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7篇 您的检索式：作者名="David Wallach"

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		题名	作者	年代	出处	被引量
	1	Effects of calcitonin on bone quality and osteoblastic function显示文摘	Stanley Wallach John R. Farley David J. Baylink Leona Brenner-Gati	1993	Calcified Tissue International1993，，5：	1
	2	Caspase-8 Blocks Kinase RIPK3-Mediated Activation of the NLRP3 Inflammasome显示文摘	Tae-Bong Kang Seung-Hoon Yang Beata Toth Andrew Kovalenko David Wallach	2013	Immunity2013，，：	1
	3	Effects of calcitonin on bone quality and osteoblastic function显示文摘	Stanley Wallach John R. Farley David J. Baylink Leona Brenner-Gati	1993	Calcified Tissue International1993，，5：	1
	4	Rethinking lda:Why priors matter显示文摘	Hanna Wallach David Mimno Andrew Mc Callum	2009	Advances in Neural Information Processing Systems222009，，：	1
	5	Statistical Methods for Predieting ResPonses to Applied Nitrogen and Caleulating Optimal Nitrogen Rates显示文摘	David Makowski Daniel Wallach Jean-Marc Meynard	2001	Agron J2001，，93：	1
	6	Survival Function of the FADD-CASPASE-8-cFLIP L Complex显示文摘	Christopher P. Dillon Andrew Oberst Ricardo Weinlich Laura J. Janke Tae-Bong Kang Tehila Ben-Moshe Tak W. Mak David Wallach Douglas R. Green	2012	Cell Reports2012，，：	1
	7	MORTl/FADD is involved in liver regeneration显示文摘AIM: To explore the role of the adaptor molecule in liver regeneration after partial hepatectomy (PH). METHODS: We used transgenic mice expressing an N-terminal truncated form of MORT1/FADD under the control of the albumin promoter. As previously shown, this transgenic protein abrogated CD95- and CD120a-mediated apoptosis in the liver. Cyclin A expression was detected using Western blotting. ELISA and RT-PCR were used to detect IL-6 and IL-6 mRNA, respectively. DNA synthesis in liver tissue was measured by BrdU staining. RESULTS: Resection of 70% of the liver was followed by a reduced early regenerative response in the transgenic group at 36 h. Accordingly, 36 h after hepatectomy, cyclin A expression was only detectable in wild-type animals. Consequently, the onset of liver mass restoration was retarded as measured by MRI volumetry and mortality was significantly higher in the transgenic group. CONCLUSION: Our data demonstrate for the first time an involvement of the death receptor molecule MORT1/ FADD in liver regeneration, beyond its well described role as part of the intracellular death signaling pathway.	Marcus Schuchmann Felix Rückert Jose F Garcia-Lazaro Andrea Karg Jürgen Burg Natalia Knorr Jürgen Siebler Eugene E Varfolomeev David Wallach Wolfgang Schreiber Ansgar W Lohse Peter R Galle	2005	World Journal of Gastroenterology2005，11，46：	0

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