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34篇 您的检索式:作者名="Buhner"
    题名 作者 年代 出处 被引量
1The crystallography,metallography and composition of gold显示文摘Hough R M Butt C R M Fischer Buhner J 2009Elements2009,5,5:1
2Fast element mapping of titanium wear around implants of different surface structures 显示文摘Meyer U Buhner M Buchter A 2006Clini Oral Impant Res2006,17,:1
3Activation of human enteric neurons by supernatants of colonic biopsy specimens from patients with irritable bowel syndrome显示文摘Buhner S Li Q Vignali S Barbara G 2009Gastroenterology2009,137,4:1
4Influence of titanium surfaces on attachment of osteoblast - like cells in Vitro 显示文摘Jayaraman M Meyer U Buhner M 2004Biomaterials2004,25,4:1
5Assessing international diversification of West German corporations显示文摘Buhner Rolf 1987Strategic Management Journal1987,8,1:1
6Barrier function of the gut and multiple organ dysfunction after cardiac surgery显示文摘Braun JP Buhner S K astrup M 0,,01:1
7Localization of angiogenic growth factors and their receptors in the human placental bed throughout normal human pregnancy 显示文摘Schiess B Innes BA Buhner JN 2008Placenta2008,14,6:1
8Assessing international diversification of west german corporations 显示文摘Buhner R 1987Strategic Management Journal1987,8,1:1
9Placental Fas and Fas ligand expression in normal early term anti molar pregnancy 显示文摘Pongcharoen S Searle RF Buhner JN 2004Placenta2004,25,41:1
10Risk of replacement of descending aorta with a standardized left heart bypass technique显示文摘 JURMANN M BUHNER B 1994J Thorac Cardiovasc Surg1994,107,1:1
11Pseudoallergic reactions in chronic urticaria are associated with altered gastroduodenal permeability显示文摘Buhner S Reese I Kuehl F 2004Allergy2004,59,10:1
12Malnutrition and impaired muscle strength in patients with Crohn’s disease and ulcerative colitis in remission显示文摘Luzia Valentini Lennart Schaper Carsten Buning Susanne Hengstermann Thomas Koernicke Wolfgang Tillinger Francesco William Guglielmi Kristina Norman Sabine Buhner Johann Ockenga Matthias Pirlich Herbert Lochs 2008Nutrition2008,,7:1
13Altered status of antioxidant vitamins and fatty acids in patients with inactive inflammatory bowel disease显示文摘Susanne Hengstermann Luzia Valentini Lennart Schaper Carsten Buning Thomas Koernicke Michaela Maritschnegg Sabine Buhner Wolfgang Tillinger Nunzia Regano Francesco Guglielmi Brigitte M. Winklhofer-Roob Herbert Lochs 2008Clinical Nutrition2008,,4:1
14Use of real-time PCR for determining copy number and zygosity in transgenic plants 显示文摘Buhner B Baldwin IT 2004Plant Cell Rep2004,23,5:1
15家族性克罗恩病肠黏膜通透性增加的遗传基础:CARD15 3020insC突变的作用显示文摘Background and aim: A genetically impaired intestinal barrier Function has long been suspected to be a predisposing factor for Crohn’s disease (CD). Recently, mutations of the capsase recruitment domain family, member 15 (CARD15) gene have been identified and associated with CD. We hypothesise that a CARD15 mutation may be associated with an impaired intestinal barrier. Methods: We studied 128 patients with quiescent CD, 129 first degree relatives (CD-R), 66 non-related household members (CD-NR), and 96 healthy controls. The three most common CARD15 polymorphisms (R702W, G908R, and 3020insC)were analysed and intestinal permeability was determined by the lactulose/mannitol ratio. Results: Intestinal permeability was significantly increased in CD and CD-R groups compared with CD-NR and controls. Values above the normal range were seen in 44% of CD and 26% of CD-R but only in 6% of CD-NR, and in none of the controls. A household community with CD patients, representing a common environment, was not associated with increased intestinal permeability in family members. However, 40% of CD first degree relatives carrying a CARD15 3020insC mutation and 75% (3/4) of those CD-R with combined 3020insC and R702W mutations had increased intestinal permeability compared with only 15% ofwild-types, indicating a genetic influence on barrier function. R702W and G908R mutations were not associated with high permeability. Conclusions: In healthy first degree relatives, high mucosal permeability is associated with the presence of a CARD15 3020insC mutation. This indicates that genetic factors may be involved in impairment of intestinal barrier function in families with IBD.Buhner S. Buning C. Genschel J. 程妍(译) 陈云茹(校) 2006世界核心医学期刊文摘(胃肠病学分册)2006,2,8:1
16Genetic basis for increased intestinal permeability in families with Crohn's disease:role of CARD15 3020insC mutation? 显示文摘Buhner S Buning C Genschel J 2006Gut2006,55,3:1
17Antibiotic use in German uni- versity hospitals 1998-2000(Project INTERUNI-Ⅱ ) 显示文摘De With K Bergner J Buhner R 2004Int J An- timicrob Agents2004,24,3:1
18Effect of detoxification of dilute - acid corn fiber hydrolysate on xylitol production显示文摘Buhner J Agblevor F A 2004Appl Biochem Bioteehnol2004,119,1:1
19The Success of Mergers in Germany显示文摘Buhner R 1991International Journal of Industrial Organization1991,,9:1
20Genetic basis for increased in- testinal permeability in families with Crohn' s disease : role of CARD15 3020ins C mutation? 显示文摘Buhner S Buning C Gensehel J 2006Gut2006,55,:1
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