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11篇 您的检索式:作者名="Brisa"
    题名 作者 年代 出处 被引量
1Repression of the Auxin Response Pathway Increases Arabidopsis Susceptibility to Necrotrophic Fungi显示文摘在植物,到 necrotrophic 病原体的抵抗取决于在不同荷尔蒙系统之间的相互影响,例如水杨酸调整的那些酸(SA ) , jasmonic 酸(JA ) ,乙烯,和 abscisic 酸。由 SA 小径发信号的植物生长素的压抑最近被显示贡献抗菌剂抵抗。这里,我们表明表明在刺激植物生长素的 SCF (Skp1CullinF 盒子) ubiquitination 小径展览有缺点的异种 axr1, axr2,和 axr6 的那 Arabidopsis 植物生长素增加的危险性到 necrotrophic 真菌 Plectosphaerella cucumerina 和 Botrytis cinerea。另外,被 SCF-ubiquitin/proteasome 机械通常为移动指向的植物生长素 transcriptional 抑压者 AXR3 的稳定发生在 P 之上。cucumerina 感染。植物生长素运输或 proteasome 功能的药理学抑制各作为在非对待的植物生长素反应异种损害野类型的植物的 necrotroph 抵抗到类似的程度。这些结果建议植物生长素发信号为到 necrotrophic 真菌 P 的抵抗是重要的。cucumerina 和 B。cinerea。SGT1b (编码 HSP90/HSC70 合作女伴的二 Arabidopsis SGT1 基因之一) 在某些抵抗(R) 基因调节到 biotrophic 病原体的植物生长素和 JA 回答和抵抗支持 SCF E3-ubiquitin ligase 建筑群的功能。我们发现 sgt1b 异种对 P 同样抵抗。是的 cucumerina 野类型的植物。相反地, auxin/SCF 发信号异种在到 obligate biotrophic oomycete 的被触发 RPP4 的抵抗是不妥协的, Hyaloperonospora parasitica。因此,在到 oomycetes 的 R 调节基因的抵抗的 SGT1b 的占优势的行动看起来在除帮助 SCF E3-ubiquitin ligases 以外的一个地点。然而, sgt1b axr1 的基因添加在到 H 的危险性的两倍异种。一旦 plantpathogen 相容性被建立, parasitica 建议调停 SCF 的 ubiquitination 贡献限制 biotrophic 病原体殖民。Francisco Llorente Paul M usksktt Andrea Sanchez-Vallet Gemma Lopez Brisa Ramos Clara Sanchez-Rodriguez Lucia Jorda Jane Parker Antonio Molina 2008Molecular Plant2008,1,3:8
2Coal combustion modeling of large power plant, for NOx abatement 显示文摘Brisa T L Cadavidb F Caillata S 2007Fuel2007,86,14:1
3Thidiazuron EnhancesAxillary and Adventitious Shoot Proliferation in Juvenile Explantsof Mediterranean Provenances of Maritime Pine Pinus pinastei显示文摘Hum^nez A Blasco M Brisa C 2011In Vitro Cellular & Developmental Biology-Plant2011,47,:1
4Clustering of fastfood restaurants around schools:A novel application of spatial statistics to the study of food environments显示文摘AUSTIN SBS STEVEN JMM BRISA SSM 0,,09:1
5Functional remediation for bipolar disorder显示文摘Anabel MA Carla T Brisa S 2011Clin Pract Epidemiol Ment Health2011,7,:1
6Preparation and characterization of polysulfone microcapsules for perfume release 显示文摘Brisa P Cinta P 2012Chem- ical Engineering Journal2012,179,1:1
7Racial Disparities in Tissue Plasminogen Activator Treatment Rate for Stroke: A Population-Based Study显示文摘Amie W. Hsia Dorothy F. Edwards Lewis B. Morgenstern Jeffrey J. Wing Nina C. Brown Regina Coles Sarah Loftin Andrea Wein Sara S. Koslosky Sabiha Fatima Brisa N. Sánchez Ali Fokar M. Chris Gibbons Nawar Shara Annapurni Jayam-Trouth Chelsea S. Kidwell 2011Stroke2011,,8:1
