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| 1 | Serological follow-up in juvenile myasthenia:Clinical and acetylcholine receptor antibody status of patients followed for at least 2 years显示文摘 | Anlar B Senbil N Kose G | 2005 | Neromuscul Dis2005，15，5： | 1 |
| 2 | 显示文摘 | Aydin OF Uner C Senbil N | 2003 | J Child Neuro12003，18，4： | 1 |
| 3 | Prothrombotic risk factors in children with hemiplegic cerebral palsy 显示文摘 | Senbil N Yiiksel D Yilmaz D | 2007 | Pediatr lnt2007，49，5： | 1 |
| 4 | Epileptic and non-epileptic cere-bral palsy: EEG and cranial imaging findings 显示文摘 | Senbil N Sonel B Aydin OF | 2002 | Brain Dev2002，24，： | 1 |
| 5 | Serological follow-up in juvenile myasthenia: clinical and acetylcholine receptor antibody status of patients followed for at least 2 years显示文摘 | Anlar B Senbil N Kose G | 2005 | Neuromuscul Disord2005，15，5： | 1 |
| 6 | Epileptic and non-epileptic cerebral palsy: EEG and cranial imaging findings 显示文摘 | Senbil N Sonel B Aydin OF | 2002 | Brain Dev2002，24，3： | 1 |
| 7 | Subacute sclerosing panencephalitis : a cause of acute vision loss显示文摘 | Senbil N Aydin OF Orer H | 2004 | Pediatr Neurol2004，31，3： | 1 |
| 8 | Epileptic and non-epileptic cerebral palsy:EEG and cranial imaging findings显示文摘 | Senbil N Sonel B Aydin OF | 2002 | Brain DEV2002，24，： | 1 |
| 9 | Serological follow-up in juvenile myasthenia:Clinical and acetylcholine receptor antibody status of patients followed for at least 2 years显示文摘 | Anlar B Senbil N Kose G | 2005 | Neromuscul Dis2005，15，5： | 1 |
| 10 | Serological follow-up in juvenile myasthenia:clinical and acetylcholine receptor antibody status of patients followed for at least 2 years显示文摘 | Anlar B Senbil N Kose G | 2005 | Neuromuscul Dis2005，15，1： | 1 |
| 11 | Epileptic and non epileptic cerebral palsy: EEG and cranial imaging findings显示文摘 | Senbil N Sonel B Aydin OF | 2002 | Brain Dev2002，24，： | 1 |
| 12 | Cerebrospinal fluid ni- tric oxide levels in subacute sclerosing panencephalitis显示文摘 | Yihnaz D Yaksel D Senbil N | 2009 | Pediatric Neurology2009，41，3： | 1 |
| 13 | Fixation-off sensitivity an generalized epileptic EEG induced by eyes closed显示文摘 | Senbil N Sayer(o) Turanly G | | 0，，： | 1 |
| 14 | Epileptic and non-epileptic cerebral palsy:EEG and cranial imaging findings显示文摘 | Senbil N Sonel B Aydin OF | 2002 | Brain Dev2002，24，3： | 1 |
| 15 | Serological follow-up in juvenile myasthenia:clinical and acetylcholine receptor antibody status of patients followed for at least 2 years显示文摘 | Anlar B Senbil N Kose G | 2005 | Neuromuscul Dis2005，15，5： | 1 |
| 16 | Imaging of acute appendicitis: Advances显示文摘We read with interest the review by Teng et al,who summarized the current approach to the diagnosis and treatment of acute appendicitis(AA).Also,the article summarizes the clinical scoring systems very effectively.In one of the previous studies conducted by our research group,we showed that the use of the Alvarado score,ultrasound and C-reactive protein values in combination provides a safe confirmation or exclusion of the diagnosis of AA.Computed tomography is particularly sensitive in detecting periappendiceal abscess,peritonitis and gangrenous changes.Computed tomography is not a good diagnostic tool in pediatric patients because of the ionizing radiation it produces.Ultrasound is a valuable diagnostic tool to differentiate AA from lymphoid hyperplasia.Presence of fluid collection in the periappendiceal and lamina propria thickness less than 1 mm are the most effective parameters in differentiating appendicitis from lymphoid hyperplasia.Although AA is the most common cause of surgical acute abdomen,it remains an important diagnostic and clinical challenge.By combining clinical scoring systems,laboratory data and appropriate imaging methods,diagnostic accuracy and adherence to treatment can be increased.Lymphoid hyperplasia and perforated appendicitis present significant diagnostic challenges in children.Additional ultrasound findings are increasingly defined to differentiate AA from these conditions. | Sonay Aydın Erdal Karavas Duzgun Can Senbil | 2022 | World Journal of Gastrointestinal Surgery2022，14，4： | 1 |
| 17 | Chronic otitis media and middle ear variants:Is there relation?显示文摘BACKGROUND Chronic otitis media(COM)is an inflammatory disease that lasts for a long time.It is common in developing countries.Hearing loss can result from COM.The relationship between variations in middle ear anatomy and COM was investigated in our study.AIM To compare the prevalence of middle ear anatomic variations between the cases with COM and healthy individuals.METHODS This retrospective study included 500 patients with COM and 500 healthy controls.The presence of those variants was determined:Koerner’s septum,facial canal dehiscence,high jugular bulb,jugular bulb dehiscence,jugular bulb diverticulum,sigmoid sinus anterior location and deep tympanic recesses.RESULTS A total of 1000 temporal bones were examined.The incidences of these variants were respectively(15.4%-18.6%),(38.6%-41.2%),(18.2%-4.6%),(2.6%-1.2%),(1.2%-0%),(8.6%-0%),(0%-0%).It was observed that only high jugular bulb(P<0.001)and anteriorly located sigmoid sinus frequencies(P=0.002)in the case group were statistically significantly higher than the control groups.CONCLUSION COM is a multifactorial disease and variants of middle ear have always been important in terms of potential risk for complication during surgery but rarely associated with COM as an etiology or as a consequence of the disease.We didn't find a positive correlation between COM and Koerner’s septum and facial canal defect.We ended up with a significant conclusion with the variants of dural venous sinuses-high jugular bulb,dehiscence of jugular bulb,diverticulum of jugular bulb and anteriorly located sigmoid sinus-that have been studied less and frequently associated with inner ear illnesses. | Fatma Dilek Gokharman Düzgün Can Senbil Sonay Aydin Erdal Karavas Ozge Ozdemir Arzu Gülsah Yalcin Pınar Nercis Kosar | 2023 | World Journal of Clinical Cases2023，11，15： | 0 |
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