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您的检索式：作者名="Sique Chen"
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| 1 | The MAX2-KAI2 module promotes salicylic acid-mediated immune responses in Arabidopsis显示文摘Arabidopsis MORE AXILLARY GROWTH2 MAX2)is a key component in the strigolactone(SL)and karrikin(KAR)signaling pathways and regulates the degradation of SUPPRESSOR OF MAX21/SMAX1-like(SMAX1/SMXL)proteins,which are transcriptional co-repressors that regulate plant architecture,as well as abiotic and biotic stress responses.The max2 mutation reduces resistance against Pseudomonas syringae pv.tomato(Pst).To uncover the mechanism of MAX2-mediated resistance,we evaluated the resistance of various SL and KAR signaling pathway mutants.The resistance of SL-deficient mutants and of dwarf 14(d14)was similar to that of the wild-type,whereas the resistance of the karrikin insensitive 2(kai2)mutant was compromised,demonstrating that the KAR signaling pathway,not the SL signaling pathway,positively regulates the immune response.We measured the resistance of smaxl and smxl mutants,as well as the double,triple,and quadruple mutants with max2,which revealed that both the smax1 mutant and smx16/7/8 triple mutant rescue the low resistance phenotype of max2 and that SMAX1 accumulation diminishes resistance.The susceptibility of smax1D,containing a degradation-insensitive form of SMAX1,further confirmed the SMAX1 function in the resistance.The relationship between the accumulation of SMAX1/SMXLs and disease resistance suggested that the inhibitory activity of SMAX1 to resistance requires SMXL6/7/8.Moreover,the exogenous application of KAR2 enhanced resistance against Pst,but KAR-induced resistance depended on salicylic acid(SA)signaling.Inhibition of karrikin signaling delayed SA-mediated defense responses and inhibited pathogen-induced protein biosynthesis.Together,we propose that the MAX2-KAI2-SMAX1 complex regulates resistance with the assistance of SMXL6/7/8 and SA signaling and that SMAX1/SMXLs possibly form a multimeric complex with their target transcription factors to fine tune immune responses. | Xiujuan Zheng Fangqian Liu Xianfeng Yang Weiqiang Li Sique Chen Xinwu Yue Qi Jia Xinli Sun | 2023 | Journal of Integrative Plant Biology2023，65，6： | 0 |
| 2 | Formation mechanisms of Ti_(3)(Si,Al)C_(2)/Al_(2)O_(3) composites from Ti_(3)AlC_(2) and SiO via low-temperature sintering显示文摘Ti_(3)SiC_(2)/Al_(2)O_(3) composites have attracted attention due to their excellent mechanical and electromagnetic properties,but the high temperatures(≥1400℃)required for the densification of aluminum oxide(Al_(2)O_(3))leads to the decomposition of Ti_(3)SiC_(2).To address this issue,Ti_(3)(Si_(x)Al_(1−x))C2/Al_(2)O_(3)(x represents the Si content)composites were synthesized for the first time via hot-pressing(HP)sintering and current-assisted sintering(CAS)of mixed Ti_(3)AlC_(2) and silicon monoxide(SiO)powders at 1300 and 1200℃,respectively.Both approaches produced composites with x values greater than 0.9,indicating that the compositions of the prepared composites were similar to those of Ti_(3)SiC_(2)/Al_(2)O_(3) composites.The synthetic mechanism involved substitution and continuous interdiffusion of Al and Si atoms.The composite prepared by CAS at 1200℃ was compacted,whereas the composite prepared by HP had a low density.The low-temperature densification mechanism is attributed to the combined effects of amorphous SiO,liquid Al,and the high heating rates for CAS.The flexural strength and hardness of the composite prepared by CAS were also comparable to those of compacted Ti_(3)SiC_(2)/Al_(2)O_(3) composites. | Zhenyu ZHANG Jun JI Yingying CHEN Deli MA Sique CHEN Hailing YANG Guopu SHI Zhi WANG Mengyong SUN Fei CHEN Shifeng HUANG Qinggang LI | 2023 | Journal of Advanced Ceramics2023，12，1： | 0 |
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