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1Dendritic cell deficiencies persist seven months after SARS-CoV-2 infection显示文摘Severe Acute Respiratory Syndrome Coronavirus(SARS-CoV)-2 infection induces an exacerbated inflammation driven by innate immunity components.Dendritic cells(DCs)play a key role in the defense against viral infections,for instance plasmacytoid DCs(pDCs),have the capacity to produce vast amounts of interferon-alpha(IFN-α).In COVID-19 there is a deficit in DC numbers and IFN-αproduction,which has been associated with disease severity.In this work,we described that in addition to the DC deficiency,several DC activation and homing markers were altered in acute COVID-19 patients,which were associated with multiple inflammatory markers.Remarkably,previously hospitalized and nonhospitalized patients remained with decreased numbers of CD1c+myeloid DCs and pDCs seven months after SARS-CoV-2 infection.Moreover,the expression of DC markers such as CD86 and CD4 were only restored in previously nonhospitalized patients,while no restoration of integrinβ7 and indoleamine 2,3-dyoxigenase(IDO)levels were observed.These findings contribute to a better understanding of the immunological sequelae of COVID-19.Alberto Pérez-Gómez Joana Vitallé Carmen Gasca-Capote Alicia Gutierrez-Valencia María Trujillo-Rodriguez Ana Serna-Gallego Esperanza Muñoz-Muela María de los Reyes Jiménez-Leon Mohamed Rafii-El-Idrissi Benhnia Inmaculada Rivas-Jeremias Cesar Sotomayor Cristina Roca-Oporto Nuria Espinosa Carmen Infante-Domínguez Juan Carlos Crespo-Rivas Alberto Fernández-Villar Alexandre Pérez-González Luis Fernando López-Cortés Eva Poveda Ezequiel Ruiz-Mateos JoséMiguel Cisneros Sonsoles Salto-Alejandre Judith Berastegui-Cabrera Pedro Camacho-Martínez Carmen Infante-Domínguez Marta Carretero-Ledesma Juan Carlos Crespo-Rivas Eduardo Márquez JoséManuel Lomas Claudio Bueno Rosario Amaya JoséAntonio Lepe Jerónimo Pachón Elisa Cordero Javier Sánchez-Céspedes Manuela Aguilar-Guisado Almudena Aguilera Clara Aguilera Teresa Aldabo-Pallas Verónica Alfaro-Lara Cristina Amodeo Javier Ampuero María Dolores Avilés Maribel Asensio Bosco Barón-Franco Lydia Barrera-Pulido Rafael Bellido-Alba Máximo Bernabeu-Wittel Candela Caballero-Eraso Macarena Cabrera Enrique Calderón Jesús Carbajal-Guerrero Manuela Cid-Cumplido Yael Corcia-Palomo Juan Delgado Antonio Domínguez-Petit Alejandro Deniz Reginal Dusseck-Brutus Ana Escoresca-Ortega Fátima Espinosa Nuria Espinosa Michelle Espinoza Carmen Ferrándiz-Millón Marta Ferrer Teresa Ferrer Ignacio Gallego-Texeira Rosa Gámez-Mancera Emilio García Horacio García-Delgado Manuel García-Gutiérrez María Luisa Gascón-Castillo Aurora González-Estrada Demetrio González Carmen Gómez-González Rocío González-León Carmen Grande-Cabrerizo Sonia Gutiérrez