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97篇 您的检索式:作者名="WILENTZ RE"
    题名 作者 年代 出处 被引量
1Pancreatic Cancer显示文摘Yeo CJ Leach SD Wilentz RE 2002Curr Probl Cancer2002,26,2:1
2Immunohistochemical labeling for dpc4 mirrors genetic status in pancreatic adenocarcinomas:a new marker of DPC4 inactivati on显示文摘Wilentz Re Su GH Dai JL 2000Am J Pathol2000,156,1:1
3Molecular pathology of pancreatic cancer 显示文摘Hruban RH Iacobuzio-Donahue C Wilentz RE 2001Cancer J2001,7,4:1
4Identification and analysis of precursors to invasive pancreatic cancer显示文摘Hruban RH Wilentz RE Maitra A 2005Methods Mol Med2005,103,1:1
5Solid - pseudopapillary tumors of the pancreas are genetically distinct from pancreatic ductal adenocarcinomas and almost always harbor beta -catenin mutations显示文摘Abraham SC Klimstra DS Wilentz RE 2002Am J Pathol2002,160,4:1
6Mucinous cystic neoplasms of the pancreas 显示文摘Wilentz RE Albores-Saavedra J Hruban RH 2000Semin Diagn Pathol2000,17,1:1
7Pathologic examination accurately predicts prognosis in mucinous cystic neoplasms of the pancreas显示文摘Wilentz RE Albores-Saavedra J Zahurak M 1999Am J Surg Pathol1999,23,11:1
8Pathologic examination accurately predicts prognosis in mucinous cystic neoplasms of the pancreas显示文摘Wilentz RE Albores-Saavedra J Zahurak M 0,,11:1
9Genetic progression in thepancreatic ducts显示文摘Hruban RH Wilentz RE Kern SE 2000Am J Pathol T2000,156,:1
10Molecular pathology of pancreatic cancer显示文摘Hruban RH Iacobuzio-Donahue C Wilentz RE Goggins M Kern SE 2001Cancer J2001,7,4:1
11Immunohistochemical labeling for DPC4 mirrors genetic status in pancreatic adenocarcinomas a new marker of DPCA inactivation显示文摘Wilentz RE Su GH Dai JL 2000Am J Pathol2000,156,1:1
12Expression of neuropilin-1 in high-grade dysplasia, invasive cancer, and metastases of the human gastrointestinal tract显示文摘Hansel DE Wilentz RE Yeo C J 2004Am J Surg Pathol2004,28,3:1
13Inactivation of the p 16 (1NK4A) tumor-suppressor gene in pancreatic duct lesions: loss of intranuclear expression显示文摘Wilentz RE Geradts J Maynard R 1998Cancer Res1998,58,20:1
14Loss of expression of Dpc4 in pancreatic intmepithelial neoplasia: evidence that DPC4 inactivation occurs late in neoplastic progression 显示文摘Wilentz RE Iacobuzio-Donahue CA Argani P 2000Cancer Res2000,60,7:1
15RPL38, FOSL1, and UPP1 are predom minantly expressed in the pancreatic duetal epithelium显示文摘Sahin F Qiu W Wilentz RE 2005Pancreas2005,30,2:1
16Loss of expression of Dpc4 in pancreatic intraepithelial neoplasia: evidence that DPC4 inactivation occurs late in neoplastic progression显示文摘Wilentz RE Iacobuzio-Donahue CA Argani P 2000Cancer Res2000,60,7:1
17Solid-pseudopapillary tumors of the pancreas are genetically distinct from pancreatic ductal adenocarcinomas and almost always harbor beta-catenin mutations显示文摘Abraham SC Klimstra DS Wilentz RE 2002Am J Pathol2002,160,4:1
18Solid-pseudopapillary tumors of the pancreas are genetically distinct from pancreatic ductal adenocarcinomas and almost always harbor β-catenin mutations 显示文摘Abraham SC Klimstra DS Wilentz RE 2002Am J Pathol2002,160,4:1
19Molecular pathology of pancreatic cancer显示文摘 Iacobuzio-Donahue C Wilentz RE 2001Cancer J2001,7,4:1
20Immunohistochemical labeling for dpc4 mirrors genetic status in pancreatic adenocarcinomas: a new marker of DPC4 inactivation显示文摘Wilentz RE Su GH Dai JL 2000Am J Pathol2000,156,1:1
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