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| 1 | The mobilization of rat’s mesenchymal stem cells into peripheral blood by LiCL and its potency differentiation显示文摘Mesenchymal stem cells (MSCs) are multipotent cells, which can differentiate into different tissues. It is still controversial whether MSCs can be isolated from adult peripheral blood (PB) under normal conditions and whether they can be mobilized in a way similar to that of hematopoietic stem cells (HSCs). In this study, rat MSCs circulating in the PB under normal conditions can be isolated and cultured, and MSCs from PB and BM can be mobilized by LiCL. The mobilized MSCs can be induced to differentiate into neuron by such factors as β-mercaptoethanol and supernatants of nerve cell cultures. The present study provides a broader perspective on the repair of neural trauma. | DENG dun ZOU ZhongMin ZHOU TaoLi AI GuoPing WANG JunPing DONG ShiWu SU YongPing | 2008 | Chinese Science Bulletin2008，53，17： | 2 |
| 2 | The Parkinson’s disease-related genes act in mitochondrial homeostasis显示文摘 | Yan Sai Zhongmin Zou Kaige Peng Zhaojun Dong | 2012 | Neuroscience and Biobehavioral Reviews2012，，9： | 1 |
| 3 | Mesenchymal stromal cells for cell therapy: besides supporting hematopoiesis显示文摘 | Lei Hao Huiqin Sun Jin Wang Tao Wang Mingke Wang Zhongmin Zou | 2012 | International Journal of Hematology2012，，1： | 1 |
| 4 | Stem Cell-Based Therapies for Ischemic Stroke显示文摘 | Lei Hao Zhongmin Zou Hong Tian Yubo Zhang Huchuan Zhou Lei Liu Antonio Salgado | 2014 | BioMed Research International2014，，： | 1 |
| 5 | Management of Lisfranc injury with anterolateral calcaneal compression fracture显示文摘To the Editor:The mechanism of Lisfranc injury is complex and may cause different patterns of fractures or dislocation of the midfoot complex.Abduction force injury is a special injury pattern,which may cause the fracture or(and)dislocation of the lateral column of the Lisfranc joint,especially the cuboid.[1,2,3]However,the anterolateral calcaneus is less commonly involved.Since the fragment of the calcaneocuboid(CC)joint facet is too uncon-spicuous to manifest on a plain X-ray,and surgeons always pay more attention to the Lisfranc joint,anterolateral calcaneal compression fracture and even Chopart joint injuries are often overlooked,which may lead to a delayed surgical management,chronic foot deformities,and functional disability. | Wenqi Gu Rui Zhang Wanjun Liu Zhongmin Shi Guohua Mei Jianfeng Xue Jian Zou Xiaokang Wang Jiantao Jiang | 2022 | Chinese Medical Journal2022，，6： | 1 |
| 6 | Preparation and properties of unidirectional columnar crystal of NdFeB alloys 显示文摘 | Zou Guangrong Chen Zhongmin Shi Zhengxing | 1990 | Rare Metals1990，9，： | 1 |
| 7 | Combustion synthesis of TiCx-TiB2 composites with hypoeutectic,eu-tectic and hypereutectic microstructures显示文摘 | ZOU BINGLIN SHEN PING GAO ZHONGMIN | | 0，，11： | 1 |
| 8 | Multiple mechanisms of SDF-1 promoting VEGF-induced endothelial differentiation o{ mesen- chymal stem cells显示文摘 | Mingke Wang Zhongmin Zou | 2014 | Int J Cardiol2014，177，3： | 1 |
| 9 | Bone Marrow Derivation of Interstitial Cells of Cajal in Small Intestine Following Intestinal Injury显示文摘 | Dengqun Liu Fengchao Wang Zhongmin Zou Shiwu Dong Junping Wang Xinze Ran Chunxue Li Chunmeng Shi Yongping Su Ronnie Gallagher | 2010 | Journal of Biomedicine and Biotechnology2010，，： | 1 |
