|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | Impact of osteoporosis in dental implants:A systematic review显示文摘AIM: To assess the failure and bone-to-implant contact rate of dental implants placed on osteoporotic subjects. METHODS: Extensive examination strategies were created to classify studies for this systematic review. MEDLINE(via Pub Med) and EMBASE database were examined for studies in English up to and including May 2014. The examination presented a combination of the MeS H words described as follow: 'osteoporosis' or 'osteopenia' or 'estrogen deficiency' AND 'implant' or 'dental implant' or 'osseointegration'. Assessment of clinical and/or histological peri-implant conditions in osteoporosis subjects treated with titanium dental implants. The examination included a combination of the MeS H terms described as follow: 'osteoporosis' or 'osteopenia' or 'estrogen deficiency' AND 'implant' or 'dental implant' or 'osseointegration'.RESULTS: Of 943 potentially eligible articles, 12 were included in the study. A total of 133 subjects with osteoporosis, 73 subjects diagnosed with osteopenia and 708 healthy subjects were assessed in this systematic review. In these subjects were installed 367, 205, 2981 dental implants in osteoporotic, osteopenic and healthy subjects, respectively. The failure rate of dental implant was 10.9% in osteoporotic subjects, 8.29% in osteopenic and 11.43% in healthy ones. Bone-to-implant contact obtained from retrieved implants ranged between 49.96% to 47.84%, for osteoporosis and non-osteoporotic subjects. CONCLUSION: Osteoporotic subjects presented higher rates of implant loss, however, there is a lower evidence to strengthen or refute the hypothesis that osteoporosis may have detrimental effects on bone healing. Consequently, final conclusions regarding the effect of osteoporosis in dental implant therapy cannot be made at this time. There are no randomized clinical trial accessible for evaluation and the retrospectivenature of the evaluated studies shall be taken in account when interpreting this study. | Gabriela Giro Leandro Chambrone Abrao Goldstein Jose Augusto Rodrigues Elton Zenóbio Magda Feres Luciene Cristina Figueiredo Alessandra Cassoni Jamil Awad Shibli | 2015 | World Journal of Orthopedics2015，6，2： | 11 |
| 2 | Mechanisms of neuroplasticity and brain degeneration: strategies for protection during the aging process显示文摘Aging is a dynamic and progressive process that begins at conception and continues until death.This process leads to a decrease in homeostasis and morphological,biochemical and psychological changes,increasing the individual’s vulnerability to various diseases.The growth in the number of aging populations has increased the prevalence of chronic degenerative diseases,impairment of the central nervous system and dementias,such as Alzheimer’s disease,whose main risk factor is age,leading to an increase of the number of individuals who need daily support for life activities.Some theories about aging suggest it is caused by an increase of cellular senescence and reactive oxygen species,which leads to inflammation,oxidation,cell membrane damage and consequently neuronal death.Also,mitochondrial mutations,which are generated throughout the aging process,can lead to changes in energy production,deficiencies in electron transport and apoptosis induction that can result in decreased function.Additionally,increasing cellular senescence and the release of proinflammatory cytokines can cause irreversible damage to neuronal cells.Recent reports point to the importance of changing lifestyle by increasing physical exercise,improving nutrition and environmental enrichment to activate neuroprotective defense mechanisms.Therefore,this review aims to address the latest information about the different mechanisms related to neuroplasticity and neuronal death and to provide strategies that can improve neuroprotection and decrease the neurodegeneration caused by aging and environmental stressors. | Mariana Toricelli Arthur Antonio Ruiz Pereira Guilherme Souza Abrao Helena Nascimento Malerba Julia Maia Hudson Sousa Buck Tania Araujo Viel | 2021 | Neural Regeneration Research2021，16，1： | 8 |
