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36篇 您的检索式:作者名="Sweeney B F"
    题名 作者 年代 出处 被引量
1Stress-dependent regulation of FOXO transcription factors by the SIRT1 deacetylase显示文摘BRUNET A SWEENEY L B STURGILL J F 2004Sci2004,303,5666:1
2Moral Intensity and Ethical Decision-making : An Empirical Examination of Undergraduate Accounting and Business Students 显示文摘Sweeney B Costello F 2009Accounting Education2009,18,1:1
3显示文摘Holden B A Sweeney D F 1986Clin Exp Optoml1986,69,:1
4Myosin light chain phosphorylation in vertebrate striated mus- cle : regulation and function 显示文摘Sweeney H L Bowman B F Stull J T 1993Am J Physiol Cell Physiol1993,264,5:1
5Stress-dependent regulation of FOXO transcription factors by the SIRT1 deacetylase显示文摘Brunet A Sweeney L B Sturgill J F Chua K F Greer P L Lin Y Tran H Ross S E Mostoslavsky R Cohen H Y Hu L S Cheng H L Jedrychowski M P Gygi S P Sinclair D A Alt F W Greenberg M E 0,,5666:1
6Stress-dependent regulation of FOXO transcription factors by the SIRT1 deacetylase显示文摘BRUNET A SWEENEY L B STURGILL J F 2004Science2004,303,5666:1
7Stress-dependent regulation of FOXO transcription factors by the SIRT1 deacetylase 显示文摘Brunet A Sweeney L B Sturgill J F 2004Science2004,303,5666:1
8Epithelial Erosions Caused by Thin High Water Content Lens显示文摘HOLDEN B A SWEENEY D F 1986Clin Exp Optom1986,69,:1
9Prevalence of proximal adenomas after an adenoma is found on flexible sigmoidoscopy 显示文摘O'Brien K Sweeney B F Jr Conger N 2003Cancer Detect Prey2003,27,4:1
10Stress-depend- ent regulation of FOXO transcription factors by the SIRT1 deaeety- lase 显示文摘BRUNET A SWEENEY L B STURGILL J F 2004Seience2004,303,:1
11Stress-de- pendent regulation of FOXO transcription factors by the SIRT1 deacetylase显示文摘BRUNET A SWEENEY L B STURGILL J F 2004Science2004,303,5666:1
12Phase Ⅱ study of pemetrexed for second-line treatment of transitional cell cancer of the urothelium 显示文摘Sweeney C J Roth B J Kabbinavar F F 2006J Clin Oncol2006,24,21:1
13Gastric intestinal metaplasia development in African American predominant United States population显示文摘BACKGROUND Gastric cancer significantly contributes to cancer mortality globally.Gastric intestinal metaplasia(GIM)is a stage in the Correa cascade and a premalignant lesion of gastric cancer.The natural history of GIM formation and progression over time is not fully understood.Currently,there are no clear guidelines on GIM surveillance or management in the United States.AIM To investigate factors associated with GIM development over time in African American-predominant study population.METHODS This is a retrospective longitudinal study in a single tertiary hospital in Washington DC.We retrieved upper esophagogastroduodenoscopies(EGDs)with gastric biopsies from the pathology department database from January 2015 to December 2020.Patients included in the study had undergone two or more EGDswith gastric biopsy.Patients with no GIM at baseline were followed up until they developed GIM or until the last available EGD.Exclusion criteria consisted of patients age<18,pregnancy,previous diagnosis of gastric cancer,and missing data including pathology results or endoscopy reports.The study population was divided into two groups based on GIM status.Univariate and multivariate Cox regression was used to estimate the hazard induced by patient demographics,EGD findings,and Helicobacter pylori(H.pylori)status on the GIM status.RESULTS Of 2375 patients who had at least 1 EGD with gastric biopsy,579 patients were included in the study.138 patients developed GIM during the study follow-up period of 1087 d on average,compared to 857 d in patients without GIM(P=0.247).The average age of GIM group was 64 years compared to 56 years in the non-GIM group(P<0.001).In the GIM group,adding one year to the age increases the risk for GIM formation by 4%(P<0.001).Over time,African Americans,Hispanic,and other ethnicities/races had an increased risk of GIM compared to Caucasians with a hazard ratio(HR)of 2.12(1.16,3.87),2.79(1.09,7.13),and 3.19(1.5,6.76)respectively.No gender difference was observed between the study populations.Gastritis was associated with an increased risk for GIM development with an HR of 1.62(1.07,2.44).On the other hand,H.pylori infection did not increase the risk for GIM.CONCLUSION An increase in age and non-Caucasian race/ethnicity are associated with an increased risk of GIM formation.The effect of H.pylori on GIM is limited in low prevalence areas.Akram I Ahmad Arielle Lee Claire Caplan Colin Wikholm Ioannis Pothoulakis Zaynab Almothafer NishthaRaval Samantha Marshall Ankit Mishra Nicole Hodgins In Guk Kang Raymond K Chang Zachary Dailey Arvin Daneshmand Anjani Kapadia Jae Hak Oh Brittney Rodriguez Abhinav Sehgal Matthew Sweeney Christopher B Swisher Daniel F Childers Corinne O'Connor Lynette M Sequeira Won Cho 2022World Journal of Gastrointestinal Endoscopy2022,14,10:1
14Myosin light chain phosphorylation in vertebrate striated muscle: Regulation and function 显示文摘Sweeney H L Bowman B F and Stull J T 1993American Journal of Physiology-cell Physiology1993,264,:1
15Straiwgradient modelling of grain size effects on fatigue of CoCr alloy显示文摘Sweeney C A O' Brien B Dunne F P E McHugh P E Leen S B 2014Acta Materialia2014,78,:1
16Stress-dependent regulation of FOXO transcription factors by the SIRT1 deacetylase 显示文摘Brunet A Sweeney L B Sturgill J F 2004Science2004,303,5666:1
17Phase Ⅱ study of pemetrexed for second-line treatment of transitional cell cancer of the urothelium 显示文摘SWEENEY C J ROTH B J KABBINAVAR F F 2006J Clin Oncol2006,24,21:1
18Stress-de- pendent regulation of FOXO transcription factors by the SIRTI deacetylase 显示文摘Brunet A Sweeney L B Sturgill J F 2004Science2004,303,5666:1
19Stress-dependent regulation of FOXO transcription factors by the SIRT1 deacetylase显示文摘Brunet A Sweeney L B Sturgill J F 2004Science2004,303,5666:1
20Stress- dependent regulation of FOXO transcription factors by the SIRT1 deacetylase显示文摘Brunet A Sweeney L B Sturgill J F 2004Science2004,303,5666:1
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