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| 1 | On-treatment serum HBsAg level is predictive of sustained off-treatment virologic response to telbivudine in HBeAg-positive chronic hepatitis B patients显示文摘 | Wei Cai Qing Xie Baoyan An Hui Wang Xiaqiu Zhou Guomin Zhao Qing Guo Ruiying Gu Shisan Bao | 2010 | Journal of Clinical Virology2010，，1： | 3 |
| 2 | Variation in external characters of goldfish,Carassius auratus显示文摘 | Chen Shisan C | 1925 | Contr Biol Lab Sci Soc China1925，1，1： | 1 |
| 3 | The inheritance of blue and brown colors in the goldfish,Carassius auratus显示文摘 | Chen Shisan C | 1934 | Journal of Genetics1934，29，： | 1 |
| 4 | Long‐term lamivudine treatment achieves regression of advanced liver fibrosis/cirrhosis in patients with chronic hepatitis B显示文摘 | Bei Xu Lanyi Lin Guoguang Xu Yan Zhuang Qing Guo Yunye Liu Hui Wang Xiaqiu Zhou Shanming Wu Shisan Bao Wei Cai Qing Xie | 2015 | J Gastroenterol Hepatol2015，，2： | 1 |
| 5 | The role of granulocyte macrophage-colony-stimulating factor in acute intestinal inflammation显示文摘mucosal 支持 inflammatory 和反煽动性的 cytokines 的不平衡在煽动性的肠疾病(IBD ) 的致病是关键的。GM-CSF 影响 hemopoietic 房间的发展。在 IBD 的 GM-CSF 的精确角色尚待被阐明。GM-CSF 基因大美人(GM-CSF-/-) 并且野类型(Wt ) 老鼠与 2.5% 葡聚糖硫酸盐钠(决策支持系统) 被质问 7 天。续起临床、病理学的变化,巨噬细胞渗入,结肠的 cytokine 生产,和细菌的计数被检验。对待决策支持系统的 GM-CSF-/- 鼠标比对待决策支持系统的 Wt 鼠标开发了更严重的尖锐大肠炎,由更大的身体重量损失思考了,更直肠的流血,并且加重的组织病理学说的变化。更多的渗透的巨噬细胞在 GM-CSF-/- 被观察,与跟随决策支持系统挑战的 Wt 老鼠相比,与单核白血球 chemoattractant protein-1 (MCP-1 ) 相关生产。结肠的 IL-17 和 TNF- 的层次在 GM-CSF-/- 老鼠,然而并非在 Wt 老鼠显著地被增加,跟随决策支持系统管理。分别地, IL-6 的水平被 1.5 褶层和 2 褶层在 GM-CSF-/- 和 Wt 老鼠的冒号增加后面的决策支持系统挑战。在 IL-4 和 IFN- 的重要变化都没在跟随决策支持系统处理的 Wt 和 GM-CSF-/- 老鼠被检测。分别地,从冒号的细菌恢复在 Wt 老鼠和 GM-CSF-/- 老鼠列在后面决策支持系统挑战关于 15-fold, 和 5 褶层被增加。这些结果建议 GM-CSF-/- 老鼠更产生尖锐导致决策支持系统的大肠炎,可能因为一副损害内脏由于减少的 GM-CSF 的天生的有免疫力的反应。 | Yinghua Xu Nicholas H Hunt Shisan Bao | 2008 | Cell Research2008，18，12： | 1 |
| 6 | The distibution of cofilin and DNase I in vivo显示文摘Actin is the principal component of the cytoskeleton, a structure that can be disassembled and reassem-bled in a matter of seconds in vivo. The state of assembly of actin in vivo is primarily regulated by one ormore actin binding proteins (ABPs). Typically, the actions of ABPs have been studied one by one, however,we propose that multiple ABPs, acting cooperatively, may be involved in the control of actin filament length.Cofilin and DNase I are two ABPs that have previously been demonstrated to form a ternary complex withactin in vitro. This is the first report to demonstrate their co-localisation in vivo, and differences in theirdistributions. Our observations strongly suggest a physiological role for higher order complexes of actin inregulation of cytoskeletal assembly during processes such as cell division. | DEEPAK CHHABRA sHISAN BAO CRISTOBAL G DOS REMEDIOS | 2002 | Cell Research2002，12，4： | 1 |
| 7 | Regulation of mucosal IgA responses in vivo:cytokines and adjuvants显示文摘 | Alan J H David R K Shisan B | 1996 | Vet Immunol Immunopathol1996，54，： | 1 |
| 8 | Krüppel-like transcription factors: A functional family显示文摘 | Richard Pearson Jacqueline Fleetwood Sally Eaton Merlin Crossley Shisan Bao | 2007 | International Journal of Biochemistry and Cell Biology2007，，： | 1 |
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