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| 1 | Stroke and depression: A bidirectional link显示文摘A number of studies have assessed the influence of depression on the risk of cardiovascular disease. A growing literature indicates a link between depression and cerebrovascular events, although the direction of this association remains unclear. Numerous data have emerged suggesting an association between depressive symptoms and subsequent risk of stroke, thus leading to the hypothesis that a direct causality between depression and stroke exists. Notwithstanding, how depression may act as a risk factor for stroke is still unclear. Depression might be linked to stroke via neuroendocrine and inflammation effects, through correlation with major comorbidities such as hypertensionand diabetes or by intervention of lifestyle behavioral mediators. Finally, antidepressant medications have recently drawn attention for a possible association with increased risk of stroke, although such findings remain uncertain. Depression has been also established as an important consequence after stroke, exerting a significant adverse impact on the course of motor recovery, social functioning and, overall, on quality of life. Post stroke depression occurs in nearly one third of stroke cases, but the exact mechanism leading to depression after stroke is still incompletely understood. In this article, we will review contemporary epidemiologic studies, discuss potential mechanisms and specific aspects of the complex relation between depression and stroke. | Elisabetta Del Zotto Paolo Costa Andrea Morotti Loris Poli Valeria de Giuli Alessia Giossi Irene Volonghi Antonio Callea Alessandro Padovani Alessandro Pezzini | 2014 | World Journal of Meta-Analysis2014，2，3： | 2 |
| 2 | Cerebral amyloid an- gio-patby: a common cause o显示文摘 | Pezzini A Del Zotto E Volonghi I | 2009 | Curt Med Cbem2009，16，20： | 1 |
| 3 | Thromboembolic complications of heparin-induced thrombocytopenia显示文摘 | Giossi A Del Zotto E Volonghi I | 2012 | Blood Coagul Fibrinolysis2012，23，6： | 1 |
| 4 | Cerebral amyloid angiopathy: a common cause of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi L | 2009 | Curr Med Chern2009，16，： | 1 |
| 5 | Cerebral amyloid angiopathy:a common cause of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi I | | 0，，： | 1 |
| 6 | Cerebral amyloidangiopathy:A comm on ca use of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi I | 2012 | Curr Med Chem2012，16，20： | 1 |
| 7 | Cerebral amyloid angiopathy: a common cause of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi I | 2009 | Curr Med Chem2009，16，： | 1 |
| 8 | Cerebral amyloid angiopathy:a common cause of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi I | 2009 | Curr Med Chem2009，16，20： | 1 |
| 9 | Pure midbrain ischemia and hypoplastic vertebrobasilar circulation显示文摘 | Nicola Gilberti Massimo Gamba Angelo Costa Veronica Vergani Raffaella Spezi Alessandro Pezzini Irene Volonghi Dikran Mardighian Roberto Gasparotti Alessandro Padovani Mauro Magoni | 2014 | Neurological Sciences2014，，： | 1 |
| 10 | Cerebral amyloid angiopathy:a common cause of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi I | | 0，，20： | 1 |
| 11 | Large middle cerebral ar- tery and panhemispheric infarction 显示文摘 | Giossi A Volonghi I Del Zotto E | 2012 | Front Neurol Neurosci2012，3，30： | 1 |
| 12 | Cerebral amyloid angiopathy: a common cause of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi I | 2009 | Curr Med Chem2009，16，： | 1 |
| 13 | Thromboembolic complications of heparin-induced thrombocytopenia显示文摘 | Giossi A Del Zotto E Volonghi I | 2012 | Blood Coagul Fibrinolysis2012，23，6： | 1 |
| 14 | Cerebral amyloid angiopathy: a common cause of cerebral hemorrhage显示文摘 | Pezzini A Del Zotto E Volonghi I | 2009 | Curr Med Chem2009，16，20： | 1 |
| 15 | Secondary prevention of ischaemic stroke显示文摘In spite of a documented reduction in incidence in highincome countries over the last decades, stroke is still a leading cause of death and disability worldwide. With the ageing of the population stroke-related economic burden is expected to increase, because of residual disability and its complications, such as cognitive impairment, high risk of falls and fractures, depression and epilepsy. Furthermore, because of the substantial rate of early and long-term vascular recurrences after the first event, secondary prevention after cerebral ischaemia is a crucial issue. This is even more important after minor stroke and transient ischaemic attack(TIA), in order to reduce the risk of potentially more severe and disabling events. To accomplish this aim, acute long-term medical and surgical treatments as well aslifestyle modifications are strongly recommended. However, apart from the well-established indications to thrombolysis, studies in acute phase after a first stroke or TIA are scarce and evidence is lacking. More trials are available for long-term secondary prevention with different classes of drugs, including antithrombotic medications for ischaemic events of arterial and cardiac origin, especially related to atrial fibrillation(antiplatelets and anticoagulants, respectively), lipid lowering agents(mainly statins), blood pressure lowering drugs, surgical and endovascular revascularization procedures. | Irene Volonghi Alessandro Padovani Elisabetta Del Zotto Alessia Giossi Paolo Costa Andrea Morotti Loris Poli Alessandro Pezzini | 2013 | World Journal of Neurology2013，3，4： | 0 |
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