|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | Therapy of antiglomerular basement membrane antibody disease: Analysis of prognostic significance of clinical, pathology and treatment factors显示文摘 | Johnson JP Moore J Austin HA | 1985 | Medicine1985，64，4： | 1 |
| 2 | A systematic review of open reduction and internal fixation of periprosthetic femur fractures with or without allografi strut, cerclage, and locked plates 显示文摘 | Moore RE Baldwin K Austin MS | 2014 | The Journal of arthroplasty2014，29，5： | 1 |
| 3 | A systematic review of open reduction and internal fixation of periprosthetic femur fractures with or without allograft strut,cerclage,and locked plates Arthroplasty显示文摘 | Moore RE Baldwin K Austin MS | 2014 | 29(5):872-8762014，29，5： | 1 |
| 4 | A systematic review of open reduction and internal fixation of periprosthetic femur fractures with or without al ograft strut,cerclage,and locked plates显示文摘 | Moore RE Baldwin K Austin MS | | 0，，05： | 1 |
| 5 | Emotional intelligence,Machiavellianism and emotional manipulation:Does EI have a dark side显示文摘 | Austin EJ Farrelly D Black C Moore H | | 0，，： | 1 |
| 6 | Mandibular 'tripod' advancement of a Class II Division 2 deepbite malocclusion 显示文摘 | Low LE Moore TE Austin KR | 2010 | Am J Orthod Dentofacial Orthop2010，137，2： | 1 |
| 7 | Therapy of antiglomerular basement membrane antibody disease:analysis of prognostic significance of clinical,pathologic and treatment factors显示文摘 | Johnson JP Moore J Jr Austin HA | | 0，，： | 1 |
| 8 | A systematic review of open reduction and internal fixation of periprosthetic femur fractures with or without allograft strut,cerclage,and locked plates显示文摘 | Moore RE Baldwin K Austin MS | 2014 | J Arthroplasty2014，29，5： | 1 |
| 9 | Therapy of anti -glomeru- lar basement membrane antibody disease:Analysis of prognostic signifi- cance of clinical, pathologic and treatment factors 显示文摘 | Johnson JP Moore J Jr Austin HA 3 rd | 1985 | Medicine ( Balti- more)1985，64，4： | 1 |
| 10 | Oxidative stress and COVID-19-associated neuronal dysfunction: mechanisms and therapeutic implications显示文摘Severe acute respiratory syndrome(SARS)-CoV-2 virus causes novel coronavirus disease 2019(COVID-19),and there is a possible role for oxidative stress in the pathophysiology of neurological diseases associated with COVID-19.Excessive oxidative stress could be responsible for the thrombosis and other neuronal dysfunctions observed in COVID-19.This review discusses the role of oxidative stress associated with SARS-CoV-2 and the mechanisms involved.Furthermore,the various therapeutics implicated in treating COVID-19 and the oxidative stress that contributes to the etiology and pathogenesis of COVID-19-induced neuronal dysfunction are discussed.Further mechanistic and clinical research to combat COVID-19 is warranted to understand the exact mechanisms,and its true clinical effects need to be investigated to minimize neurological complications from COVID-19. | Dylan R.Bowen Suhrud Pathak Rishi M.Nadar Rachel D.Parise Sindhu Ramesh Manoj Govindarajulu Austin Moore Jun Ren Timothy Moore Muralikrishnan Dhanasekaran | 2023 | Acta Biochimica et Biophysica Sinica2023，55，8： | 0 |
您所在的机构暂未开通此部分的功能，请联系管理员