Carlos Hernández-Quiles Inmaculada Concepción Herrera-Melero Marta Herrero-Romero Luis Jara Carlos Jiménez-Juan Silvia Jiménez-Jorge Mercedes Jiménez-Sánchez Julia Lanseros-Tenllado Carmina López Isabel López Álvaro López-Barrios Luis F.López-Cortés Rafael Luque-Márquez Daniel Macías-García Guillermo Martín-Gutiérrez Luis Martín-Villén JoséMolina Aurora Morillo María Dolores Navarro-Amuedo Dolores Nieto-Martín Francisco Ortega María Paniagua-García Amelia Peña-Rodríguez Esther Pérez Manuel Poyato Julia Praena-Segovia Rafaela Ríos Cristina Roca-Oporto Jesús F.Rodríguez María Jesús Rodríguez-Hernández Santiago Rodríguez-Suárez Ángel Rodríguez-Villodres Nieves Romero-Rodríguez Ricardo Ruiz Zida Ruiz de Azua Celia Salamanca Sonia Sánchez Víctor Manuel Sánchez-Montagut César Sotomayor Alejandro Suárez Benjumea Javier Toral 2021Cellular & Molecular Immunology2021,18,9:2
2Apheis: Health Impact Assessment of Long-term Exposure to PM 2.5 in 23 European Cities显示文摘Elena Boldo Sylvia Medina Alain Tertre Fintan Hurley Hans-Guido Mücke Ferrán Ballester Inmaculada Aguilera 2006European Journal of Epidemiology2006,,6:1
3Home Outdoor NO2 and New Onset of Self-Reported Asthma in Adults显示文摘Bénédicte Jacquemin Jordi Sunyer Bertil Forsberg Inmaculada Aguilera David Briggs Raquel García-Esteban Thomas G?tschi Joachim Heinrich Bengt J?rvholm Debbie Jarvis Danielle Vienneau Nino Künzli 2009Epidemiology2009,,1:1
4长期交通相关空气污染暴露与亚临床动脉粥样硬化的关联:REGICOR研究显示文摘[背景]长期空气污染暴露对动脉粥样硬化形成慢性过程影响的流行病学证据并不多。[目的]通过测量颈动脉内膜中层厚度(IMT)和踝臂指数(ABI),探讨长期交通相关空气污染暴露与亚临床动脉粥样硬化的关联。[方法]采用REGICOR(RegistreGironidelCor:赫罗纳心脏病注册)研究中2780名参加者复检期间(2007--2010年)的数据进行横断面分析,REGICOR是在西班牙赫罗纳进行的、以人群为基础的前瞻性队列研究。Marcela Rivera Xavier Basagaa Inmaculada Aguilera Maria Foraster David Agis Eric de Groot Laura Perez Michelle A.Mendez Laura Bouso Jaume Targa Rafael Ramos Joan Sala Jaume Marrugat Roberto Elosua Nino Künzli 2013环境与职业医学2013,30,5:1
5生命早期室外空气污染暴露与婴幼儿呼吸系统健康、耳部感染和湿疹:INMA研究显示文摘[背景]产前和生命早期可能是空气污染对婴幼儿健康造成有害影响的重要窗口期。[目的]选择西班牙出生队列中2199名婴儿,研究他们在孕期和出生第1年空气污染暴露与年龄为12~18个月时的呼吸系统疾病、耳部感染和湿疹之间的关联。[方法]父母提供医生诊断的呼吸道感染(LRTI)、喘息、湿疹和耳部感染等信息。使用经时间调整的土地利用回归模型,估计个体二氧化氮(NO2)和苯的暴露情况。采用对数二项式回归模型和联合随机效应meta分析,评估4个研究地点空气污染暴露对健康结果的影响。[结果]在孕期,NO2均值每增加10μg/m3与LRT[I相对危险度(RR)=1.05,95%CI:0.98~1.12]和耳部感染(RR=1.18,95%CI:0.98~1.41)相关联。NO2每增加1个四分位距(IQR),LRTI的RR为1.08(95%CI:0.97~1.21),耳部感染的RR为1.31(95%CI:0.97~1.76)。与NO2相比,苯暴露均值每增加1个IQR,其与LRTI具有类似的关联性(RR=1.06,95%CI:0.94~1.19),与耳部感染的关联性稍弱(RR=1.17,95%CI:0.93~1.46)。母亲孕期大多数时间在家里的婴幼儿,其关联性稍强。由于出生第1年的空气污染暴露与产前暴露高度相关,所以无法分辨各个暴露时期的相对重要性。[结论]研究结果表明生命早期环境空气污染暴露可能增加婴幼儿上、下呼吸道感染的风险。Inmaculada Aguilera Marie Pedersen Raquel Garcia-Esteban Ferran Ballester Mikel Basterrechea Ana Esplugues Ana Fernández-Somoano Aitana Lertxundi Adonina Tardón Jordi Sunyer 2013环境与职业医学2013,30,6:0
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