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7篇 您的检索式：作者名="CHAI Fen"

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		题名	作者	年代	出处	被引量
	1	Virtual test system of permanent - magnetic DC motor 显示文摘	CUI Shumei WANG Yue CHAI Fen	2003	Journal of Harbin Institute of Technology2003，10，2：	1
	2	Virtual Test System of Permanent-Magnet DC Motor显示文摘	Cui Shu-Mei Wang Yue Chai Fen	2003	Journal of Harbin Institute of Technology2003，2，：	1
	3	One-pot triethanolamine-assisted hydrothermal synthesis of Ag/ZnO heterostructure microspheres with enhanced photocatalytic activity显示文摘	Bo Chai Xing Wang Siqing Cheng Huan Zhou Fen Zhang	2013	Ceramics International2013，，：	1
	4	Virtual Test System of Permanent-Magnet DC Motor显示文摘	Cui Shu-Mei Wang Yue Chai Fen Hong-Xing Wu Bao-Ting Liu Shu-Kang Cheng	2003	Journal of Harbin institute of Techno-logy Journal of Harbin Institute of Technology2003，，2：	1
	5	Visible light photocatalytic performance of hierarchical BiOBr microspheres synthesized via a reactable ionic liquid显示文摘	Bo Chai Huan Zhou Fen Zhang Xiang Liao Meixia Ren	2014	Materials Science in Semiconductor Processing2014，，：	1
	6	Virtual Test System of Permanent- Magnet DC Motor 显示文摘	CUI Shumei WANG Yue CHAI Fen	2003	Journal of Harbin Institute of Technology2003，，2：	1
	7	Semaphorin 7A promotes human vascular smooth muscle cell proliferation and migration through theβ-catenin signaling pathway显示文摘Background:Vascular smooth muscle cells(VSMCs)undergo a conversion from a contractile phenotype to a proliferative synthetic phenotype,contributing to the pathogenesis of cardiovascular diseases.Semaphorin 7A(SEMA7A)is a glycosylphosphatidylinositol-anchored membrane protein that plays an important role in vascular homeostasis by regulating endothelial cell behaviors.However,the expression and role of SEMA7A in VSMCs remain unclear.Methods:In this study,we screened for VSMC-regulating genes in publicly available datasets and analyzed the expression of SEMA7A in human coronary artery smooth muscle cells(hCASMCs)treated with platelet-derived growth factor-BB(PDGF-BB).The effects of SEMA7A overexpression and knockdown on hCASMC proliferation and migration were examined.The signaling pathways involved in the action of SEMA7A in hCASMCs were determined.Results:Bioinformatic analysis showed that SEMA7A was significantly dysregulated in VSMCs treated with oxidized low-density lipoprotein or overexpressing progerin,a pro-atherogenic gene.The PDGF-BB stimulation led to a concentration-and time-dependent induction of SEMA7A.Depletion of SEMA7A attenuated PDGF-BB-induced hCASMC proliferation and migration.Conversely,overexpression of SEMA7A enhanced hCASMC proliferation and migration.Mechanistically,SEMA7A stimulated the activation of theβ-catenin pathway and upregulated c-Myc,CCND1,and MMP7.Knockdown ofβ-catenin impaired SEMA7A-induced hCASMC proliferation and migration.Conclusions:SEMA7A triggers phenotype switching in VSMCs through theβ-catenin signaling pathway and may serve as a potential therapeutic target for cardiovascular diseases.	XIAOSU SONG FEN GAO HONG LI WEIWEI QIN CHANJUAN CHAI GUOJUAN SHI HUIYU YANG	2023	BIOCELL2023，47，4：	0

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