| 3 | Can chemokines be used as biomarkers for endometriosis? A systematic review 显示文摘 | Borrelli GM Abrao MS Mechsner S | 2014 | Hum Re- prod2014，29，： | 1 |
| 4 | Randomized clinical trial of a levonorgestrel-releasing intrauterine system and a depot GnRH analogue for the treatment of chronic pelvic pain in women with endometriosis显示文摘 | Petta CA Ferriani RA Abrao MS | 2005 | Hum Reprod2005，20，7： | 1 |
| 5 | Histological classifica- tion of endometriosis as a predictor of response to treatment显示文摘 | Abrao M S Neme R M Carvalho F M | 2003 | lnt J Gynaecol Obstet2003，82，1： | 1 |
| 6 | Vascular density and distribution of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 (Flk-1) are significantly higher in patients with deeply infiltrating endometriosis affecting the rectum显示文摘 | Machado DE Abrao MS Berardo PT | 2008 | Fertil Steril2008，90，1： | 1 |
| 7 | Expression of FoxM1 is required for the proliferation of medulloblastoma cells and indicates worse survival of patients 显示文摘 | Priller M Poschl J Abrao L | 2011 | Clin Cancer Res2011，17，21： | 1 |
| 8 | Vascular density and distribution of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 ( Flk-1 ) are significantly higher in patients with deeply infiltrating endometriosis affecting the rectum 显示文摘 | Machado DE Abrao MS Berardo PT | 2008 | Fertil Steril2008，90，1： | 1 |
| 9 | Endometriosis of the ureter and bladder are not associated diseases显示文摘 | Abrao MS Dies JA Jr Bellelis P | | 0，，： | 1 |
| 10 | Anatomical causes of female infertility and their management显示文摘 | Abrao MS Muzii L Marana R | 2013 | Int J Gynaecol Obstet2013，1232，： | 1 |
| 11 | Randomized clinical trial of a levonorgestrel releasing intrauterine system and a depot GnRH an- alogue for the treatment of chronic pelvic pain in women with endome- triosis 显示文摘 | Petta CA Ferriani RA Abrao MS | 2005 | Hum Reprod2005，20，7： | 1 |
| 12 | Evaluation of CA-125 and soluble CD-23 in patients with pelvic endometriosis: a case-control study显示文摘 | Ramos IM Podgaec S Abrao MS | 2012 | Rev Assoc M6d Bras2012，58，1： | 1 |
| 13 | Evaluation of pain in three hysterosalpingography techniques:metal cannula with and without paracervical blockage and balloon catheter显示文摘 | De Mello JF Sr Abrao MS Cerri GG | | 0，，： | 1 |
| 14 | Rapid improvementof acute schizophrenia symptoms after intravenous sodium nitroprusside :a randomized,double - blind,placebo - controlled trial 显示文摘 | Hallak JE Maia - de - Oliveira JP Abrao J | 2013 | JAMAPsychiatry2013，70，7： | 1 |
| 15 | Randomized clinical trial of a levonorgestrel-releasing intrauterine system and a depot GnRH analogue for the treatment of chronic pelvic pain in women with endometriosis 显示文摘 | Petta CA Ferriani RA Abrao MS | 2005 | Hum Reprod2005，20，7： | 1 |
| 16 | Effect of low-dose droperidol on the QT interval during and after general anesthesia:a Placebo-controlled study显示文摘 | White PF son D Abrao J Klein KW Navarette B | | 0，，06： | 1 |
| 17 | Endometriosis of the ureter and bladder are not associated diseases显示文摘 | Abrao MS Dias JA Jr Bellelis P | 2009 | Fertil Steril2009，91，5： | 1 |
| 18 | Deeply infiltrating endometriosis affecting the rectum and lymph nodes显示文摘 | Mauricio S. Abrao Sergio Podgaec Joao Antonio Dias Marcelo Averbach Ray Garry Luis Fernando Ferraz Silva Filomena M. Carvalho | 2006 | Fertility and Sterility2006，，3： | 1 |
| 19 | Hard turning: AISI 4340 high strength low alloy steel and AISI D2 coht work tool steel显示文摘 | J G Lima RF Avila A M Abrao | 2005 | Journal of Materials Processing Technology2005，169，3： | 1 |
| 20 | Can chemokines be used as biomarkers for endometriosis? A systematic re- view显示文摘 | Borrelli GM Abrao MS Mechsner S | 2014 | Hum Reprod2014，29，2： | 1 |
您所在的机构暂未开通此部分的功能，请联系管理员