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5篇 您的检索式:作者名="YAO Jinmei"
    题名 作者 年代 出处 被引量
1High-power linear-polarization burst-mode all-fibre laser and generation of frequency-adjustable microwave signal显示文摘Narrowband microwave generation with tuneable frequency is demonstrated by illuminating a photoconductive semiconductor switch(PCSS)with a burst-mode fibre laser.The whole system is composed of a high-power linearly polarized burst-mode pulsed fibre laser and a linear-state PCSS.To obtain a high-performance microwave signal,a desired envelope of burst is necessary and a pulse pre-compensation technique is adopted to avoid envelope distortion induced by the gain-saturation effect.Resulting from the technique,homogenous peak power distribution in each burst is ensured.The maximum energy of the laser burst pulse reaches 200μJ with a burst duration of 100 ns at the average power of 10 W,corresponding to a peak power of 4 kW.When the PCSS is illuminated by the burst-mode fibre laser,narrowband microwave generation with tuneable frequency(0.80-1.12 GHz)is obtained with a power up to 300 W.To the best of the authors’knowledge,it is the first demonstration of frequency-tuneable narrowband microwave generation based on a fibre laser.The high-power burst-mode fibre laser reported here has great potential for generating high-power arbitrary microwave signals for a great deal of applicable demands such as smart adaptive radar and intelligent high-power microwave systems.Xuan He Bin Zhang Shuailin Liu Jinmei Yao Qilin Wu Yuxin Zhao Tao Xun Linyong Yang Jing Hou 2021High Power Laser Science and Engineering2021,9,2:2
2Investigation on gas generation of Li 4 Ti 5 O 12 /LiNi 1/3 Co 1/3 Mn 1/3 O 2 cells at elevated temperature显示文摘Kai Wu Jun Yang Yang Liu Yao Zhang Chenyun Wang Jinmei Xu Feng Ning Deyu Wang 2013Journal of Power Sources2013,,:2
3Adaptive Differential Evolution Algorithm based on Multiple Population显示文摘LIU Chunguang XU Yumei YAO Jinmei YIN Zhelin 2015新教育时代(电子杂志)2015,,11:0
4Phase-separated bimetal enhanced sodium storage:Dimer-like Sn-Bi@C heterostructures with high capacity and long cycle life显示文摘Phase boundaries facilitate the charge transportation and alleviate the intrinsic stress upon cycles.Therefore,how to achieve regular phase boundaries is very attractive.Herein,dimer-like Sn-Bi@C nanostructures,where is a well-defined phase boundary between Sn and Bi,have been prepared by a two-step process for the first time.The phase boundary not only provides additional and fast transportation for Na+,but also mitigates the structure stress/strain upon cycling.Therefore,Sn-Bi@C exhibits a high capacity(472.1 m A h g^(-1)at 2 A g^(-1)for 200 cycles),an ultra-long cyclic life(355.6 mA h g^(-1)at 5 A g^(-1)for 4500cycles)and an excellent rate performance(372 mA h g^(-1)at 10 A g^(-1))for sodium storage,much higher than those of Sn@C,Bi@C,and Sn@C+Bi@C.Notably,the full cells of Sn-Bi@C//Na_(3)V_(2)(PO_(4))_(3)/rGO(SnBi@C//NVP/rGO)demonstrate impressive performance(323 mA h g^(-1)at 2 A g^(-1)for 300 cycles).The underlying mechanism for such an excellent performance is elucidated by in-situ X-ray diffraction,exsitu scanning electron microscopy/high-resolution transmission electron microscopy and atomic force microscopy,revealing the good electrode stability and improved mechanical properties of Sn-Bi@C.The synthetic method is extended to dimer-like Sn-Pb@C and Sn-Ag@C heterostructures,which also exhibit the good cycle stability for sodium storage.Xiaoxiao Hou Yansong Zhu Qian Yao Jinmei Song Chunsheng Wang Yanli Zhou Suyuan Zeng Jian Yang Yitai Qian 2023Journal of Energy Chemistry2023,,4:0
5TRIB3 promotes pulmonary fibrosis through inhibiting SLUG degradation by physically interacting with MDM2显示文摘Pulmonary fibrosis(PF)is the pathological structure of incurable fibroproliferative lung diseases that are attributed to the repeated lung injury-caused failure of lung alveolar regeneration(LAR).Here,we report that repetitive lung damage results in a progressive accumulation of the transcriptional repressor SLUG in alveolar epithelial type II cells(AEC2s).The abnormal increased SLUG inhibits AEC2s from self-renewal and differentiation into alveolar epithelial type I cells(AEC1s).We found that the elevated SLUG represses the expression of the phosphate transporter SLC34A2 in AEC2s,which reduces intracellular phosphate and represses the phosphorylation of JNK and P38 MAPK,two critical kinases supporting LAR,leading to LAR failure.TRIB3,a stress sensor,interacts with the E3 ligase MDM2 to suppress SLUG degradation in AEC2s by impeding MDM2-catalyzed SLUG ubiquitination.Targeting SLUG degradation by disturbing the TRIB3/MDM2 interaction using a new synthetic staple peptide restores LAR capacity and exhibits potent therapeutic efficacy against experimental PF.Our study reveals a mechanism of the TRIB3—MDM2—SLUG—SLC34A2 axis causing the LAR failure in PF,which confers a potential strategy for treating patients with fibroproliferative lung diseases.Xiaoxi Lv Shanshan Liu Chang Liu Yunxuan Li Tingting Zhang Jie Qi Ke Li Fang Hua Bing Cui Xiaowei Zhang Yuxin Liu Jiaojiao Yu Jinmei Yu Li Li Xia Li Zhigang Yao Bo Huang 2023Acta Pharmaceutica Sinica B2023,13,4:0
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