| 10 | Elite sd1 alleles in japonica rice and their breeding applications in northeast China显示文摘The Green Revolution gene sd1 has been used extensively in modern rice breeding,especially in indica cultivars.However,elite sd1 alleles and related germplasm resources used for japonica rice breeding have not been identified,and extensive efforts are needed for japonica rice breeding to obtain new dwarfing sources.Data from MBKbase-Rice revealed seven sd1 haplotypes in indica and four in japonica rice.Two new sd1 alleles were identified in indica rice.In 295 japonica accessions from northeast Asia,except for the weak functional allele SD1-EQ,sd1-r was the major allele,reducing plant height in comparison with SD1-EQ.Japonica germplasm resources carrying reported sd1 alleles were identified by genotype searching and further verified by literature search,genealogical analysis,and d Caps markers.Pedigrees and geographic distribution showed that sd1-r is an excellent allele widely used in northern China and Tohoku in Japan,and sd1-j is commonly used in east China and Kyushu in Japan.Dongnong-and Xiushui-series cultivars carrying sd1-r and sd1-j,respectively,are essential branches of the backbone parents of Chinese japonica rice,Akihikari and Ce21,with the largest number of descendants and derived generations.In semi-dwarf japonica rice breeding,sd1-d was introgressed into Daohuaxiang 2(DHX2).Dwarf and semi-dwarf lines carrying sd1-d were selected and designated as 1279 and 1280,respectively,after withstanding typhoon-induced strong winds and heavy rains in 2020,and are anticipated to become useful intermediate materials for future genetic research and breeding.This work will facilitate the introduction,parental selection,and marker-assisted breeding,and provide a material basis for the next step in identifying favorable genes that selected together with the sd1 alleles in japonica backbone parents. | Hanjing Sha Hualong Liu Guangxin Zhao Zhongmin Han Huilin Chang Jingguo Wang Hongliang Zheng Jifeng Zhang Yang Yu Yuqiang Liu Detang Zou Shoujun Nie Jun Fang | 2022 | The Crop Journal2022，10，1： | 0 |
| 11 | Targeting TRPV1-mediated autophagy attenuates nitrogen mustard-induced dermal toxicity显示文摘Nitrogen mustard(NM)causes severe vesicating skin injury,which lacks effective targeted therapies.The major limitation is that the specific mechanism of NM-induced skin injury is not well understood.Recently,autophagy has been found to play important roles in physical and chemical exposure-caused cutaneous injuries.However,whether autophagy contributes to NM-induced dermal toxicity is unclear.Herein,we initially confrmed that NM dose-dependently caused cell death and induced autophagy in keratinocytes.Suppression of autophagy by 3-methyladenine,chloroquine,and baflomycin A1 or ATG5 siRNA attenuated NM-induced keratinocyte cell death.Furthermore,NM increased transient receptor potential vanilloid 1(TRPV1)expression,intracellular Ca2+content,and the activities of Car+/calmodulin-dependent kinase kinaseβ(CaMKKB),AMP-activated protein kinase(AMPK),unc-51-like kinase 1(ULK1),and mammalian target of rapamycin(mTOR).NM-induced autophagy in keratinocytes was abolished by treatment with inhibitors of TRPV1(capsazepine),CaMKKβ(STO-609),AMPK(compound C),and ULK1(SBI-0206965)as well as TRPV1,CaMKKB,and AMPK siRNA transfection.In addition,an mTOR inhibitor(rapamycin)had no signifcant effect on NM-stimulated autophagy or cell death of keratinocytes.Finally,the results of the in vivo experiment in NM-treated skin tissues were consistent with the findings of the in vitro experiment.In conclusion,NM-caused dermal toxicity by overactivating autophagy partially through the activation of TRPV1-Ca2+-CaMKKB-AMPK-ULK1 signaling pathway.These results suggest that blocking TRPV1-dependent autophagy could be a potential treatment strategy for NM-caused cutaneous injury. | Mingliang Chen Xunhu Dong Haoyue Deng Feng Ye Yuanpeng Zhao Jin Cheng Guorong Dan Jiqing Zhao Yan Sai Xiuwu Bian Zhongmin Zou | 2021 | Signal Transduction and Targeted Therapy2021，6，2： | 0 |
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