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20篇 您的检索式:作者名="RAFF A C"
    题名 作者 年代 出处 被引量
1Microtubule architecture specified by a β-tubulin isoform显示文摘Raff E C Fackenthal J D Hutchens J A 1997Science1997,275,:1
2Programmed cell death and the control of cell survival lesions from the nervous sysytem显示文摘 Barres B A Burne J F 1993Science1993,262,95:1
3Axonal self-destruction and neurodegeration 显示文摘Raff M C Whitmire A V Finn J T 2002Science2002,296,5569:1
4Stimulation of NADPH oxidase by oxidized low-density lipoprotein induces proliferation of human vascular endothelial cells显示文摘Heinloth A Heermeler K Raff U Wanner C and Galle J 2001J Am Soc Nephrol2001,11,:1
5Dual effect of oxidized LDL on cell cycle in human endothelial cells through oxidative stress显示文摘Heinloth A Heermeler K Raff U Wanner C and Galle J 2001Kidney Internat2001,59,78:1
6Axonal self-destruction and neurodegenerati-on 显示文摘Raff M C Whitmore A V Finn J T 2002Science2002,296,5569:1
7Effects of telmisartan and ramipril on adiponectin and blood pressure in patients with type 2 diabetes显示文摘Delles C Raff U Mimran A 0,,12:1
8Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy显示文摘Susztak K Raff A C Schiffer M 2006Diabetes2006,55,1:1
9Microtubule architecture specified by a beta-tubulin isoform显示文摘Raff E C Fackenthal J D Hutchens J A 1997Science1997,275,5296:1
10Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy显示文摘SUSZTAK K RAFF A C SCHIFFER M 2006Diabetes2006,55,:1
11A role for sonic hedgehog in axon-to- astrocyte signalling in the rodent optic nerve 显示文摘WALLACE V A RAFF M C 1999Development1999,126,13:1
12Glucose- induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy 显示文摘SUSZTAK K RAFF A C SCHIFFER M 2006Diabetes2006,55,1:1
13Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy显示文摘Susztak K Raff A C Schiffer M 2006Diabetes2006,55,1:1
14Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy显示文摘Susztak K Raff A C Schiffer M 0,,1:1
15Programmed cell death and the control of cell survival: lessons from the nervous system 显示文摘RAFF M C BARRES B A BUME J F 1993Science1993,262,:1
16Glucose -in- duced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy 显示文摘SUSZTAK K RAFF A C SCHIFFER M 2006Dia- betes2006,55,1:1
17Glucose-induced reac- tive oxygen species cause apoptosis of podocytes and podocyte deple- tion at the onset of diabetic nephropatby 显示文摘SUSZTAK K RAFF A C SCHIFFER M 2006Diabetes2006,55,1:1
18Effects of telmisartan and ramipril on adiponectin and blood pressure in patients with type 2diabetes显示文摘Delles C Raff U Mirnran A 2008Am J Hypertens2008,21,12:1
19Glucose-induced reactive oxy- gen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy 显示文摘Susztak K Raff A C Schiffer M 2006Diabetes2006,55,1:1
20替米沙坦和雷米普利对2型糖尿病患者脂联素和血压的影响显示文摘本文对87名2型糖尿病患者的基线水平和使用替米沙坦(最终剂量80mg,n=45)或雷米普利(最终剂量10mg,n=42)治疗9周后的血清脂联素水平进行评价。结果显示,患者的脂联素水平与基线水平收缩压(SBP:r=0.240,P〈0.05)和舒张压(DBP:r=0.227,P〈0.05)、以及替米沙坦或雷米普利治疗后的SBP(r=0.228,P〈0.05)和DBP(r=-0.286,P〈0.05)均呈负相关。脂联素水平的改变与SBP(r=-0.357,P〈0.0I)和DBP(r=-0.286,P〈0.01)的改变有关。Delles C Raff U Mimran A 2009药品评价2009,6,6:0
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