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6篇 您的检索式:作者名="Daizong Li"
    题名 作者 年代 出处 被引量
1Effect of quinoicl redox mediators on the aerobic decolorization of azo dyes by cells and cell extracts from Escherichia coli 显示文摘CUI Daizong LI Guofang ZHAO Dan 2015Environmen- tal Science and Pollution Research2015,22,6:1
2Efficacy and Acceptability of Orthokeratology for Slowing Myopic Progression in Children: A Systematic Review and Meta-Analysis显示文摘Daizong Wen Jinhai Huang Hao Chen Fangjun Bao Giacomo Savini Antonio Calossi Haisi Chen Xuexi Li Qinmei Wang Antonio Queiros 2015Journal of Ophthalmology2015,,:1
3Purification and characterization of an azoreductase from Escherichia coli CD-2 possessing quinone reductase activity显示文摘CUI Daizong LI Guofang ZHAO Dan 2012Proeess Bio- chemistry2012,47,54:1
4Microbial com- munity structures in mixed bacterial consortia for azo dye treatment under aerobic and anaerobic eonditions显示文摘CUI Daizong LI Guofang ZHAO Dan 2012Journal of Hazardous Materials2012,,6:1
5Bioinformatic analysis of embryo development related small heat shock protein Hsp26 in Artemia species显示文摘Artemia 胚胎能忍受极端温度,长期的血缺氧,干燥和紧张的条件的另外的宽变化。胚胎怎么熬过这些压力,是一个很有趣、未解决的题目。为了解决这个问题,我们分析了核苷酸并且为 Hsp26 推出了蛋白质顺序,对 Artemia 胚胎开发特定的分子的女伴。Hsp26 的 cDNAs 从八 Artemia 种被定序并且推断 Hsp26 氨基酸序列被分析。帮助计算机的分析显示 5-untranslated 区域和所有 3 introns 在开发期间为 Hsp26 基因表示包含许多通常认为的行动 cis 元素,包括热吃惊元素(HSE ) , Dfd, dl, CF2-II, Hb 和 AP-1 有约束力的地点。Hsp26 3-untranslated 终止者的第二等的结构为 transcriptional 结束包含基本结构基础。Hsp26 份额从另外的有机体与 sHSPs (小热吃惊蛋白质) 定序类似。推出的蛋白质的物理化学的性质蛋白质扑灭系数,等电位的点和 antigenic 地点例如理论分子的重量,也被获得。一七肽的原子本地化信号(NLS ) PFRRRMM 被发现,它建议 Hsp26 蛋白质被假设在原子核内被定位。特定的 phosphorylation 地点也是的丝氨酸, threonine 和酷氨酸和 kinase 的 phosphorylation 地点的数字在 Hsp26 蛋白质顺序定位了。这些研究将帮助我们完成 Hsp26 的更好的理解,为 sHSPs 的宽广含意在甲壳纲的动物胚胎开发工作。Jiaqing WANG Lin HOU Zhenfeng HE Daizong Li Lijuan JIANG 2012Frontiers in Biology2012,7,4:0
6The inhibitory effect of transthyretin gene on growth of human hepatoma cells显示文摘Transthyretin(TTR) gene was highly expressed in normal liver and it has been found to be deleted in part of DNA samples from human hepatic cancer. Its mRNA expression was suppressed in most hepatoma samples. In order to study the biological effect of TTR gene on the growth of hepatoma cells, a recombinant vector containing TTR cDNA was constructed by pCMV, then it was transfected into hepatoma cell lines SMMC-7721 and Q3.It has been demonstrated that the inhibition of growth rate of TTR cDNA transfected hepatoma cells was about 50%in strength compared with that of the control. This inhibition was further enhanced when the transfected hepatoma cells were treated with all-trans retinoic acid. Hepatoma cells of cell lines PLC/PRF/5, SMMC-7721 and Q3 as well as hepatoma cells SMMC-7721 transfected with pCMV or pCMV-TTR were analyzed for TTR expression by Northern hybridization. The low level of TTR expression was found in both hepatoma cell lines and in SMMC-7721cells transfected with pCMV alone. However, a remarkable TTR mRNA expression was observed in hepatoma SMMC-7721 cells transfected with pCMV-TTR. It seems possible that TTR gene might be a candidate of cancer suppressor gene for human hepatic cancer.LIU CHAOTING JIN YAO DAIZONG LI HUIQIU JIANG JIANREN GU.(National Laboratory for Oncogenes and Related Genes,Shanghai Cancer Institute, Shanghai 200032, China.) 1994Cell Research1994,4,1:0
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