|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | New Small Shelly Fossils(Acanthocassis and Xinlispina Gen.Nov.) from the Fortunian Stage(Early Cambrian) in Southern China显示文摘We describe new material of three-dimensionally phosphatized small shelly fossils Acanthocassis and Xinlispina gen.nov.from the Fortunian Stage(early Cambrian) of southern Shaanxi and northern Sichuan Provinces.The new materials allow description of the delicate morphology of these skeletons, and also their skeletogenesis.Acanthocassis and Xinlispina have comparable morphological features: a main branch and radially arranged sub-branches.They differ from each other in many details, e.g., the presence of a single central branch only in Xinlispina, and the presence of small nodes only in Acanthocassis.Acanthocassis cannot be a naked anthopolyp or hydropolyp because of the more-or-less stiffened cuticle and the absence of a mouth.Acanthocassisand Xinlispina differ from coeval sponge spicules and chancelloriid sclerites and rosettes of crossed pedicellariae of echinoderms, and might be cuticular ornaments of animals of unclear affinities.They occur in the first small shelly fossil assemblage zone, and could be adopted as auxiliary fossils for biostratigraphic correlation in southern China. | SHAO Tiequan LIU Yunhuan WANG Qi ZHANG Huaqiao TANG Hanhua CAO Xi HE Huahua ZHANG Yanan LI Yuan ZHENG Panlin ZHU Chengyu HU Jiaxi | 2015 | Acta Geologica Sinica(English Edition)2015，89，5： | 2 |
| 2 | 显示文摘 | Wang Fangwei Zhang Panlin | 1999 | J Phys C: Condens Matter1999，11，： | 1 |
| 3 | DNADam ageEffectsofHydroxyapatiteUltrofinePowderonW-256 Sarcom a Cells and Lym phocytes in Rats显示文摘To explore the anticancer mechanism and DNA damages of hydroxyapatite ultrofine powder (HAUFP) on lymphocytes of rats, DNA damages in W 256 sarcoma cells and lymphocytes of rats were measured by single cell gel electrophoresis (SCGE). The results showed that HAUFP damaged DNA of W 256 sarcoma cells obviously but only cause slight damage of DNA of lymphocytes in rats. It is suggested that HAUFP selectively damaged DNA of tumor cells with only mild damage of lymphocyte DNA. HAUFP has powerful anticancer effect and little genetic toxicity. | XIA Qinghua 1, HU Panlin 2, CHEN Daoda 1, ZHANG Jianpin 1, TIAN Yuan 1, ZHANG Jinghui 1 1 Department of General Surgery, Xiehe Hospital, Tongji Medical University, Wuhan 430022 2 Departmnet of General Surgery, Puqi Peoples Hospital, | 1999 | Journal of Huazhong University of Science and Technology(Medical Sciences)1999，19，3： | 1 |
| 4 | Effect of manganese substitution in R2AlFe16-xMnx(R = Y, Ho, Pr, Nd)compounds显示文摘 | Hao Yanming Yah Qiwei Zhang Panlin | 1997 | ACTA Physica Sinica1997，6，： | 1 |
| 5 | Formation and magnetic properties of Dy2Fe17-xMnxC(x = 0 - 17) prepared by arc-melting显示文摘 | Wang Fangwei Zhang Panlin Shoa Baogen | 1996 | Acta Physica Sinica (Overseas Edition)1996，5，： | 1 |
| 6 | Neutron Diffraction Studies of the Magnetic Structure of Ho_2Fe_9Ga_(8~x)Alx | Chen Dongfeng Gou Cheng Sun Kai China Institute of Atomic Energy,P.O.Box 275-30,Beijing 102413Yan Qiwei Zhang Panlin Shen Baogen Cheng Zhaohua State Key Laboratory of Magnetism,Intstitute of Physics,the Chinese Academy of Sciences,P.O.Box 603,Beijing 100080 | 1997 | Chinese journal of nuclear physics1997，，1： | 0 |
