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您的检索式：作者名="Yang Qinchun"
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| 1 | Evaluating the ecological performance of wetland restoration in the Yellow River Delta, China 显示文摘 | Cui Baoshan Yang Qinchun Yang Zhifeng | 2009 | Ecological Engineering2009，35，7： | 1 |
| 2 | Suspended sediment transport of the Hangzhou Bay and its related hydrodynamic analyses显示文摘The 25-h measurements of current speed, flow direction, water depth, suspended sediment concentration and salinity were carried out at six anchored stations in the study area during spring and neap tides in winter of 1987 and summer of 1989. Caculations and analyses of the data obtained show that large amounts of suspended sediments are moved back and forth under the action of tidal current, and the net transport of sediment is small, with its predominance upstream in winter and downstream in summer. These calculations and analyses also suggest that the advective transport of sediment is dominant, while the vertical gravitational circulation of the suspended sediment comes next. Meantime, it is indicated that tidal currents play a major role in the suspended sediment transport, and residual flows have effect on the net transport of the suspended sediment, which is more remarkable during neap tide than during spring tide. | Li Bogen, Yang Zhaoqing and Xie Qinchun Second Institute of Oceanography, State Oceanic Administration, P. O. Box 1207, Hangzhou 310012, China | 1993 | Acta Oceanologica Sinica1993，12，1： | 0 |
| 3 | MicroRNA-33/33^(*) inhibit the activation of MAVS through AMPK in antiviral innate immunity显示文摘Innate immunity plays a prominent role in the host defense against pathogens and must be precisely regulated.As vital orchestrators in cholesterol homeostasis,microRNA-33/33*have been widely investigated in cellular metabolism.However,their role in antiviral innate immunity is largely unknown.Here,we report that VSV stimulation decreased the expression of miR-33/33*through an IFNAR-dependent manner in macrophages.Overexpression of miR-33/33*resulted in impaired RIG-I signaling,enhancing viral load and lethality whereas attenuating type I interferon production both in vitro and in vivo.In addition,miR-33/33*specifically prevented the mitochondrial adaptor mitochondrial antiviral-signaling protein(MAVS)from forming activated aggregates by targeting adenosine monophosphate activated protein kinase(AMPK),subsequently impeding the mitophagy-mediated elimination of damaged mitochondria and disturbing mitochondrial homeostasis which is indispensable for efficient MAVS activation.Our findings establish miR-33/33*as negative modulators of the RNA virus-triggered innate immune response and identify a previously unknown regulatory mechanism linking mitochondrial homeostasis with antiviral signaling pathways. | Danhui Liu Qinchun Tan Jie Zhu Yuanyuan Zhang Yue Xue Yinjing Song Yang Liu Qingqing Wang Lihua Lai | 2021 | Cellular & Molecular Immunology2021，18，6： | 0 |
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