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| 1 | Error sensing strategy for active acoustic structure based on distributed displacement sensors显示文摘察觉到途径的 nearfield 错误,是在活跃声学的结构的实现遇到的关键问题之一,理论上被调查。基本想法包含一小部分 PVDF 电影对到为测量总数的主要面板和第二等的面板的表面的 boned 被散发健全力量。首先,在这份报纸,与活跃声学的结构联系的一个理论模型被建立。第二,为 PVDF 对的形状系数的公式基于察觉到的模式和 PVDF 建模的声学的放射被导出。最后,很多计算机模拟被执行,在散发的健全电源的 PVDF 对和相应减小的形状是结果表明的 calculated.The 察觉到的建议 nearfield 错误的可行性策略。 | CHEN Ke'an CHEN Guoyue LI Shuang PAN Haoran | 2007 | Chinese Journal of Acoustics2007，26，3： | 4 |
| 2 | Coexpression of bile salt hydrolase gene and catalase gene remarkably improves oxidative stress and bile salt resistance in Lactobacillus casei 显示文摘 | Guohong Wang Sheng Yin Haoran An | 2010 | J Ind Mierobiol Biotechnol2010，9，： | 1 |
| 3 | Characterization of an anti-Listeria bacteriocin produced by Lactobacillus planlarmn LB-BI isolated from koumiss, a traditionally fermentt-d dairy product from China显示文摘 | Ying Xie Haoran An Yanling Hao el al | 2011 | Food Control2011，22，： | 1 |
| 4 | A CMA-ES-Based Adversarial Attack Against Black-Box Object Detectors显示文摘Object detection is one of the essential tasks of computer vision.Object detectors based on the deep neural network have been used more and more widely in safe-sensitive applications,like face recognition,video surveillance,autonomous driving,and other tasks.It has been proved that object detectors are vulnerable to adversarial attacks.We propose a novel black-box attack method,which can successfully attack regression-based and region-based object detectors.We introduce methods to reduce search dimensions,reduce the dimension of optimization problems and reduce the number of queries by using the Covariance matrix adaptation Evolution strategy(CMA-ES)as the primary method to generate adversarial examples.Our method only adds adversarial perturbations in the object box to achieve a precise attack.Our proposed attack can hide the specified object with an attack success rate of 86%and an average number of queries of 5,124,and hide all objects with a success rate of 74%and an average number of queries of 6,154.Our work illustrates the effectiveness of the CMA-ES method to generate adversarial examples and proves the vulnerability of the object detectors against the adversarial attacks. | LYU Haoran TAN Yu’an XUE Yuan WANG Yajie XUE Jingfeng | 2021 | Chinese Journal of Electronics2021，30，3： | 0 |
| 5 | 白介素25介导的自分泌循环在银屑病发育中的促炎功能与机制研究显示文摘文章简介银屑病是一种由免疫系统所介导的临床上的自身免疫性皮肤疾病,具体表现为皮肤表皮增厚、银屑形成、真皮炎症细胞浸润等,且伴随一系列的并发症,全世界大约1%~2%的人受到该疾病的困扰。虽然从淋巴细胞中产生的白介素17已经被证实在银屑病的发育过程中起到关键作用. | 徐妙 Huiping Lu Young-Hee Lee Yelin Wu Kewei Liu Yuling Shi Haoran An Jingren Zhang Xiaohu Wang 赖玉平 董晨 | 2019 | 科学新闻2019，0，2： | 0 |
| 6 | Activation of NLRP3 Inflammasome via Drp1 Overexpression in Kupffer Cells Aggravates Ischemia-reperfusion Injury in Hepatic Steatosis显示文摘Background and Aims:Donors with fatty livers are considered to address the shortage of livers for transplantation,but those livers are particularly sensitive to ischemia-reperfusion injury(IRI),and an increased incidence of graft failure is observed.Kupffer cells account for 20–35%of liver nonparenchymal cells,and have been shown to participate in the process of IRI and inflammatory reactions of hepatic steatosis.NOD-like receptor thermal protein domain-associated protein 3(NLRP3)is an intracellular sensor activated by Kupffer cells to promote generation and participates in IRI.Dynamics-associated protein 1(Drp1)is one of the main proteins regulating mitochondrial division and exacerbates IRI by affecting mitochondrial dynamics.The mechanism of interaction of Kupffer cells with Drp1 and NLRP3 to aggravate IRI has not been clarified.Methods:A mouse model of hepatic steatosis was established by feeding the mice with a high-fat diet.In vitro experiments were performed using AML12 normal mouse liver cells and RAW264.7 mononuclear macrophage cells cultured in medium with palmitate and oleic acid.Western blotting and immunohistochemical(IHC)staining were used to detect the expression of NLRPP3 and Drp1 in IRI in the control and high-fat diet groups.The expression of F4/80+cells during IRI in hepatic steatosis was verified by IHC staining,and the role of NLRPP3 and Drp1 in Kupffer-cell mediated IRI was investigated by targeting Drp-1 inhibition.Results:Drp1 and NLRP3 expression was increased during IRI in hepatic steatosis,and the expression of Drp1 and NLRP3 were decreased after the elimination of Kupffer cells.That indicated Kupffer cells were involved in the process of IRI in hepatic steatosis through the action of Drp1 and NLRP3.After Drp1 inhibition,liver function was restored and NLRP3 expression level was reduced.Conclusions:Kupffer cells aggravated IRI in hepatic steatosis via NLRP3 and Drp1.Drp1 inhibitors might be useful as specific therapeutics to alleviate IRI in hepatic steatosis and may have promise in case of liver donor shortage. | Lu Zhang Mingfu Wang Ran An Jun Dai Shujun Liu Ming Chen Haoran Ding | 2023 | Journal of Clinical and Translational Hepatology2023，11，5： | 0 |
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