| 7 | EFFECTS OF THE LATENT HEAT AND PARTICLE DISTRIBUTION ON THE DILEPTON SPECTRUM显示文摘The previously derived relativistic hydrodynamic equation from energy-momentum conservation is generalized to include the particle number conservation.From calculation on the basis of the new formalism we find that the particle distribution and latent heat significantly change the characteristic dilepton spectrum for the formation of a quark matter,which had been predicted by the other author. | HE Zejun ZHANG Jiaju LI Panlin QIU Xijun | 1990 | Chinese Physics Letters1990，7，8： | 0 |
| 8 | Association of variants in the KIF1A gene with amyotrophic lateral sclerosis显示文摘Background:Amyotrophic lateral sclerosis(ALS)is a devastating progressive neurodegenerative disease that affects neurons in the central nervous system and the spinal cord.As in many other neurodegenerative disorders,the genetic risk factors and pathogenesis of ALS involve dysregulation of cytoskeleton and neuronal transport.Notably,sen-sory and motor neuron diseases such as hereditary sensory and autonomic neuropathy type 2(HSAN2)and spastic paraplegia 30(SPG30)share several causative genes with ALS,as well as having common clinical phenotypes.KIF1A encodes a kinesin 3 motor that transports presynaptic vesicle precursors(SVPs)and dense core vesicles and has been reported as a causative gene for HSAN2 and SPG30.Methods:Here,we analyzed whole-exome sequencing data from 941 patients with ALS to investigate the genetic association of KIF1A with ALS.Results:We identified rare damage variants(RDVs)in the KIF1A gene associated with ALS and delineated the clini-cal characteristics of ALS patients with KIF1A RDVs.Clinically,these patients tended to exhibit sensory disturbance.Interestingly,the majority of these variants are located at the C-terminal cargo-binding region of the KIF1A protein.Functional examination revealed that the ALS-associated KIF1A variants located in the C-terminal region preferentially enhanced the binding of SVPs containing RAB3A,VAMP2,and synaptophysin.Expression of several disease-related KIF1A mutants in cultured mouse cortical neurons led to enhanced colocalization of RAB3A or VAMP2 with the KIF1A motor.Conclusions:Our study highlighted the importance of KIF1A motor-mediated transport in the pathogenesis of ALS,indicating KIF1A as an important player in the oligogenic scenario of ALS. | Panlin Liao Yanchun Yuan Zhen Liu Xiaorong Hou Wanzhen Li Jin Wen Kexuan Zhang Bin Jiao Lu Shen Hong Jiang Jifeng Guo Beisha Tang Zhuohua Zhang Zhonghua Hu Junling Wang | 2022 | Translational Neurodegeneration2022，11，1： | 0 |
| 9 | PINK1-mediated Drp1^(S616) phosphorylation modulates synaptic development and plasticity via promoting mitochondrial fission显示文摘Dynamic change of mitochondrial morphology and distribution along neuronal branches are essential for neural circuitry formation and synaptic efficacy.However,the underlying mechanism remains elusive.We show here that Pink1 knockout(KO)mice display defective dendritic spine maturation,reduced axonal synaptic vesicles,abnormal synaptic connection,and attenuated long-term synaptic potentiation(LTP).Drp1 activation via ^(S616) phosphorylation rescues deficits of spine maturation in Pink1 KO neurons. | Qingtao Gao Runyi Tian Hailong Han Jesse Slone Caifang Wang Xiao Ke Tongmei Zhang Xiangyu Li Yuhong He Panlin Liao Fang Wang Ye Chen Shiqing Fu Kexuan Zhang Fangfang Zeng Yingxuan Yang Zhuo Li Jieqiong Tan Jiada Li Youming Lu Taosheng Huang Zhonghua Hu Zhuohua Zhang | 2022 | Signal Transduction and Targeted Therapy2022，7，5： | 0 |
您所在的机构暂未开通此部分的功能，请联系管理员