent显示文摘 | Doz E Noulin N Boichot E | 2008 | J Immunol2008，180，： | 1 |
| 5 | Cigarette smoke-inducedMyD88 signaling dependent 显示文摘 | Doz E Noulin N Boichot E | 2008 | J Immunol2008，180，2： | 1 |
| 6 | Both hemopoietic and resident cells are required for MyD88-dependent pulmonary inflammatory response to inhaled endotoxin显示文摘 | Noulin N Quesniaux VF Schnyder-Candrian S | 2005 | Immunol2005，175，10： | 1 |
| 7 | Cigarette smoke induced pulmonary inflammation is TLR4/MyD88 and IL 1R1/MyD88 signaling dependent显示文摘 | Doz E Noulin N Boichot E | | 0，，： | 1 |
| 8 | Both hemopoietic and resident cells are required for MyD88 - dependent pulmonary inflammatory response to inhaled endotoxin 显示文摘 | Noulin N Quesniaux VF Schnyder - Candrian S | 2005 | Immunol2005，175，8： | 1 |
| 9 | T cellderived TNF down-regulates acute airway response to endotoxin显示文摘 | Togbe D Grivennikov SI Noulin N | 2007 | Eur J Immuno12007，37，7： | 1 |
| 10 | Cigarette smoke-induced pulmonary inflammation is TLR4/MyD88 and IL-1R1/MyD88 signaling dependent显示文摘 | Doz E Noulin N Boichot E | | 0，，： | 1 |
| 11 | Cigarette smoke-induced pulmonary inflammation is TLR4/MyD88 and IL-1R1/MyD88 signaling dependent显示文摘 | Doz E Noulin N Boichot E | | 0，，： | 1 |
| 12 | Thrombocytopenia and plasmodi-um falciparum malaria in children with different exposures显示文摘 | Noulin F Lesage F Legros AH | 2003 | ArchDis Child2003，88，6： | 1 |
| 13 | Cigarette smoke- induced pulmonary inflammation is TLR4/MyD88 and IL-1R1/ MyD88 signaling dependent显示文摘 | DOZ E NOULIN N BOICHOT E et ol | 2008 | J Immunol2008，180，2： | 1 |
| 14 | Diagnosis and treatment of juvenile nasopharyngeal angiofibroma 显示文摘 | Paris J 6uelfucci B Noulin 6 | 2001 | Eur Arch Otorhinolaryngol2001，258，3： | 1 |
| 15 | Cigarette smoke-induced pulmonary inflammation is TLR4/MyD88 and IL-1R1/MyD88signaling dependent显示文摘 | Doz E Noulin N Boichot E | 2008 | J Immunol2008，180，2： | 1 |
| 16 | Thrombocytopenia and plasmodi- um falciparum malaria in children with different exposures 显示文摘 | Noulin F Lesage F Legros AH | 2003 | Arch Dis Child2003，88，6： | 1 |
| 17 | Two types of K^+ channels at the basolateral membrane of proximal tubule :inhibitory effect of taurine显示文摘 | NOULIN J F BROCHIERO E LAPOINTE J Y | 1999 | Am J Physiol1999，277，22： | 1 |
| 18 | Cigarette smoke-induced pulmonary inflammation is TLR4/MyD88 and IL-1R1/MyD88 signaling dependent显示文摘 | Doz E Noulin N Boichot E | 2008 | J Immunol2008，180，2： | 1 |
| 19 | Malaria modeling: In vitro stem cells vs in vivo models显示文摘The recent development of stem cell research and the possibility of generating cells that can be stably and permanently modified in their genome open a broad horizon in the world of in vitro modeling. The malaria field is gaining new opportunities from this importantbreakthrough and novel tools were adapted and opened new frontiers for malaria research. In addition to the new in vitro systems, in recent years there were also significant advances in the development of new animal models that allows studying the entire cell cycle of human malaria. In this paper, we review the different protocols available to study human Plasmodium species either by using stem cell or alternative animal models. | Florian Noulin | 2016 | World Journal of Stem Cells2016，8，3： | 0 |
| 20 | Unexpected encounter of the parasitic kind显示文摘Both parasitology and stem cell research are important disciplines in their own right.Parasites are a real threat to human health causing a broad spectrum of diseases and significant annual rates morbidity and mortality globally.Stem cell research,on the other hand,focuses on the potential for regenerative medicine for a range of diseases including cancer and regenerative therapies.Though these two topics might appear distant,there are some“unexpected encounters”.In this review,we summarise the various links between parasites and stem cells.First,we discuss how parasites’own stem cells represent interesting models of regeneration that can be translated to human stem cell regeneration.Second,we explore the interactions between parasites and host stem cells during the course of infection.Third,we investigate from a clinical perspective,how stem cell regeneration can be exploited to help circumvent the damage induced by parasitic infection and its potential to serve as treatment options for parasitic diseases in the future.Finally,we discuss the importance of screening for pathogens during organ transplantation by presenting some clinical cases of parasitic infection following stem cell therapy. | Holly Matthews Florian Noulin | 2019 | World Journal of Stem Cells2019，11，11： | 0 |
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