8TNFα和IL-6体外对颗粒物的反应:随颗粒物大小、季节、多环芳烃和土壤含量的变化显示文摘[背景]颗粒物(PM)与人类健康之间的关联随着季节变化而不同,这种现象可能与颗粒物的成分以及毒性相关的季节性交互作用有关。[目的]检测PM成分的季节性变化,并使用多个PM样本评估PM的体外促炎症可能性。[方法]以5个污染源不同的城市地区为采集地点,每周采集样本,在多雨温暖季节和干燥寒冷季节累积采集90份PM_(10)和PM_(2.5)样本。对其中检测到的元素、多环芳烃(PAHs)和内毒素进行主成分分析(PCA)。在人单核细胞(THP-1)中检测PM诱导肿瘤坏死因子α(TNFα)和白介素6(IL-6)分泌的可能性,并用组分评分建立促炎症反应的模型。[结果]PM的成分随其大小和季节而变化。PCA确定了PM中两种随季节变化的主要组分。燃烧相关的成分(如钒、苯并(a)芘、苯并(a)蒽)组成组分1(C_1),土壤相关的成分(如内毒素、硅、铝)组成组分2(C_2)。多雨温暖季节采集的PM中C_2含量较高,而干燥寒冷季节采集的PM(尤其是PM_(2.5))中C_1含量较高。细胞因子生成水平与PM_(10)和C_2(多雨温暖季节)正相关,但与PM_(2.5)和C_1(干燥寒冷季节)负相关。C_2含量较高的PM暴露水平越高,TNFα分泌越多;样本含有≥0.1%的C_1相关PAHs时,无论样本中C_2含量高低,暴露于该样本后TNFα对PM的反应性分泌都会降低。IL-6检测结果提示PM成分和颗粒大小之间存在复杂的交互作用。[结论]TNFα分泌具有季节性并与PM大小相关,PM中的土壤含量及PAH含量的变化是其根本原因。这些结果提示,流行病学研究中一些健康结局与PM之间关联的季节性差异能用PM组分的混合物来解释。Natalia Manzano-León Jesús Serrano-Lomelin Brisa N.Sánchez Raúl Quintana-Belmares Elizabeth Vega Inés Vázquez-López Leonora Rojas-Bracho Maria Tania López-Villegas Felipe Vadillo-Ortega Andrea De Vizcaya-Ruiz Irma Rosas Perez Marie S.O’Neill Alvaro R.Osornio-Vargas 季麟(翻译) 金泰廙(审校) 2016环境与职业医学2016,33,12:1
9Comparison of cytokine levels in depressed, manic and euthymie patients with bipolar disorder 显示文摘Brietzke E Stertza L Brisa SF 2009Journal of Affective Disorders2009,11,:1
10Aqueous extracts from Tenebrio molitor larval and pupal stages inhibit early hepatocarcinogenesis in vivo显示文摘Hepatocellular carcinoma(HCC),which is the most frequent primary liver malignancy,is ranked as the sixth most common cancer and the third leading cause of cancer-related deaths worldwide,with its incidence expected to continue rising.One of the reasons is that most patients are diagnosed at an advanced stage when therapeutic options are ineffective.The development of HCC is attributed to a chronic exposition to either one or a combination of low amounts of different hepatotoxins,such as in hepatitis virus infection,alcohol consumption,aflatoxin from contaminated foods,metabolic factors,and exposure to chemical carcinogens from tobacco smoke(Fomer et al.,2018).Armando ZEPEDA-BASTIDA Juan OCAMPO-LOPEZ Brisa Rodope ALARCON-SANCHEZU Osiris German IDELFONSO-GARCIA Sandra ROSAS-MADRIGAL Diana Ivette APARJCIO-BAUTISTA Julio Isael PEREZ-CARREON Saul VILLA-TREVINO Jaime ARELLANES-ROBLEDO 2021Journal of Zhejiang University-Science B(Biomedicine & Biotechnology)2021,22,12:0
11Erratum to:Aqueous extracts from Tenebrio molitor larval and pupal stages inhibit early hepatocarcinogenesis in vivo显示文摘Following an Assessment by the Autonomous University of Hidalgo State and the National Institute of Genomic Medicine,this erratum corrects the authorship of this article by adding Dulce Maria MORENO-GARCiA as the first author.The published article(Zepeda-Bastida et al.,2021)has now been updated with the corrected authorship,author contributions,and acknowledgments,which may be seen in this erratum.Dulce María Moreno-García Armando Zepeda-Bastida Juan Ocampo-López Brisa Rodope Alarcón-Sánchez Osiris Germán Idelfonso-García Sandra Rosas-Madrigal Diana Ivette Aparicio-Bautista Julio Isael Pérez-Carreón Saúl Villa-Treviño Jaime Arellanes-Robledo 2023Journal of Zhejiang University-Science B(Biomedicine & Biotechnology)2023,24,4:0
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