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8篇 您的检索式：作者名="TANNER CAMPBELL"

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		题名	作者	年代	出处	被引量
	1	Risk factors and prevalence of perioperative cognitive dysfunction in abdominal aneurysm patients显示文摘	Benoit ACT Campbell BI Tanner JR	2005	J Vase Surg2005，42，5：	1
	2	Risk fastors and prevalence of perioperative cognitive dysfunction in abdominal aneurysm patients显示文摘	Benoit AG Campbell BI Tanner JR	2005	J Vasc Surg2005，42，：	1
	3	Risk factors and prevalence of perioperative cognitive dysfunction in abdominal aneurysm patients显示文摘	Benoit AG Campbell BI Tanner JR	2005	J Vasc Surg2005，42，5：	1
	4	Risk factors and provalence of perioperative cognitive dysfunction in abdominal aneurysm patients显示文摘	Benoit AG Campbell BI Tanner JR	2005	Journal of Vascular Surgery2005，42，5：	1
	5	The upregulation of endothelin and its receptors in porcine coronary arteries in a double balloon injury modal of restenosis显示文摘	Katwa LC Campbell SE Tanner MA	1999	Basic Res Cardiol1999，94，6：	1
	6	Risk factors and prevalence of perioperative cognitive dysfunction in abdominal aneurysm patients显示文摘	Benoit AG Campbell BI Tanner JR	2005	J Vasc Surg2005，42，5：	1
	7	The upregulation of endo thelin and its receptors in porcine coronary arteries in a double balloon injury modek of restenosis 显示文摘	Katwa LC Campbell SE Tanner MA	1999	Basic Res Cardiol1999，94，6：	1
	8	Regulatory role of NFAT1 signaling in articular chondrocyteactivities and osteoarthritis pathogenesis显示文摘Osteoarthritis (OA), the most common form of joint disease, is characterized clinically by joint pain, stiffness,and deformity. OA is now considered a whole joint disease;however, the breakdown of the articular cartilage remains themajor hallmark of the disease. Current treatments targeting OA symptoms have a limited impact on impeding orreversing the OA progression. Understanding the molecular and cellular mechanisms underlying OA development isa critical barrier to progress in OA therapy. Recent studies by the current authors’ group and others have revealedthat the nuclear factor of activated T cell 1 (NFAT1), a member of the NFAT family of transcription factors, regulatesthe expression of many anabolic and catabolic genes in articular chondrocytes of adult mice. Mice lacking NFAT1exhibit normal skeletal development but display OA in both appendicular and spinal facet joints as adults. Thisreview mainly focuses on the recent advances in the regulatory role of NFAT1 transcription factor in the activities ofarticular chondrocytes and its implication in the pathogenesis of OA.	MINGCAI ZHANG TANNER CAMPBELL SPENCER FALCON JINXI WANG	2023	BIOCELL2023，47，10：	0

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