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55篇 您的检索式：作者名="Tarnavski"

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	题名	作者	年代	出处	被引量
1	Endothelial-to-mesenchymal transition contributes to cardiac fibrosis显示文摘	Zeisberg EM Tarnavski O Zeisberg M		0，，08：	1
2	Inhibition of mTOR signaling with rapamycin regresses established cardiac hypertrophy induced by pressure overload显示文摘	McMullen JR Sherwood MC Tarnavski O	2004	Circulation2004，109，24：	1
3	Inhibition of mTOR signaling with rapamycin regresses established cardiac hypertrophy induced by pressure overload显示文摘	McMullen JR Sherwood MC Tarnavski O	2004	Circulation2004，109，24：	1
4	Endothelial-to-mesenchymal transition contributes to cardiac fibrosis 显示文摘	Zeisberg EM Tarnavski O Zeisberg M	2007	Nat Med2007，13，8：	1
5	Inhibition of roTOR signaling with rapamycin regresses established cardiac hypertrophy induced by pressure overload显示文摘	McMullen JR Sherwood MC Tarnavski O	2004	Circulation2004，109，24：	1
6	Rapamycin attenuates load-induced cardiac hypertrophy in mice 显示文摘	Shioi T McMullen JR Tarnavski O	2003	Circulation2003，107，12：	1
7	Endothelial-tomesenchymal transition contributes to cardiac fibrosis显示文摘	Zeisberg EM Tarnavski O Zeisberg M	2007	Nat Med2007，13，：	1
8	Endothelialto-mesenchymal transition contributes to cardiac fibrosis显示文摘	Zeisberg EM Tarnavski O Zeisberg M	2007	Nat Med2007，13，8：	1
9	Mouse cardiac surgery: o comprehensive techniques for the generation of mouse models of human diseases and their application for genomic studies显示文摘	Tarnavski O McMullen JR Schinke M	2004	Physiol Genomics2004，16，：	1
10	Multiscale Geostatistical Analysis of AVHRR, SPOT-VGT, and MODIS Global NDVI Products 显示文摘	Tarnavsky E Garrigues S Brown M E	2008	Remote Sensing of Environment2008，112，2：	1
11	Mouse cardiac surgery: comprehensive techniques for the generation of mouse models of human diseases and their application for genomic studies显示文摘	TARNAVSKI O MCMULLEN J R SCHINKE M	2004	Physiol Ge nomics2004，16，：	1
12	Endothelial-to-mesenchymal transition contributes to cardiac fibrosis显示文摘	Zeisberg EM Tarnavski O Zeisberg M	2007	Nat Med2007，13，8：	1
13	Inhibition of roTOR signaling with rapamycin regresses established cardiac hypertrophy induced by pressure overload 显示文摘	MeMullen JR Sherwood MC Tarnavski O	2004	Circulation2004，109，24：	1
14	Endothelial-to-mesenchymal transition contributes to cardiac fibrosis显示文摘	ZEISBERG E M TARNAVSKI O ZEISBERG M	2007	Nat Med2007，13，：	1
15	Endo- thelial-to-mesenchymal transition contributes to cardiac fibrosis显示文摘	Zeisberg E M Tarnavski O Zeisberg M	2007	Nat Med2007，13，8：	1
16	Inhibition of mTOR signaling with rapamycin regresses established cardiac hypertrophy induced by pressure overload显示文摘	Mcmullen J.R Sherwood M.C Tarnavski O Zhang L Dorfman A.L Shioi T Izumo S		0，，：	1
17	Inhibition of mTOR signaling with rapamycin regressesestablished cardi- ac hypertrophy induced by pressure overload显示文摘	Mcmullen JR Sherwood MC Tarnavski O	2004	Circulation2004，109，24：	1
18	Endothelial- to-mesenchymal transition contributes to cardiac fibrosis 显示文摘	Zeisberg EM Tarnavski O Zeisberg M	2007	Nat Med2007，13，8：	1
19	Endothelial-to-mesenchymal transition contributes to cardiac fibrosis 显示文摘	ZEISBERGEM TARNAVSKI O ZEISBERG M	2007	Nat Med2007，13，8：	1
20	Rapamycin attenuates load-induced cardiac hypertrophy in mice 显示文摘	Shioi T McMuUen JR Tarnavski O	2003	Circulation2003，107，12：	1

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