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21篇 您的检索式:作者名="Thanabalasingham"
    题名 作者 年代 出处 被引量
1Dynamic allocation of subcarriers and transmit powers in an OFDMA cellular network 显示文摘Hanly S Andrew L Thanabalasingham T 2009IEEE Transactions on Information Theory2009,55,12:1
2Negative regulation of Glil and Gli2 activator function by Suppressor of fused through multiple mechanisms显示文摘Barnfield PC Zhang X Thanabalasingham V 2005Differentiation2005,73,8:1
3Assessment of high sensitivity C-reactive protein levels as diagnostic dis- criminator of maturity onset diabetes of the young due to HNF1A mutations 显示文摘Owen KR Thanabalasingham G James TJ 2010Diabetes Care2010,33,9:1
4Assessment of high-sensitivity C-creactive protein levels as diagnostic discriminator of maturity-onset diabetes of the young due to HNF1A mutations显示文摘OwenKR Thanabalasingham G James TJ 0,,09:1
5Do the limits of prolactin in disconnection hyperprolactinaemia need re-definition? A study of 226 patients with histologically verified non- functioning pituitary macroadenoma 显示文摘Karavitaki N Thanabalasingham G Shore HC 2006Clin Endocrinol ( Oxf)2006,65,4:1
6Diagnosis and management of maturity onset diabetes of the young (MODY) 显示文摘Thanabalasingham G Owen KR 2011BMJ2011,343,:1
7Refractory hypercaleaemia secondary to parathyroid carcinoma:response to high-dose denosumab显示文摘Karuppiah D Thanabalasingham G Shine B 2014Eur J Endocr2014,171,1:1
8Diagnosis and management of maturity onset diabetes of the young(MODY) 显示文摘Thanabalasingham G Owen KR 2011BMJ2011,343,:1
9Dynamicallocation of subcarriers and transmit powers in an OFDMA cellular network 显示文摘Hanly S Andrew L and Thanabalasingham T 2009IEEE Transactions on Information Theory2009,55,12:1
10Do the limits of serum prolactin in disconnection hyperprolactinaemia need re-definition? A study of 226 patients with histologically verified non-functioning pituitary macroadenoma 显示文摘Karavitaki N Thanabalasingham G Shore HC 2006Clin Endo- crinol2006,65,4:1
11Systematic assess-ment of etiology in adults with a clinical diagnosis of young-onsettype 2 diabetes is a successful strategy for identifying maturity-onsetdiabetes of the young 显示文摘Thanabalasingham G Pal A Selwood MP 2012Diabetes Care2012,35,6:1
12Assessment of high sensitivity C reactive protein levels as diagnostic discriminator of maturity-onset diabetes of the young due to HNF1A mutations显示文摘OWEN K R THANABALASINGHAM G JAMES T J 2010Diabetes Care2010,33,9:1
13Do the limits of serum prolactin in disconnection hyperprolac- tinaemia need re-definition? A study of 226 patients with histologically verified non-functioning pituitary macroade- noma 显示文摘Karavitaki N Thanabalasingham G Shore HC et 01 2006Clin Endocrinol (Oxf)2006,65,4:1
14Assessment of high-sensitivity C-reactive protein levels as diagnostic discriminator of maturity-onset diabetes of the young due to HNFIA mutations显示文摘Owen KR Thanabalasingham G James TJ 0,,09:1
15Negative regulation of Gli1 and Gli2 activator function by Suppressor of fused through multiple mechanisms显示文摘Barnfield P C Zhang X Thanabalasingham V 2005Differentiation2005,73,8:1
16A large multicentre Europeanstudy validates highsensitivity C - reactive protein (hs -CRP) as a clinical biomarker for the diagnosis of diabetes subtypes 显示文摘Thanabalasingham G Shah N Vaxillaire M 2011Diabetotogia2011,54,11:1
17Diagnosis and management of maturity onset diabetes of the young(MODY)显示文摘Thanabalasingham G Owen K R 2011BMJ2011,343,:1
18Do the limits of serum prolaetin in diseonneetion hyperprolaetinaemia need re-definition? A study of 226 patients with histologically verified non-functioning pituitary maeroadenoma 显示文摘Karavitaki N Thanabalasingham G Shore HC 2006Clin Endoerinol ( Oxf)2006,65,4:1
19Do the limits of serum prolaetin in disconnection hyperprolactinaemia need re- definition? A study of 226 patients with histologically 显示文摘Karavitaki N Thanabalasingham G Shore HC 2006Clin Endocrinol (Oxf)2006,65,4:1
20Negative regulation of gill and gli2 activator function by suppressor of fused through multiple mechanisms 显示文摘Barnfield PC Zhang X Thanabalasingham V 2005Differentiation2005,73,8:1
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