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| 1 | Aetiology of Legg-Calvé-Perthes disease:A systematic review显示文摘BACKGROUND Legg-Calvé-Perthes disease(LCPD) is a clinical condition affecting the femoral head of children during their growth. Its prevalence is set to be between 0.4/100000 to 29.0/100000 children less than 15 years of age with a peak of incidence in children aged from 4 years to 8 years. LCPD aetiology has been widely studied, but it is still poorly understood.AIM To analyse the available literature to document the up-to-date evidence on LCPD aetiology.METHODS A systematic review of the literature was performed regarding LCPD aetiology,using the following inclusion criteria: studies of any level of evidence, reporting clinical or preclinical results and dealing with the aetiology or pathogenesis of LCPD. Two reviewers searched the PubMed and Science Direct databases from their date of inception to the 20th of May 2018 in accordance with the Preferred Reporting Items for Systemic Reviews and Meta-Analyses guidelines. To achieve the maximum sensitivity of the search strategy, we combined the terms: ‘‘Perthes disease OR LCPD OR children avascular femoral head necrosis' with 'pathology OR aetiology OR biomechanics OR genetics' as either key words or MeSH terms.RESULTS We include 64 articles in this review. The available evidence on LCPD aetiology is still debated. Several hypotheses have been researched, but none of them was found decisive. While emerging evidence showed the role of environmental risk factors and evidence from twin studies did not support a major role for genetic factors, a congenital or acquired predisposition cannot be excluded in disease pathogenesis. One of the most supported theories involved mechanical induced ischemia that evolved into avascular necrosis of the femoral head in sensible patients.CONCLUSION The literature available on the aetiology of LCPD presents major limitations in terms of great heterogeneity and a lack of high-profile studies. Although a lot of studies focused on the genetic, biomechanical and radiological background of the disease, there is a lack of consensus on one or multiple major actors of the etiopathogenesis. More studies are needed to understand the complex and multifactorial genesis of the avascular necrosis characterizing the disease. | Vito Pavone Emanuele Chisari Andrea Vescio Claudio Lizzio Giuseppe Sessa Gianluca Testa | 2019 | World Journal of Orthopedics2019，10，3： | 7 |
| 2 | Arthroereisis in juvenile flexible flatfoot:Which device should we implant?A systematic review of literature published in the last 5 years显示文摘BACKGROUND Flexible flatfoot(FFF)is a very common condition in children,characterized by the loss of the medial arch and by an increase in the support base with valgus of the hindfoot.Arthroereisis(AR)procedures are widely performed corrective surgeries and are classified as subtalar AR and calcaneo-stop(CS).AIM We investigated the literature published in the last 5 years with the aim of providing an update on the evidence related to AR treatment in FFF patients.We report the principal findings of subtalar AR and CS procedures concerning clinical and radiological outcomes and complication rates in the general population,young athletes,and obese people according to material device.METHODS Following the guidelines of the Preferred Reporting Items for Systematic Reviews and Meta-Analyses,a systematic review of studies published in the past 5 years and included the PubMed and Science Direct databases was performed on May 6,2020.The research string used was(pediatric OR children OR Juvenile NOT adult)AND(flexible NOT rigid)AND(flat foot OR pes planus)AND(calcaneo-Stop OR arthroereisis OR subtalar extra-articular screw OR SESA OR subtalar arthroereisis OR endosinotarsal).The risk of bias assessment was performed using the Dutch checklist form for prognosis.RESULTS A total of 47 articles were found.Ultimately,after reading the full text and checking reference lists,we selected 17 articles that met the inclusion and exclusion criteria.A total of 1864 FFFs were identified.Eight studies concerned the subtalar AR(47.1%)and nine concerning CS(52.9%).The average age of patients at start of treatment was 11.8 years,the average follow-up of the studies was 71.9 mo(range 29.1-130).Globally,complications occurred in 153 of the 1864 FFF treated,with a rate of 8.2%.CONCLUSION Both AR procedures are valid surgical techniques for treating FFF.Surgeon experience,implant cost,and cosmetic correction are the most common considerations included in the orthopedic device decision-making process.In obese patients,the subtalar AR is not recommended.In adolescents who need to improve sports performance,the CS screw had better results compared with other implants. | Andrea Vescio Gianluca Testa Mirko Amico Claudio Lizzio Marco Sapienza Piero Pavone Vito Pavone | 2021 | World Journal of Orthopedics2021，12，6： | 3 |
| 3 | The role of nuclear factorkappa B in the biology and treatment of multiple myeloma显示文摘 | Berenson JR Ma HM Vescio R | 2001 | Semin Oncol2001，28，6： | 1 |
