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| 1 | Management of chronic hepatitis B in children: An unresolved issue显示文摘 | Claudia Della Corte Valerio Nobili Donatella Comparcola Francesca Cainelli Sandro Vento | 2014 | J Gastroenterol Hepatol2014，，5： | 2 |
| 2 | Reactivation of replication of hepatitis B and C viruses after immunosuppressive therapy: an unresolved issue显示文摘 | Sandro Vento Francesca Cainelli Maria Serena Longhi | 2002 | Lancet Oncology2002，，6： | 1 |
| 3 | Infections in patients with cancer undergoing chemotherapy: aetiology, prevention, and treatment显示文摘 | Sandro Vento Francesca Cainelli | 2003 | Lancet Oncology2003，，10： | 1 |
| 4 | Reactivation of replication of hepatitis B and C viruses after immunosuppressive therapy: an unresolved issue显示文摘 | Sandro Vento Francesca Cainelli Maria Serena Longhi | 2002 | Lancet Oncology2002，，6： | 1 |
| 5 | Lung infections after cancer chemotherapy显示文摘 | Sandro Vento Francesca Cainelli Zelalem Temesgen | 2008 | Lancet Oncology2008，，10： | 1 |
| 6 | Infections and solid organ transplant rejection: a cause-and-effect relationship?显示文摘 | Francesca Cainelli Sandro Vento | 2002 | Lancet Infect Dis2002，2，7： | 1 |
| 7 | 病毒是自身免疫性肝炎的触发因素吗?(上)显示文摘自身免疫性肝炎(AH)是一种少见的疾病,病因学不明,以女性为主,人群发病率为0.01-0.02%,特征性的血清学变化为血清IgG显著升高。自然史以反复发生在肝小叶内和汇管区交界面的坏死性炎症(碎片状坏死)最终导致肝硬化甚至肝衰竭,在所有自身免疫性疾病中它对皮质激素治疗的反应最好。 | Sandro Vento Francesca Cainelli 姚勤伟(编译) 杨月(审校) | 2006 | 传染病网络动态2006，，6： | 0 |
| 8 | 病毒是自身免疫性肝炎的触发因素吗?(下)显示文摘4.病毒在AH2型中是否也起了这种作用? 这种自身免疫性肝病标志性的靶点,即LKM1,是细胞色素P450IID6(CYP2D6),肝脏P450酶家族的一个成员。Manns等人认为针对由LKM1抗体识别的CYP2D6主要抗原决定簇的反应可能通过对丙型肝炎病毒(HCV)或单纯疱疹病毒1型(HSVI)的交叉反应而产生,因为HCV E1包膜区域的310-324位氨基酸和HSV1的快速调节蛋白IE175的156-170位氮基酸与CYP2D6免疫决定区的254.271位氨基酸具有序列同源性,可被85%的AH2型患者所识别。然而Klein等人证明针对细胞色素P2D6(CYP2D6196-218)的线性B细胞抗原决定簇的反应发生于多到68%的AH2型患者,而在LKM1+ve的HCV感染患者身上的发生率只有18%。 | Sandro Vento Francesca Cainelli 姚勤伟(编译) 杨月(审校) | 2006 | 传染病网络动态2006，，7： | 0 |
| 9 | Defensive medicine:It is time to finally slow down an epidemic显示文摘Defensive medicine is widespread and practiced the world over, with serious consequences for patients, doctors, and healthcare costs. Even students and resi-dents are exposed to defensive medicine practices and taught to take malpractice liability into consideration when making clinical decisions. Defensive medicine is generally thought to stem from physicians' perception that they can easily be sued by patients or their relatives who seek compensation for presumed medical errors. However, in our view the growth of defensive medicine should be seen in the context of larger changes in the conception of medicine that have taken place in the last few decades, undermining the patient–physician trust, which has traditionally been the main source of professional satisfaction for physicians. These changes include the following: time directly spent with patients has been overtaken by time devoted to electronic health records and desk work; family doctors have played a progressively less central role; clinical reasoning is being replaced by guidelines and algorithms; the public at large and a number of young physicians tend to believe that medicine is a perfect science rather than an imperfect art, as it continues to be; and modern societies do not tolerate the inevitable morbidity and mortality. To finally reduce the increasing defensive behavior of doctors around the world, the decriminalization of medical errors and the assurance that they can be dealt with in civil courts or by medical organizations in all countries could help but it would not suffice. Physicians and surgeons should be allowed to spend the time they need with their patients and should give clinical reasoning the importance it deserves. The institutions should support the doctors who have experienced adverse patient events, and the media should stop reporting with excessive evidence presumed medical errors and subject physicians to 'public trials' before they are eventually judged in court. | Sandro Vento Francesca Cainelli Alfredo Vallone | 2018 | World Journal of Clinical Cases2018，6，11： | 0 |
| 10 | 转氨酶作为死亡预测因子的价值显示文摘转氨酶[丙氨酸氨基转移酶(ALT)和天冬氨酸氨基转移酶(AST)]早在20世纪50年代就已被证明可用作肝细胞损害的标记物。一项新研究提示,这些酶的浓度增加可以预测未来10年内的死亡情况。Lee及其同事对1995年在梅奥诊所(Rochester,MN,USA)就诊的所有Olmsted郡成年患者进行了鉴别,并从实验室数据库中提取了他们的酶检验结果。对这些患者进行了11年的随访。结果显示,AST浓度为正常上限的1~2倍时, | Sandro Vento Valerio Nobili 刘莉君(译) | 2009 | 世界临床医学2009，3，3： | 0 |
| 11 | 白介素-2治疗HIV-1感染患者及其与CD4+T细胞的关系显示文摘自从HIV开始流行以来,白介素.2-直处于对该病进行免疫治疗的前沿,早在1983年内源性白介素.2的Ⅰ期研究就已开展。这种尝试的重要依据就是相继观察到某些功能上的缺陷即HIV-1感染患者的CD4+T细胞中对抗原和丝分裂素增殖反应的降低,而这些缺陷是与白介素.2产量的降低相关的,反之分泌白介素.2的CD4+T细胞其完整性是与良好的临床转归相关的。白介素.2随机试验多以5天一周期作为起始引导方案,每8周进行一次,共进行3-6个周期。随后,大多数接受治疗患者仅需要少数几个周期即可维持CD4+T细胞的增殖。 | Sandro Vento 杨月(编译) | 2006 | 传染病网络动态2006，，8： | 0 |
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