| 4 | Assessment of thymic output in adults after hematopoietic stem - cell transplantation and prediction of T - cell reconstitution 显示文摘 | Douek DC Vescio RA Betts MR | 2000 | Lancet2000，55，9218： | 1 |
| 5 | Kaposi's sarcoma-associated herpes virus infection of bone marrow dendritic ceils from multiple myeloma patients显示文摘 | Rettig MB Ma HJ Vescio RA | 1997 | Science1997，276，5320： | 1 |
| 6 | The role of nuclear factor-kappaB in the biology and treatment of multiple myeloma显示文摘 | Berenson JR Ma HM Vescio R | 2001 | Semin Oncol2001，28，6： | 1 |
| 7 | Functional results of colorectal and coloanal anastomosis with and without pouch显示文摘 | Santangelo M Romano G Vescio G | 2001 | Ann Ital Chir2001，72，4： | 1 |
| 8 | Assessment of thymic output in adults after haematopoietic stem-cell transplantation and prediction of T-cell reconstitution 显示文摘 | Douek DC Vescio RA Betts MR | 2000 | Lancet2000，355，9218： | 1 |
| 9 | Assessment of thymic output in adults after haematopoietic stem-eel/transplantation and prediction of T-cell reconstitution 显示文摘 | Douek DC Vescio RA Betts MR | 2000 | Lancet2000，355，9218： | 1 |
| 10 | A review of research on the impact of professional learning communities on teaching practice and student learning 显示文摘 | Vescio V Ross D & Adams A | 2008 | Teaching and Teacher Education2008，，1： | 1 |
| 11 | Growing pains: What do we know about etiology? A systematic review显示文摘BACKGROUND Growing pains is the most common cause of musculoskeletal pain in early childhood and was first described in 1823 by French physician Marcel Duchamp.Although it has been researched extensively, the etiology is still unknown.Several theories have been proposed throughout the years.AIM Analyze the available scientific literature to provide an update on the latest evidence on the etiology.METHODS According to the guidelines of the Preferred Reporting Items for Systematic Reviews and Meta-Analyses, the scientific literature on the etiology of growing pains was systematically reviewed using the following inclusion criteria: studies of any level of evidence reporting clinical or preclinical results and dealing with the etiology of growing pains. The medical electronic databases PubMed and Web of Science were searched by two independent authors on October 20, 2018.The search string used was '(growing pains OR benign nocturnal limb pains OR musculoskeletal pains) AND(etiology OR pathogenesis) AND(pediatrics)'.RESULTS A total of 32 articles were included. The etiology of growing pains still remains poorly understood. Many theories have been proposed, but none of them are decisive. A lower pain threshold has been found among patients suffering from growing pains in comparison to healthy controls. Furthermore, evidence suggests an association between growing pains and reduced bone strength in young patients, although this finding still remains controversial. Changes in the vascular perfusion pattern have also been studied. However, the etiology of growing pains does not seem related to a vascular component. The anatomical/mechanical theory has not been supported, but the role of vitamin D deficiency has been investigated many times. Strong recent evidence indicates a genetic susceptibility in the pathogenesis of growing pains. Furthermore,psychological factors also seem to play a strong role in the onset.CONCLUSION The scientific literature about the etiology of growing pains presents heterogeneity and lack of consensus; more studies are needed to understand the genesis of benign musculoskeletal pain syndrome of childhood. | Vito Pavone Andrea Vescio Fabiana Valenti Marco Sapienza Giuseppe Sessa Gianluca Testa | 2019 | World Journal of Orthopedics2019，10，4： | 1 |
| 12 | A Review of Research on the Impact of Professional Learning Communities on Teaching Practice and Student Learning显示文摘 | Vicki Vescio Dorene Ross Alyson Adams | 2008 | Teaching and Teacher Education2008，，24： | 1 |
| 13 | The role of nuclear factor kappaB in the biology and treatment of multiple myeloma显示文摘 | Berenson JR Ma HM Vescio R | | 0，，06： | 1 |
| 14 | Functional results of colorectal and coloanal anastomosis with and without pouch显示文摘 | Santanglo M Romano G Vescio G | | 0，，04： | 1 |
| 15 | Overcoming drug resistance in multiple myeloma: the emergence of therapeutic approaches to induce apoptosis 显示文摘 | Ma MH Vescio RA | 2003 | J Clin Oncol2003，21，22： | 1 |
| 16 | 显示文摘 | Rettig M Ma H Vescio R | 1997 | Science1997，276，： | 1 |
| 17 | Transplantation of CD34 + peripheral blood progenitor cells after nigh 2 dose chemotherapy for patients with advanced multiple myeloma显示文摘 | Schiller G Vescio R Freytes C | 1995 | {H}Blood1995，，1： | 1 |
| 18 | 显示文摘 | Ma H Vescio R Danielian M | 1998 | Blood1998，92，： | 1 |
| 19 | Purging of autologous peripheral-blood stem cells using CD34 selection does not improve overall or progressive-free survival after high-dose chemotherapy for multiple myeloma:results of amulticenter randomized controlled trail显示文摘 | Stewart AK Vescio R Schiller G | 2001 | J Clin Oncol2001，19，17： | 1 |
| 20 | Madelung's disease: case report and discussion of treatment option 显示文摘 | Vescio G Battaglia M | 2001 | Ann Plast Surg2001，46，1： | 1 |
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